University of Surrey (UNIS)
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Maths lecture notes I have taken this semester. Contain enough examples to understand content. Notes may have written see other examples however these are not needed to understand the content (simply to remind me to watch lectures).
Could be used as summary notes for revision.
This document covers theme 1 of second year immunology in detail, including notes obtained from the KUBY textbook integrated throughout. These notes closely follow the lectures, and provide detailed lecture notes covering the basic components of immunology - all that is needed to ace the THEME 1 exam and cover the final exam
In depth notes covering THEME 2 of the immunology course on adaptive immunity. This is a mixture of immunology lecture notes combined with 'Kuby' text book notes to provide a comprehensive immunology revision tool for year 2 study
- Define key concept 
- Contrast relevant theoretical frameworks Link concept to theoretical frameworks (Institutional theory, resource base view(entry mode), transaction cost analysis, agency theory, relational view)
- Acknowledge great thinkers (NO year or book)
Resource base view – Barney
Transaction cost analysis – Williamson
-Implications for managers (similar to our reports) 
-Limitation of the theories - you add few critical concepts-points / where these concepts are sufficient ...
Exam notes on everthing, Including self study. Very detailed with example Questions of how each is applied
In this lecture we cover the regulatory DNA sequences and the protein through which they function. That includes distant elements and proximal promoter elements as well as transcription factors. We also analyse DNA binding domains, activation domains etc. We have the Nuclear receptor family and the mechanisms of activation through modulation of chromatin, recruitment of RNA polymerase and stimulation of elongation.
Description of RNA processing starting from basic knowledge about mRNAs. Continue to splicing with the spliceosome and how it's achieved. We also have alternative splicing and its control through activator or repressor proteins. That can lead to mutation of splice sites or splicing factors, and therefore to diseases.
How we control translation