Women's Health Care, 12th Edition Exam.
ANS: C
1. 1. In planning for home care
Prolonged bed rest may cause adverse ettects such as
of a woman with preterm la-
weight loss, loss of appetite, muscle wasting, weakness,
bor, which concern should the
bone demineralization, decreased cardiac output, risk for
nurse need to address? a. Nurs-
thrombophlebitis, alteration in bowel functions, sleep dis-
ing assessments are different
turbance, and prolonged postpartum recovery. Nursing as-
from those performed in the
sessments ditter somewhat from those performed in the
hospital setting.
acute care setting, but this concern does not need to be
b. Restricted activity and med-
addressed. Restricted activity and medications may prevent
ications are necessary to pre-
preterm labor but not in all women. In addition, the plan
vent a recurrence of preterm la-
of care is individualized to meet the needs of each client.
bor.
c. Prolonged bed rest may cause Many women receive home health nurse visits, but care is
negative physiologic effects. individualized for each woman.
d. Home health care providers
are necessary.
2. 2. Which nursing intervention is ANS: B
paramount when providing care Terbutaline is a beta2-adrenergic agonist that attects the
to a client with preterm labor mother's cardiopulmonary and metabolic systems. Signs
who has received terbutaline? of cardiopulmonary decompensation include adventitious
a. Assess deep tendon reflexes breath sounds and dyspnea. An assessment for dyspnea
(DTRs). and crackles is important for the nurse to perform if the
b. Assess for dyspnea and crack- woman is taking magnesium sulfate. Assessing DTRs does
les.
c. Assess for bradycardia. 3. 3. In evaluating the effective- ness
d. Assess for hypoglycemia. of magnesium sulfate for
, Chapter 32: Labor and Birth Complications Lowdermilk: Maternity &
Women's Health Care, 12th Edition Exam.
not address the possible respiratory side
ettects of us- ing terbutaline. Since
terbutaline is a beta2-adrenergic agonist, it
can lead to hyperglycemia, not hypoglycemia.
Beta2-adrenergic agonist drugs cause
tachycardia, not bradycardia.
ANS: D
The therapeutic range for magnesium sulfate
management
, Chapter 32: Labor and Birth Complications Lowdermilk: Maternity &
Women's Health Care, 12th Edition Exam.
the treatment of preterm labor, is 4 to 7.5 mg/dl. A serum magnesium level of 10 mg/dl
which finding alerts the nurse to could lead to signs and symptoms of magnesium toxicity,
possible side effects? including oliguria and respiratory distress. Urine output of
a. Urine output of 160 ml in 4 160 ml in 4 hours, DTRs of 2+, and a RR of 16 breaths per
hours minute are all normal findings
b. DTRs 2+ and no clonus
c. Respiratory rate (RR) of 16
breaths per minute
d. Serum magnesium level of 10
mg/dl
4. 4. A woman in preterm labor at ANS: A
30 weeks of gestation receives Antenatal glucocorticoids administered as IM injections to
two 12-mg intramuscular (IM) the mother accelerate fetal lung maturity. Propranolol (In-
doses of betamethasone. What deral) is given to reduce the ettects of ritodrine administra-
is the purpose of this pharmaco- tion. Betamethasone has no ettect on uterine contractions.
logic intervention? Calcium gluconate is given to reverse the respiratory depres-
a. To stimulate fetal surfactant sive ettects of magnesium sulfate therapy.
production
b. To reduce maternal and fetal
tachycardia associated with rito-
drine administration
c. To suppress uterine contrac-
tions
d. To maintain adequate mater-
nal respiratory effort and ven-
tilation during magnesium sul-
fate therapy
5. 5. A primigravida at 40 weeks of ANS: C
gestation is having uterine con- The contraction pattern observed in this woman signifies