Pkb Study guides, Class notes & Summaries

Notes taken on the topic of buffers as taught by Dr. Williamson in CHEM 120 at TAMU. These notes show some examples of RICE tables used for buffers to calculate Ka, Kb, pH, and/or pOH. They then describe and show how to use the Henderson Hasselbalch Equation to calculate the pH or pOH of a buffer solution which is an imperative skill in chemistry. Finally, they describe how else these buffers are sued and other applications.

CHEM 102 Winter 18 Exam 3 (B) Questions and Answers Potentially useful data: x  2 a 1. Please choose the letter “B” as your answer for this question. 2. Consider the following endothermic chemical equilibrium. What change below would cause an increase in the I2 (g) concentration? 2 NOI (g) ⇌ 2 NO (g) + I2 (g) a) decreasing the temperature b) adding more NO c) increasing the pressure d) adding more NOI e) All of the above. 3. At 25 oC, an aqueous solution has [H3O+]...

Introduction to mathematical quantification of acidity and basicity and the theories of acidity

Everything you need to ace thermodynamic and chemical reactivity class. Formula sheet (cheat sheet), all the formulas, graphs and concepts on a double-sided page that you can use during open book exams/ as a formula sheet. Colour-coded for easy memorization. includes: -ideal gas law and real gas. -Chemical equilibrium -Thermochemistry (first law of thermodynamics, enthalpy and heat, Hess law). -Thermochemistry (second and third laws, entropy, free energy G and spontaneity). -kinetics...

An overview of calcium signalling. Describes the control and the difference between transient and sustained signalling. The PKB activation pathway is also discussed in relation to calcium signalling.

Lecture of LIFE305 Cell Signalling on phosphatidylinositol 3-kinase signalling. Information on the structure and function of phosphatidylinositols/phosphoinositides and their role in cell signalling. In-depth, step-wise overview of the PI3K pathway with detailed information on the different PI3K isoforms, PI3K signalling regulation by phosphatases/phospholipases, Akt activation, mTOR signalling and PI3K inhibitors.