GNRS 515 Patho Exam 1 Questions With
Correct Answers
Cellular |Adaptation |- |CORRECT |ANSWER✔✔-1.Atrophy: |decrease |in |cell |size
2.Hypertrophy: |increase |in |cell |size
3.Hyperplasia: |increase |in |cell |number
4. |Metaplasia: |reversible |replacement |of |one |mature |cell |type |by |another |less |mature |cell |
type
5.Dysplasia |(atypical |hyperplasia): |deranged |cellular |growth |(size, |shape, |and |organization)
Metaplasia |- |CORRECT |ANSWER✔✔-Reversible |replacement |of |one |cell |by |another |cell |type
Bronchial |metaplasia: |(in |smokers)
- |normal |columnar |ciliated |epithelial |cells, |are |replaced |by |stratified |squamous |epithelial |cells |
(no |mucus |secretion, |no |cilia)
- |Can |be |reversed
- |Can |be |transformed |into |cancer
Barrett |esophagus:
- |normal |squamous |epithelial |lining |of |the |esophagus, |is |replaced |by |columnar |epithelium |
(goblet |cells |of |the |intestines)
,- |occurs |as |an |adaptation |to |the |chronic |irritation |of |GERD
- |predisposes |to |esophageal |adenocarcinoma
Non-dividing |cells, |such |as |myocardial |fibers, |are |capable |of |hypertrophy, |but |not |hyperplasia
A.True
B.False |- |CORRECT |ANSWER✔✔-A.True
Dysplasia |is |a |common |type |of |normal |cellular |adaptation
A.True
B.False |- |CORRECT |ANSWER✔✔-B.False
Hypertrophy |and |hyperplasia |rarely |occur |together.
A.True
B.False |- |CORRECT |ANSWER✔✔-B.False
A |man |with |a |history |of |smoking |has |a |bronchial |biopsy |showing |that |the |normal |columnar |
ciliated |epithelial |cells |have |been |replaced |by |stratified |squamous |epithelial |cells. |The |man |is |
correctly |told |that |this |process |could |be |reversed |if |he |quits |smoking
A.True
B.False |- |CORRECT |ANSWER✔✔-A.True
Dysplastic |changes |may |be |reversible, |but |more |commonly |become |malignant.
,A.True
B.False |- |CORRECT |ANSWER✔✔-A.True
Which |type |of |cell |adaptation |occurs |when |normal |columnar |ciliated |epithelial |cells |of |the |
bronchial |lining |have |been |replaced |by |stratified |squamous |epithelial |cells?
A.Hyperplasia
B.Metaplasia
C.Dysplasia
D.Anaplasia |- |CORRECT |ANSWER✔✔-B.Metaplasia
The |mammary |glands |enlarge |during |pregnancy |primarily |as |a |consequence |of:
A.compensatory |hyperplasia
B.hormonal |hyperplasia
C.hormonal |anaplasia
D.hormonal |dysplasia |- |CORRECT |ANSWER✔✔-B.hormonal |hyperplasia
When |the |heart's |workload |increases, |what |changes |occur |to |the |myocardial |cells?
A.They |divide
B.They |increase |in |size
C.They |increase |in |number
D.They |undergo |metaplasia |- |CORRECT |ANSWER✔✔-B.They |increase |in |size
After |ovulation, |the |uterine |endometrial |cells |divide |under |the |influence |of |estrogen; |this |is |an
|example |of |hormonal:
, A.hyperplasia.
B.dysplasia.
C.Hypertrophy
D.Anaplasia |- |CORRECT |ANSWER✔✔-A.hyperplasia.
The |abnormal |proliferation |of |cells |in |response |to |excessive |hormonal |stimulation |is |called:
A.dysplasia.
