NURS 216 FINAL: Elimination – Questions With Reviewed
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Sherpath: Diabetes Mellitus Chapter 21 Test Case Study 4 (Short
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Practice questions for this set
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Ascending colon
Transverse colon
Descending colon
Sigmoid colon
Choose an answer
1 4 parts of large intestine 2 elimination
3 H+K+-ATPase enzyme 4 enzymes used in digestion
Don't know?
Terms in this set (81)
elimination the excretion of waste products. The passage of stool through
the intestinal tract and dispelling of the stool by means of
intestinal smooth muscle contraction. It usually occurs under
voluntary control
common clinical presentations - acidity
- transition time too slow (constipation)
- transition time too fast (diarrhea)
- N&V
- Liver disease
2 parts of the digestive system - GI tract / alimentary canal
- accessory organs: salivary glands, liver, gallbladder,
pancreas
, where are most substances absorbed? small intestine
enzymes used in digestion stomach HCL + enzymes, pancreatic juice, bile
where does digestion begin? contraction the mucosa
parietal cells have a proton pump that produce and squeeze out HCl acid
gastric juice the combined secretion of the chief and parietal cells
Hcl acid secreted by the parietal cells, helps to breakdown food,
activates pepsinogen and kills microbes
Pepsinogen secreted by the gastric chief cells
- is the inactive form of pepsin that chemically breaks down
proteins
enteroendecrine (G) cells specialized cells that are found within the Gi tract. They
secrete gastric and histamine which in turn stimulate the
secretion of HCl
Histamine (H2) when excreted it stimulates the parietal cells to produce more
HCl
- is a secondary stimuli
H+K+-ATPase enzyme mediates the release of HCl into the stomach through a
proton pump
Why is it important to control HCl acid in the - To prevent common disorders in the upper GI tract:
body? 1. GERD
2. Peptic Ulcer Disease
gastroesophageal reflux disease (GERD) - caused by the acidic stomach contents entering the
esophagus
pathophysiology of GERD 1. Transient weakenings or relaxation of the lower esophageal
sphincter
- patients with decreased motility can increase acid reflux
2. Delayed gastric emptying
- causes mucosal injury
common complications of GERD Esophageal
- Erosion of mucosa
- Ulcerations
Pain
Inflammation
Decreased appetite
GERD treatment Decrease acidity
Avoid irritants
Surgery: Fundoplication
H2 receptor antagonists - work by slowing acid secretion and are often drug of choice
in treating PUD and GERD
MOA: blocks H2 receptors = prevents stimulation for acid
secretion
- INE
(cimetidine, ranitidine, famotidine)
Solutions
Save
Students also studied
Sherpath: Diabetes Mellitus Chapter 21 Test Case Study 4 (Short
30 terms 24 terms 22 terms
jesssbee Preview kenanderson2017 Preview Brittany182
Practice questions for this set
Learn 1 /7 Study using Learn
Ascending colon
Transverse colon
Descending colon
Sigmoid colon
Choose an answer
1 4 parts of large intestine 2 elimination
3 H+K+-ATPase enzyme 4 enzymes used in digestion
Don't know?
Terms in this set (81)
elimination the excretion of waste products. The passage of stool through
the intestinal tract and dispelling of the stool by means of
intestinal smooth muscle contraction. It usually occurs under
voluntary control
common clinical presentations - acidity
- transition time too slow (constipation)
- transition time too fast (diarrhea)
- N&V
- Liver disease
2 parts of the digestive system - GI tract / alimentary canal
- accessory organs: salivary glands, liver, gallbladder,
pancreas
, where are most substances absorbed? small intestine
enzymes used in digestion stomach HCL + enzymes, pancreatic juice, bile
where does digestion begin? contraction the mucosa
parietal cells have a proton pump that produce and squeeze out HCl acid
gastric juice the combined secretion of the chief and parietal cells
Hcl acid secreted by the parietal cells, helps to breakdown food,
activates pepsinogen and kills microbes
Pepsinogen secreted by the gastric chief cells
- is the inactive form of pepsin that chemically breaks down
proteins
enteroendecrine (G) cells specialized cells that are found within the Gi tract. They
secrete gastric and histamine which in turn stimulate the
secretion of HCl
Histamine (H2) when excreted it stimulates the parietal cells to produce more
HCl
- is a secondary stimuli
H+K+-ATPase enzyme mediates the release of HCl into the stomach through a
proton pump
Why is it important to control HCl acid in the - To prevent common disorders in the upper GI tract:
body? 1. GERD
2. Peptic Ulcer Disease
gastroesophageal reflux disease (GERD) - caused by the acidic stomach contents entering the
esophagus
pathophysiology of GERD 1. Transient weakenings or relaxation of the lower esophageal
sphincter
- patients with decreased motility can increase acid reflux
2. Delayed gastric emptying
- causes mucosal injury
common complications of GERD Esophageal
- Erosion of mucosa
- Ulcerations
Pain
Inflammation
Decreased appetite
GERD treatment Decrease acidity
Avoid irritants
Surgery: Fundoplication
H2 receptor antagonists - work by slowing acid secretion and are often drug of choice
in treating PUD and GERD
MOA: blocks H2 receptors = prevents stimulation for acid
secretion
- INE
(cimetidine, ranitidine, famotidine)
