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NSG 3280 Unit 3 & 4 Study Guide/NSG 3280 Unit 3 & 4: Updated A+ Guide Solution

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Primary vs. Secondary Skin Lesions • Primary – lesions as they originally appear • Secondary – lesions that have been modified from their original appearance Herpes Simplex Virus 1 & 2 Pathophysiology • Herpes virus lives latent in nerve (most often the trigeminal) after initial infection and reactivates during: o Stress o Sun exposure o Menstruation Clinical Manifestations • Burning → redness & vesicles → pustules & ulcers → crust • Heals in 10-14 days • Herpes Simplex 1common locations: Lips, face, and mouth • Herpes Simplex 2 common location: Genital region Herpes Zoster Pathophysiology • Virus lives latent in the spinal ganglia and activates during stress. Clinical Manifestations • Pain at areas of vesicles • Vesicles with a red base (continue to pop up for 3-5 days) that follow the dermatome (nerve track) → vesicles dry out & crust→ fall off • Whole process takes 2-3 weeks • Vesicles are unilateral; usually on face and trunk • In older adults: pain is worse; duration is longer Complication • Permanent blindness can occur if lesions involve the eyes. • Post-herpetic neuralgia – pain that continues after skin lesions have cleared o Can last months to years o More common in those 60 years oldSuperficial Fungus Pathophysiology & Epidemiology • Infections by these fungi: o Microsporum o Trichophyton o Epidermophyton • These infections are called “tinea” followed by the skin region they are infecting o Tinea captitis = scalp o Tinea barbae = beard o Tinea faciei = face o Tinea corporis = trunk o Tinea manus = hand o Tinea cruris = groin o Tinea pedis = foot Clinical Manifestations • Red macules or plaques • Feet may have vesicles • Peripheral scaling and central clearing Yeast Infections Pathophysiology & Etiology • Caused by the fungus candida albicans • Fungal infection of external tissue Clinical Manifestations • Mouth – thrush • Skin folds – intertrigo • Systemic – mucocutaneous candidiasis Impetigo Pathophysiology & Etiology • Bacterial skin infection caused by staphylococcus & streptococcus • Staph is often carried in nasal area • Very contagious Clinical Manifestations • Vesicles, pustules, and yellow crustsSeborrheic Dermatitis Clinical Manifestations • Considered “papulosquamous” - red or purple plaques with scales • Occur in areas with oil glands • Newborns: “cradle cap” • Adolescents & Adults: “dandruff”

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NSG 3280 UNIT 3 & 4 Study Guide
Primary vs. Secondary Skin Lesions

• Primary – lesions as they originally appear
• Secondary – lesions that have been modified from their original appearance


Herpes Simplex Virus 1 & 2
Pathophysiology

• Herpes virus lives latent in nerve (most often the trigeminal) after initial infection and
reactivates during: o Stress o Sun exposure o Menstruation

Clinical Manifestations

• Burning → redness & vesicles → pustules & ulcers → crust
• Heals in 10-14 days
• Herpes Simplex 1common locations: Lips, face, and mouth
• Herpes Simplex 2 common location: Genital region

Herpes Zoster
Pathophysiology

• Virus lives latent in the spinal ganglia and activates during stress.

Clinical Manifestations

• Pain at areas of vesicles
• Vesicles with a red base (continue to pop up for 3-5 days) that follow the dermatome
(nerve track) → vesicles dry out & crust→ fall off
• Whole process takes 2-3 weeks
• Vesicles are unilateral; usually on face and trunk
• In older adults: pain is worse; duration is longer

Complication

• Permanent blindness can occur if lesions involve the eyes.
• Post-herpetic neuralgia – pain that continues after skin lesions have cleared o Can
last months to years
o More common in those > 60 years old

, Superficial Fungus
Pathophysiology & Epidemiology

• Infections by these fungi: o Microsporum o
Trichophyton o Epidermophyton
• These infections are called “tinea” followed by the skin
region they are infecting o Tinea captitis = scalp o
Tinea barbae = beard o Tinea faciei = face o Tinea
corporis = trunk o Tinea manus = hand o Tinea cruris =
groin o Tinea pedis = foot

Clinical Manifestations

• Red macules or plaques
• Feet may have vesicles
• Peripheral scaling and central clearing

Yeast Infections
Pathophysiology & Etiology

• Caused by the fungus candida albicans
• Fungal infection of external tissue

Clinical Manifestations

• Mouth – thrush
• Skin folds – intertrigo
• Systemic – mucocutaneous candidiasis

Impetigo
Pathophysiology & Etiology

• Bacterial skin infection caused by staphylococcus & streptococcus
• Staph is often carried in nasal area
• Very contagious
Clinical Manifestations

• Vesicles, pustules, and yellow crusts

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