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NURS 5338 UNIVERSITY OF TEXAS AND ARLINGTON EXAM YEAR 2024/2025 / ACCURATE CURRENTLY TESTING EXAM VERSIONS WITH ACTUAL QUESTIONS AND DETAILED ANSWERS WITH A STUDY GUIDE / EXPERT VERIFIED FOR GURANTEED PASS/ LATEST UPDATE.

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NURS 5338 UNIVERSITY OF TEXAS AND ARLINGTON EXAM YEAR 2024/2025 / ACCURATE CURRENTLY TESTING EXAM VERSIONS WITH ACTUAL QUESTIONS AND DETAILED ANSWERS WITH A STUDY GUIDE / EXPERT VERIFIED FOR GURANTEED PASS/ LATEST UPDATE.

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NURS 5338 UNIVERSITY OF TEXAS AND ARLINGTON EXAM YEAR ACCURATE CURRENTLY
TESTING EXAM VERSIONS WITH ACTUAL QUESTIONS AND DETAILED ANSWERS WITH A
STUDY GUIDE / EXPERT VERIFIED FOR GURANTEED PASS/ LATEST UPDATE.





Atrophy - (answer)E. Cells decrease in size

P. Still functional

Physiologic: thymus gland in early childhood

Pathological: disuse



Hypertrophy - (answer)E. Increase in cell size

P. Increased workload

Physiologic: weightlifting

Pathologic: cardiomegaly from HTN



Hyperplasia - (answer)E. Increase in cell number

P. Increased cellular division

Physiologic: liver regeneration

Pathologic: endometrial- usually r/t hormones



Dysplasia - (answer)E. Cells change in size, shape, organization

P. AKA atypical hyperplasia, a disorderly proliferation

Physiologic: N/A

Pathologic: squamous dysplasia of cervix from HPV



Metaplasia - (answer)E. one cell type replaced with another

P. reprogramming of stem cells, reversible

Physiologic: N/A

Pathologic: stratified squamous cells in bronchial lining r/t cigarette smoke



Hypoxia injury - (answer)E. inadequate oxygenation of tissues

, NURS 5338 UNIVERSITY OF TEXAS AND ARLINGTON EXAM YEAR ACCURATE CURRENTLY
TESTING EXAM VERSIONS WITH ACTUAL QUESTIONS AND DETAILED ANSWERS WITH A
STUDY GUIDE / EXPERT VERIFIED FOR GURANTEED PASS/ LATEST UPDATE.





P. decrease in mitochondrial function, decreased production of ATP increases anaerobic metabolism.
eventual cell death.

C.M. hypoxia, cyanosis, cognitive impairment, lethargy



Free radical and ROS - (answer)E. normal byproduct of ATP production, will overwhelm the
mitochondria- exhaust intracellular antioxidants

P. lipid peroxidation, damage proteins, fragment DNA

C.M. development in Alzheimer's, heart disease, Parkinson's disease, Amyotrophic Lateral Sclerosis



Ethanol - (answer)E. mood altering drug, long term effects on liver and nutritional status

P. metabolized by liver, generates free radicals

C.M. CNS depression, nutrient deficiencies-Mag, Vit B6, thiamine, PO4, inflammation and fatty
infiltration of liver, hepatomegaly, leads to liver failure irreversible



Oncosis - (answer)Na and H2O enter cell and cause swelling. Organ increases in weight, becomes
distended and pale. Associated with high fever, hypocalcemia, certain infections



Fatty Infiltration - (answer)intracellular accumulation of lipids in the liver

liver fails to metabolize lipids. usually from ETOH or high fat diet. can lead to cirrhosis



dystrophic calcification - (answer)accumulation of Ca in dead or dying tissues

calcium salt clump and harden- interfere with cellular structure and function

r/t pulmonary TB, atherosclerosis, injured heart valves, chronic pancreatitis



metastatic calcification - (answer)accumulation of Ca in normal tissue

result of hypercalcemia r/t hyperparathyroidism, hyperthyroidism, toxic levels of Vit D. Can also r/t
hyperphosphatemia in renal failure

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