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NUR 327 Final Exam Questions With Correct Answers

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NUR 327 Final Exam Questions With Correct Answers

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NUR 327 Final Exam Questions With Correct
Answers

1. Interpret the components of an arterial blood gas and the normal values for
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each component.: pH = pH of the blood
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PaCO2 = Respiratory | |


HCO3 = Metabolic
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pH 7.35 - 7.45
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PaCO2 35 - 45 | | |


HCO3 22 - 26 | | |




What would I expect to happen if something else is abnormal?
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2. Evaluate arterial blood gas results for uncompensated, partially compensat-
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ed, and fully compensated acid base imbalances.: pH: 7.18 PaCO2: 53 HCO3: 25
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pH: 7.30 PaCO2: 30 HCO3: 18
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****know how to identify based on scenario s/s**** | | | | | | |


3. Respiratory Alkalosis: Cause: | |


Temperature increased in body (fever) | | | |


Aspirin toxicity
| |


Controlledmechanical ventilation | |


Hyperventilation
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hYsteria (anxiety) |


Pain / Pregnancy / Pneumonia Neurologic
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injuries (head injury/stroke) Embolism &
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Edema
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Asthma due to hyperventilation | | |




Manifestations:
- Fast RR >20 | |


- neuro changes: confused, tired, elevated HR
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- Tetany
- EKG changes |


- muscle cramps |



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,- positive Chvostek's sign (twitch of facial muscles after tapping facial nerve)
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Tx:
- Teach breathing techniques to slow breathing
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,- watch K and Ca levels (hypo) | | | | |


- low potassium levels | |


- Watch for hyperventilation in ventilated patients
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Nursing care: |


- watch K and Ca levels | | | |


- Watch for hyperventilation in ventilated patients
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4. Compare the clinical manifestations, causes, treatments, and nursing care | | | | | | | |


for clients experiencing acid base imbalances including respiratory acidosis,
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respiratory alkalosis, metabolic acidosis, and metabolic alkalosis.: *What do I
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expect to see vs what is a complication*
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- at risk for metabolic acidosis to now they have acidosis
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-- how is the body going to compensate (RR incrs)
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DIC - might expect | | |


Vasooclusive crisis - might expect | | | |


Seizure - might expect respiratory acidosis UC | | | | | |


- might expect metabolic acidosis
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5. Respiratory Acidosis: Causes: | |


anything that interferes with respiratory function and retention of CO2
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- bronchoconstriction
- bacterial pneumonia |


- Pulmonary embolism (PE) | |


Drugs
|


Edema
Pneumonia
|


Respiratory center of brain is damaged | | | | |


Emboli
|


Spasms of brachial tubules | | |


Sac elasticity of alveoli sac is damaged (COPD or Emphysema)
| | | | | | | | |




Manifestations:
- confusion, disorientation, decreased level of consciousness, | | | | |


- drowsy
- HA
- Respiratory depression <12 | |


- low BP |


- increased ICP (severe) | |




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, Tx:
- improve ventilation *Breath faster* | | |


- pulmonary hygiene: increased fluid intake, and ambulation | | | | | |


- admin O2 |


- encourage coughing and deep breathing (if awake) | | | | | |


- respiratory Tx if indicated | | |


- hold narcotics/sedatives
|


- watch potassium levels | |


- administer antibiotics (if indicated) | | |


- May need ET tube if pt in resp distress
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Nursing care: |


- monitor for hyperkalemia | |


6. Metabolic Acidosis: Causes: | |


anything that increases H+ or decreased HCO3 | | | | | |


- renal impairment |


- diabetes
- starvation
- prolonged diarrhea |


Accumulation of lactate leading to lactic acidosis (sepsis) | | | | | | |


Chronic diarrhea (most common)
| | | |


Impaired renal function (most common) | | | |


Diabetic ketoacidosis (DKA)
| | |


|Salicylates toxicity |




Manifestations:
Neurological changes
| |


HA
|


fatigue
tachypne
|


a low BP | |


cold clammy skin
| |


vomiting
|


nausea
increased respiratory rate Kussmaul | | |


breathing - fast and deep confused
| | | | | |


weak
ECG - T waves (hyperkalemia)
| | | |




4 |/ |49

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