Nurs 3310 Test 1 (Black) Exam Questions
With correct Answers
Endocrine |System |- |VERIFIED |ANSWER✔✔-Composed |of |cells |and |organs |that |manufacture |and
|secrete |hormones. |It |is |a |system |of |communication |that |controls |many |life-long |bodily |responses |and
|functions
Thymus |- |VERIFIED |ANSWER✔✔-larger |in |children |while |they |develop |an |immune |system. |It |gets
|smaller |without |use |(as |we |age).
Glands |- |VERIFIED |ANSWER✔✔-may |produce |or |secret |hormone
Hormones |- |VERIFIED |ANSWER✔✔-goes |to |its |target |and |causes |an |action |or |response
Target |cells |- |VERIFIED |ANSWER✔✔-read |and |follow |hormone's |instructions
how |does |the |endocrine |system |respond |to |stimuli? |- |VERIFIED |ANSWER✔✔-by |releasing |hormones
|from |endocrine |glands
Hormones |regulate |four |major |body |functions: |- |VERIFIED |ANSWER✔✔-Reproduction
Growth |and |development
Homeostasis
Metabolism
All |hormones |share |certain |general |characteristics: |- |VERIFIED |ANSWER✔✔--Rates |and |patterns
-Operate |within |a |feedback |system
-Affect |target |cells |with |specific |receptors |and |then |act |to |initiate |specific |cell |functions |or |activities
|(they |respond |only |to |those |hormones |for |which |they |have |receptors)
-Excreted |by |kidneys |or |deactivated |by |the |liver |or |cellular |mechanisms
,Negative |feedback |loop |in |the |endocrine |system |- |VERIFIED |ANSWER✔✔-cold |exposure |à
|hypothalamus |(thyrotropin- |releasing |hormone) |à |anterior |pituitary |(thyrotropin) |à |thyroid |à
|thyroxine. |Then |a |negative |feedback |loop |tells |anterior |pituitary |to |stop |releasing |thyrotropin.
Hyposecretion |- |VERIFIED |ANSWER✔✔-gland |releases |an |inadequate |amount |of |hormone |to |meet
|physiologic |needs; |not |enough |secreted
Hypersecretion |- |VERIFIED |ANSWER✔✔-increased |secretion. |Too |much |secreted
Hyporesponsiveness |- |VERIFIED |ANSWER✔✔-of |the |target |organ |will |cause |the |same |set |of |clinical
|symptoms |as |hyposecretion. |usually |caused |by |deficiency |of |receptors
Hyposecretion |- |VERIFIED |ANSWER✔✔--NOT |ENOUGH
-Congenital
-Absence |of |enzyme |needed |for |synthesis
-Disruption |of |blood |flow
-Infection, |inflammation, |immune |response
-Neoplasms
-Any |growth |will |affect |the |release |of |hormones
Hypersecretion |- |VERIFIED |ANSWER✔✔--TOO |MUCH
-Excessive |stimulation
-Hyperplasia |of |the |gland
-Hormone |producing |tumor |of |the |gland
Hypothalamus |& |Pituitary |Gland |- |VERIFIED |ANSWER✔✔-Regulates |hormone |secretion |from |all |major
|endocrine |organs |except |the |pancreas |and |parathyroid |glands
Hypothalamus |- |VERIFIED |ANSWER✔✔-the |primary |organ |of |the |body |concerned |with |maintaining
|homeostasis; |that |is, |keeping |the |body's |internal |environment |constant. |Sends |several |hormones |to
|anterior |or |posterior |pituitary |gland
,Pituitary |- |VERIFIED |ANSWER✔✔-the |MASTER |GLAND |of |the |body
Hypothalamus |- |VERIFIED |ANSWER✔✔-the |POWER |behind |the |throne
Posterior |Pituitary |- |VERIFIED |ANSWER✔✔-Nerve |cell |bodies |of |the |hypothalamus |synthesize |two
|hormones: |ADH |and |Oxytocin.
These |hormones |are |produced |in |the |hypothalamus |and |stored |in |the |posterior |pituitary |until
|stimulated |- |VERIFIED |ANSWER✔✔-ADH |and |oxytocin
Oxytocin |- |VERIFIED |ANSWER✔✔--protein |hormone |stored |and |released |from |the |posterior |pituitary
-little |effect |in |non-pregnant |women
-increases |intensity |of |labor |- |stimulates |contraction |of |uterine |smooth |muscle
-stimulates |contraction |of |the |smooth |muscle |lining |of |the |milk |ducts |of |the |breast, |causing |letdown
|of |stored |milk |into |nipples
pitocin |- |VERIFIED |ANSWER✔✔-a |synthetic |derivative |of |oxytocin, |it |is |used |to |initiate |and |speed
|labor
ADH |- |VERIFIED |ANSWER✔✔--A |protein |hormone, |stored |and |released |from |the |posterior |pituitary.