B.pathologic |dysplasia
C.Hyperplasia
D.pathologic |hyperplasia |- |CORRECT |ANSWER✔✔-D.pathologic |hyperplasia
Removal |of |part |of |the |liver |leads |to |______________ |of |the |remaining |liver |cells
A.dysplasia
B.metaplasia
C.compensatory |hyperplasia
D.compensatory |dysplasia |- |CORRECT |ANSWER✔✔-C.compensatory |hyperplasia
Manifestations |of |cellular |injury |- |CORRECT |ANSWER✔✔-Water
-Cause: |Hypoxia, |reduced |ATP, |increased |intracellular |Na
-Ex: |Early |in |cell |injury
Lipids
-Cause: |Imbalance |in |production, |utilization, |or |mobilization |of |lipids
Correct Answers
Cellular |Adaptation |- |CORRECT |ANSWER✔✔-1.Atrophy: |decrease |in |cell |size
2.Hypertrophy: |increase |in |cell |size
3.Hyperplasia: |increase |in |cell |number
4. |Metaplasia: |reversible |replacement |of |one |mature |cell |type |by |another |less |mature |cell |
type
5.Dysplasia |(atypical |hyperplasia): |deranged |cellular |growth |(size, |shape, |and |organization)
Metaplasia |- |CORRECT |ANSWER✔✔-Reversible |replacement |of |one |cell |by |another |cell |type
Bronchial |metaplasia: |(in |smokers)
- |normal |columnar |ciliated |epithelial |cells, |are |replaced |by |stratified |squamous |epithelial |cells |
(no |mucus |secretion, |no |cilia)
- |Can |be |reversed
- |Can |be |transformed |into |cancer
Barrett |esophagus:
- |normal |squamous |epithelial |lining |of |the |esophagus, |is |replaced |by |columnar |epithelium |
(goblet |cells |of |the |intestines)
,- |occurs |as |an |adaptation |to |the |chronic |irritation |of |GERD
- |predisposes |to |esophageal |adenocarcinoma
Non-dividing |cells, |such |as |myocardial |fibers, |are |capable |of |hypertrophy, |but |not |hyperplasia
A.True
B.False |- |CORRECT |ANSWER✔✔-A.True
Dysplasia |is |a |common |type |of |normal |cellular |adaptation
A.True
B.False |- |CORRECT |ANSWER✔✔-B.False
Hypertrophy |and |hyperplasia |rarely |occur |together.
A.True
B.False |- |CORRECT |ANSWER✔✔-B.False
A |man |with |a |history |of |smoking |has |a |bronchial |biopsy |showing |that |the |normal |columnar |
ciliated |epithelial |cells |have |been |replaced |by |stratified |squamous |epithelial |cells. |The |man |is |
correctly |told |that |this |process |could |be |reversed |if |he |quits |smoking
A.True
B.False |- |CORRECT |ANSWER✔✔-A.True
Dysplastic |changes |may |be |reversible, |but |more |commonly |become |malignant.
,A.True
B.False |- |CORRECT |ANSWER✔✔-A.True
Which |type |of |cell |adaptation |occurs |when |normal |columnar |ciliated |epithelial |cells |of |the |
bronchial |lining |have |been |replaced |by |stratified |squamous |epithelial |cells?
A.Hyperplasia
B.Metaplasia
C.Dysplasia
D.Anaplasia |- |CORRECT |ANSWER✔✔-B.Metaplasia
The |mammary |glands |enlarge |during |pregnancy |primarily |as |a |consequence |of:
A.compensatory |hyperplasia
B.hormonal |hyperplasia
C.hormonal |anaplasia
D.hormonal |dysplasia |- |CORRECT |ANSWER✔✔-B.hormonal |hyperplasia
When |the |heart's |workload |increases, |what |changes |occur |to |the |myocardial |cells?
A.They |divide
B.They |increase |in |size
C.They |increase |in |number
D.They |undergo |metaplasia |- |CORRECT |ANSWER✔✔-B.They |increase |in |size
After |ovulation, |the |uterine |endometrial |cells |divide |under |the |influence |of |estrogen; |this |is |an
|example |of |hormonal:
, A.hyperplasia.
B.dysplasia.
C.Hypertrophy
D.Anaplasia |- |CORRECT |ANSWER✔✔-A.hyperplasia.
The |abnormal |proliferation |of |cells |in |response |to |excessive |hormonal |stimulation |is |called:
A.dysplasia.
B.pathologic |dysplasia
C.Hyperplasia
D.pathologic |hyperplasia |- |CORRECT |ANSWER✔✔-D.pathologic |hyperplasia
Removal |of |part |of |the |liver |leads |to |______________ |of |the |remaining |liver |cells
A.dysplasia
B.metaplasia
C.compensatory |hyperplasia
D.compensatory |dysplasia |- |CORRECT |ANSWER✔✔-C.compensatory |hyperplasia
Manifestations |of |cellular |injury |- |CORRECT |ANSWER✔✔-Water
-Cause: |Hypoxia, |reduced |ATP, |increased |intracellular |Na
-Ex: |Early |in |cell |injury
Lipids
-Cause: |Imbalance |in |production, |utilization, |or |mobilization |of |lipids