10/30/25, 1:44 PM Bio253 Final Exam Flashcards | Quizlet
Science Biology Physiology
Bio253 Final Exam
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Medical Physiology Exam 3: Digesti... A&P1 Chapter 1 Musculoskeletal System NPTE Homeo
292 terms 46 terms 39 terms 18 terms
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Practice questions for this set
Learn 1 /7 Study with Learn
The veins ability to rapidly increase its volume capacity at lower pressures explains why
veins are known as capacitance vessels.
Veins are called capacitance vessels because they can hold a large amount of blood.
Choose an answer
1 Blood Volume 2 Capacitance
Blood Cannot Flow From Point A To Point B
3 Blood Pressure 4 In A Vessel Unless The Pressure Is Higher At
A Than At B.
Don't know?
Terms in this set (174)
Lab 1: EMG and Muscular Conditions ...
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, 10/30/25, 1:44 PM Bio253 Final Exam Flashcards | Quizlet
A recording of the electrical activity of skeletal muscle at rest and during
Ekectromyography (EMG)
contraction.
Intramuscular EMG Needle electrodes are inserted through the skin into the muscle.
Surface EMG Electrodes are placed on the skin's surface.
A neuron and the muscle fibers it controls. Motor unit recruitment is one way to
A motor unit
increase contractile strength of a whole muscle.
a phenomenon is which contraction of an agonist muscle leads to minor activity in
Co-activation the antagonist muscle. Coordination between agonist and antagonist muscles are
essential for a range of movement. It helps to stabilize the joint.
signals are produced by the electrical stimulation of a motor nerve supplying a
Evoked EMG muscle. muscle contraction are initiated by an action potential in the motor nerve
fibers innervating the muscle.
sometimes called "prime movers" as they are the primary muscle responsible for
Agonist muscles
generating movement through their contraction.
Antagonist muscles control (or oppose) specific movement, ad return the muscle to its initial position.
Muscle Fatigue ...
Adenosine thriphosphate (ATP) can be remade from adenosine diphosphate (ADP)
by the addition of a phosphate group- this can occur by three pathways:
1. When enough O2 is present, pyruvate (from the breakdown of fats, glycogen or
glucose) can enter the citric acid cycle and is broken down to CO2 and H2O. This is
known as aerobic glycolysis and it generates large amounts of ATP through oxidative
phosphorylation (the electron transport chain).
Psychological causes of fatigue 2. If O2 supplies are insufficient, pyruvate cannot enter the citric acid cycle and
instead gets converted to lactic acid in a process called anaerobic glycolysis. This
makes small amount of ATP but does not require O2.
3. In resting muscle, some ATP transfers a phosphate to creatine obtained through
the circulation from the liver, creating a store of phosphocreatine. During intense
exercise, phosphocreatine can be used to resynthesize ATP, allowing contraction to
continue.
When ATP production fails to keep pace with ATP usage, muscles contract less and
less effectively.
Excessive accumulation of lactic acid and associated H+, together with iconic
imbalances also contribute to muscle fatigue. The pH drop associated with the lactic
Physiological causes of fatigue acid production may cause fatigue and limits the usefulness of anaerobic mechanism
for ATP production.
Some specialized muscle fibers are more resistant to fatigue than others. Fore
example, muscles may have more mitochondria and hence a greater capacity for
oxidative metabolism, or they may have greater stores of phosphocreatine.
- Changes in one's motivation or sense of effort
- Decline in central nervous system command of motor units (central drive).
- Failure of neuromuscular propagation. Neurons may temporarily run low on
Factors that have been proposed to neurotransmitter.
explain muscle fatigue: - Reduction in Ca2+ release in excitation-contraction coupling.
- Metabolic changes in the muscle cell (such as build-up of lactic acid which can
make the skeletal muscle acidic inhibiting any futher anaerobic glycolysis).
- Reduction in muscle blood flow owing to compression of blood vessels.