-Stimulus: |increased |plasma |osmolality |(concentration |of |solute |in |circulating |blood) |is |sensed |by
|osmoreceptors |in |the |hypothalamus |which |then |stimulates |ADH |release.
Does |ADH |make |you |retain |or |diurese? |- |VERIFIED |ANSWER✔✔-Retain |urine. |Hold |onto |fluid |because
|the |blood |is |too |thick.
What |do |you |retain |with |ADH? |- |VERIFIED |ANSWER✔✔-water
ADH |aids |in |the |control |of |blood |volume |by |acting |on |the |kidneys |to |________ |more |water |back |in
|the |blood |- |VERIFIED |ANSWER✔✔-absorb
, ADH |is |also |referred |to |as |______________ |because |of |its |ability |to |cause |vasoconstriction |if |plasma
|levels |are |extremely |elevated |- |VERIFIED |ANSWER✔✔-vasopressin
Syndrome |of |Inappropriate |ADH |secretion |(SIADH) |- |VERIFIED |ANSWER✔✔-high |levels |of |ADH |without
|normal |physiologic |stimuli |for |its |release
Diabetes |Insipidus |(DI) |- |VERIFIED |ANSWER✔✔-insufficient |secretion |of |antidiuretic |hormone
|(vasopressin)
fluid |is |vascular |space |with |DI |- |VERIFIED |ANSWER✔✔-decreased |(concentrated)
fluid |is |vascular |space |with |SIADH |- |VERIFIED |ANSWER✔✔-increased |(diluted)
sodium |level |in |DI |- |VERIFIED |ANSWER✔✔-hypernatremia
sodium |level |is |SIADH |- |VERIFIED |ANSWER✔✔-hyponatremia
specific |gravity |in |DI |- |VERIFIED |ANSWER✔✔-decreased |(low)
specific |gravity |in |SIADH |- |VERIFIED |ANSWER✔✔-increased |(high)
Brain |cells |in |DI |- |VERIFIED |ANSWER✔✔-shrink
Brain |cells |in |SIADH |- |VERIFIED |ANSWER✔✔-swell
Weight |in |DI |- |VERIFIED |ANSWER✔✔-decreases
Weight |is |SIADH |- |VERIFIED |ANSWER✔✔-increases
With correct Answers
Endocrine |System |- |VERIFIED |ANSWER✔✔-Composed |of |cells |and |organs |that |manufacture |and
|secrete |hormones. |It |is |a |system |of |communication |that |controls |many |life-long |bodily |responses |and
|functions
Thymus |- |VERIFIED |ANSWER✔✔-larger |in |children |while |they |develop |an |immune |system. |It |gets
|smaller |without |use |(as |we |age).
Glands |- |VERIFIED |ANSWER✔✔-may |produce |or |secret |hormone
Hormones |- |VERIFIED |ANSWER✔✔-goes |to |its |target |and |causes |an |action |or |response
Target |cells |- |VERIFIED |ANSWER✔✔-read |and |follow |hormone's |instructions
how |does |the |endocrine |system |respond |to |stimuli? |- |VERIFIED |ANSWER✔✔-by |releasing |hormones
|from |endocrine |glands
Hormones |regulate |four |major |body |functions: |- |VERIFIED |ANSWER✔✔-Reproduction
Growth |and |development
Homeostasis
Metabolism
All |hormones |share |certain |general |characteristics: |- |VERIFIED |ANSWER✔✔--Rates |and |patterns
-Operate |within |a |feedback |system
-Affect |target |cells |with |specific |receptors |and |then |act |to |initiate |specific |cell |functions |or |activities
|(they |respond |only |to |those |hormones |for |which |they |have |receptors)
-Excreted |by |kidneys |or |deactivated |by |the |liver |or |cellular |mechanisms
,Negative |feedback |loop |in |the |endocrine |system |- |VERIFIED |ANSWER✔✔-cold |exposure |à
|hypothalamus |(thyrotropin- |releasing |hormone) |à |anterior |pituitary |(thyrotropin) |à |thyroid |à
|thyroxine. |Then |a |negative |feedback |loop |tells |anterior |pituitary |to |stop |releasing |thyrotropin.