In skeletal muscle, ATP is not generated lactic acid metabolism
through:
https://quizlet.com/906316346/bio253-final-exam-flash-cards/ 2/8
Science Biology Physiology
Bio253 Final Exam
Leave the first rating
Save
Students also studied
Medical Physiology Exam 3: Digesti... A&P1 Chapter 1 Musculoskeletal System NPTE Homeo
292 terms 46 terms 39 terms 18 terms
brinka2 Preview Melissa_Mellion1 Preview quizlette411183736 Preview MM
Practice questions for this set
Learn 1 /7 Study with Learn
The veins ability to rapidly increase its volume capacity at lower pressures explains why
veins are known as capacitance vessels.
Veins are called capacitance vessels because they can hold a large amount of blood.
Choose an answer
1 Blood Volume 2 Capacitance
Blood Cannot Flow From Point A To Point B
3 Blood Pressure 4 In A Vessel Unless The Pressure Is Higher At
A Than At B.
Don't know?
Terms in this set (174)
Lab 1: EMG and Muscular Conditions ...
https://quizlet.com/906316346/bio253-final-exam-flash-cards/ 1/8
, 10/30/25, 1:44 PM Bio253 Final Exam Flashcards | Quizlet
A recording of the electrical activity of skeletal muscle at rest and during
Ekectromyography (EMG)
contraction.
Intramuscular EMG Needle electrodes are inserted through the skin into the muscle.
Surface EMG Electrodes are placed on the skin's surface.
A neuron and the muscle fibers it controls. Motor unit recruitment is one way to
A motor unit
increase contractile strength of a whole muscle.
a phenomenon is which contraction of an agonist muscle leads to minor activity in
Co-activation the antagonist muscle. Coordination between agonist and antagonist muscles are
essential for a range of movement. It helps to stabilize the joint.
signals are produced by the electrical stimulation of a motor nerve supplying a
Evoked EMG muscle. muscle contraction are initiated by an action potential in the motor nerve
fibers innervating the muscle.
sometimes called "prime movers" as they are the primary muscle responsible for
Agonist muscles
generating movement through their contraction.
Antagonist muscles control (or oppose) specific movement, ad return the muscle to its initial position.
Muscle Fatigue ...
Adenosine thriphosphate (ATP) can be remade from adenosine diphosphate (ADP)
by the addition of a phosphate group- this can occur by three pathways:
1. When enough O2 is present, pyruvate (from the breakdown of fats, glycogen or
glucose) can enter the citric acid cycle and is broken down to CO2 and H2O. This is
known as aerobic glycolysis and it generates large amounts of ATP through oxidative
phosphorylation (the electron transport chain).
Psychological causes of fatigue 2. If O2 supplies are insufficient, pyruvate cannot enter the citric acid cycle and
instead gets converted to lactic acid in a process called anaerobic glycolysis. This
makes small amount of ATP but does not require O2.
3. In resting muscle, some ATP transfers a phosphate to creatine obtained through
the circulation from the liver, creating a store of phosphocreatine. During intense
exercise, phosphocreatine can be used to resynthesize ATP, allowing contraction to
continue.
When ATP production fails to keep pace with ATP usage, muscles contract less and
less effectively.
Excessive accumulation of lactic acid and associated H+, together with iconic
imbalances also contribute to muscle fatigue. The pH drop associated with the lactic
Physiological causes of fatigue acid production may cause fatigue and limits the usefulness of anaerobic mechanism
for ATP production.
Some specialized muscle fibers are more resistant to fatigue than others. Fore
example, muscles may have more mitochondria and hence a greater capacity for
oxidative metabolism, or they may have greater stores of phosphocreatine.
- Changes in one's motivation or sense of effort
- Decline in central nervous system command of motor units (central drive).
- Failure of neuromuscular propagation. Neurons may temporarily run low on
Factors that have been proposed to neurotransmitter.
explain muscle fatigue: - Reduction in Ca2+ release in excitation-contraction coupling.
- Metabolic changes in the muscle cell (such as build-up of lactic acid which can
make the skeletal muscle acidic inhibiting any futher anaerobic glycolysis).
- Reduction in muscle blood flow owing to compression of blood vessels.
In skeletal muscle, ATP is not generated lactic acid metabolism
through:
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