Hyposecretion |- |VERIFIED |ANSWER✔✔-gland |releases |an |inadequate |amount |of |hormone |to |meet
|physiologic |needs; |not |enough |secreted
Hypersecretion |- |VERIFIED |ANSWER✔✔-increased |secretion. |Too |much |secreted
Hyporesponsiveness |- |VERIFIED |ANSWER✔✔-of |the |target |organ |will |cause |the |same |set |of |clinical
|symptoms |as |hyposecretion. |usually |caused |by |deficiency |of |receptors
Hyposecretion |- |VERIFIED |ANSWER✔✔--NOT |ENOUGH
-Congenital
-Absence |of |enzyme |needed |for |synthesis
-Disruption |of |blood |flow
-Infection, |inflammation, |immune |response
-Neoplasms
-Any |growth |will |affect |the |release |of |hormones
Hypersecretion |- |VERIFIED |ANSWER✔✔--TOO |MUCH
-Excessive |stimulation
-Hyperplasia |of |the |gland
-Hormone |producing |tumor |of |the |gland
Hypothalamus |& |Pituitary |Gland |- |VERIFIED |ANSWER✔✔-Regulates |hormone |secretion |from |all |major
|endocrine |organs |except |the |pancreas |and |parathyroid |glands
Hypothalamus |- |VERIFIED |ANSWER✔✔-the |primary |organ |of |the |body |concerned |with |maintaining
|homeostasis; |that |is, |keeping |the |body's |internal |environment |constant. |Sends |several |hormones |to
|anterior |or |posterior |pituitary |gland
,Pituitary |- |VERIFIED |ANSWER✔✔-the |MASTER |GLAND |of |the |body
Hypothalamus |- |VERIFIED |ANSWER✔✔-the |POWER |behind |the |throne
Posterior |Pituitary |- |VERIFIED |ANSWER✔✔-Nerve |cell |bodies |of |the |hypothalamus |synthesize |two
|hormones: |ADH |and |Oxytocin.
These |hormones |are |produced |in |the |hypothalamus |and |stored |in |the |posterior |pituitary |until
|stimulated |- |VERIFIED |ANSWER✔✔-ADH |and |oxytocin
Oxytocin |- |VERIFIED |ANSWER✔✔--protein |hormone |stored |and |released |from |the |posterior |pituitary
-little |effect |in |non-pregnant |women
-increases |intensity |of |labor |- |stimulates |contraction |of |uterine |smooth |muscle
-stimulates |contraction |of |the |smooth |muscle |lining |of |the |milk |ducts |of |the |breast, |causing |letdown
|of |stored |milk |into |nipples
pitocin |- |VERIFIED |ANSWER✔✔-a |synthetic |derivative |of |oxytocin, |it |is |used |to |initiate |and |speed
|labor
ADH |- |VERIFIED |ANSWER✔✔--A |protein |hormone, |stored |and |released |from |the |posterior |pituitary.
-Stimulus: |increased |plasma |osmolality |(concentration |of |solute |in |circulating |blood) |is |sensed |by
|osmoreceptors |in |the |hypothalamus |which |then |stimulates |ADH |release.
Does |ADH |make |you |retain |or |diurese? |- |VERIFIED |ANSWER✔✔-Retain |urine. |Hold |onto |fluid |because
|the |blood |is |too |thick.
What |do |you |retain |with |ADH? |- |VERIFIED |ANSWER✔✔-water
ADH |aids |in |the |control |of |blood |volume |by |acting |on |the |kidneys |to |________ |more |water |back |in
|the |blood |- |VERIFIED |ANSWER✔✔-absorb
, ADH |is |also |referred |to |as |______________ |because |of |its |ability |to |cause |vasoconstriction |if |plasma
|levels |are |extremely |elevated |- |VERIFIED |ANSWER✔✔-vasopressin
Syndrome |of |Inappropriate |ADH |secretion |(SIADH) |- |VERIFIED |ANSWER✔✔-high |levels |of |ADH |without
|normal |physiologic |stimuli |for |its |release
Diabetes |Insipidus |(DI) |- |VERIFIED |ANSWER✔✔-insufficient |secretion |of |antidiuretic |hormone
|(vasopressin)
fluid |is |vascular |space |with |DI |- |VERIFIED |ANSWER✔✔-decreased |(concentrated)
fluid |is |vascular |space |with |SIADH |- |VERIFIED |ANSWER✔✔-increased |(diluted)
sodium |level |in |DI |- |VERIFIED |ANSWER✔✔-hypernatremia
sodium |level |is |SIADH |- |VERIFIED |ANSWER✔✔-hyponatremia
specific |gravity |in |DI |- |VERIFIED |ANSWER✔✔-decreased |(low)
specific |gravity |in |SIADH |- |VERIFIED |ANSWER✔✔-increased |(high)
Brain |cells |in |DI |- |VERIFIED |ANSWER✔✔-shrink
Brain |cells |in |SIADH |- |VERIFIED |ANSWER✔✔-swell
Weight |in |DI |- |VERIFIED |ANSWER✔✔-decreases
Weight |is |SIADH |- |VERIFIED |ANSWER✔✔-increases
