Maryville NURS 611 Exam 3 | UPDATE|COMPREHENSIVE QUESTIONS
Maryville NURS 611 Exam 3
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1. What are clinical manifestations of Constipation, decreased heat rate, and lethargy
hypothyroidism?
2. Thyroid-stimulating hormone Negative feedback. Feedback systems provide precise
(TSH) is released to stimulate thy- monitoring and control of the cellular environment.
roid hormone (TH) and is inhibited Negative feedback occurs because the changing chemi-
when plasma levels of TH are ade- cal, neural, or endocrine response to a stimulus negates
quate. This is an example of: the initiating change that triggered the release of the
hormone.
3. Lipid-soluble hormone receptors Inside the plasma membrane in the cytoplasm.
are located:
4. The releasing hormones that are Portal hypophyseal blood vessels.
made in the hypothalamus travel
to the anterior pituitary via the:
5. Which mineral is needed for thy- Iodide.
roid-stimulating hormone (TSH) to
stimulate the secretion of thyroid
hormone (TH)?
6. What effect does hyperphos- Decreases serum calcium.
phatemia have on other elec-
trolytes?
7. Insulin transports which elec- Potassium.
trolyte in the cell?
8. Which second messenger is stim- Cyclic adenosine monophosphate (cAMP). Sec-
ulated by epinephrine binding to a ond-messenger molecules are the initial link between
β-adrenergic receptor? the first signal (hormone) and the inside of the cell.
For example, the binding of epinephrine to a β adren-
, Maryville NURS 611 Exam 3
Study online at https://quizlet.com/_h357ma
ergic-receptor subtype activates (through a stimulatory
G protein) the enzyme, adenylyl cyclase. Adenylyl cy-
clase catalyzes the conversion of adenosine triphosphate
(ATP) to the second messenger, 3', and 5'-cAMP.
9. Regulation of the release of cat- Neural. The release of hormones occurs either in re-
echolamines from the adrenal sponse to an alteration in the cellular environment
medulla is an example of which or in the process of maintaining a regulated level of
type of regulation? certain hormones or certain substances. Several dif-
ferent mechanisms, one of which is neural control
(e.g., stress-induced release of catecholamines from the
adrenal medulla), regulate the release of hormones.
10. Which hormone does the second Angiotensin II. Ca++ is considered an important second
messenger calcium (Ca++) bind to messenger that facilitates the binding of a hormone
activate phospholipase C through (e.g., norepinephrine, angiotensin II) to a surface re-
a G protein? ceptor, activating the enzyme phospholipase C through
a G protein inside the plasma membrane.
11. The control of calcium in cells is Acts as a second messenger. In addition to being an
important because it: important ion that participates in a multitude of cellular
actions, Ca++ is considered an important second mes-
senger.
12. Where is antidiuretic hormone Hypothalamus; renal tubular cells. Once synthesized in
(ADH) synthesized, and where the hypothalamus, ADH acts on the vasopressin 2 (V2)
does it act? receptors of the renal duct cells to increase their perme-
ability.
13. How does a faulty negative-feed- Excessive hormone production results from a failure to
back mechanism result in a hor- turn off the system. Negative- feedback systems are
monal imbalance? important in maintaining hormones within physiolog-
ic ranges. The lack of negative-feedback inhibition on
, Maryville NURS 611 Exam 3
Study online at https://quizlet.com/_h357ma
hormonal release often results in pathologic conditions.
Excessive hormone production, which is the result of
the failure to turn off the system, can cause various
hormonal imbalances and related conditions.
14. A deficiency of which chemical Iodine. The only cause of hypothyroidism from among
may result in hypothyroidism? the provided options is a deficiency of endemic iodine.
15. What imbalance lessens the rate The overall effect of parathyroid hormone (PTH)is to in-
of secretion of parathyroid hor- crease serum calcium and to decrease serum phosphate
mone concentration.
16. Which condition may result from Hypothyroidism. If the tumor exerts sufficient pressure,
pressure exerted by a pituitary tu- then thyroid and adrenal hypofunction may occur be-
mor? cause of lack of thyroid-stimulating hormone (TSH) and
adrenocorticotropic hormone (ACTH). These result in the
symptoms of hypothyroidism and hypocortisolism.
17. Which substance is a water-solu- Insulin. Peptide or protein hormones, such as insulin,
ble protein hormone? pituitary, hypothalamic, and parathyroid, are water sol-
uble and circulate in free (unbound) forms.
18. Which of the following is a Cortisol. Cortisol and adrenal androgens are lipid-solu-
lipid-soluble hormone? ble hormones and are primarily bound to a carrier or
transport protein in circulation.
19. Most protein hormones are trans- Free in an unbound, water-soluble form. Peptide or pro-
ported in the bloodstream and tein hormones, such as insulin, pituitary, hypothalamic,
are: and parathyroid, are water soluble and circulate in free
(unbound) forms.
20. When insulin binds its receptors
on muscle cells, an increase in glu-
, Maryville NURS 611 Exam 3
Study online at https://quizlet.com/_h357ma
cose uptake by the muscle cells is Direct. Direct effects are the obvious changes in cell
the result. This is an example of function that specifically result from the stimulation by
what type of effect by a hormone? a particular hormone.
21. Graves disease develops from Thyroid-stimulating immunoglobulin that causes the
a(n): overproduction of thyroid hormones. The pathologic
features of Graves disease indicates that normal reg-
ulatory mechanisms are overridden by abnormal im-
munologic mechanisms that result in the stimulation of
excessive TH.
22. Pathologic changes associated High levels of circulating thyroid-stimulating im-
with Graves disease include: munoglobulins. The only option that correctly describes
the changes associated with Graves disease identi-
fies high levels of circulating thyroid-stimulating im-
munoglobulins that are found in more than 95% of
individuals diagnosed with the disease.
23. The signs of thyrotoxic crisis in- Hyperthermia and tachycardia
clude:
24. The level of thyroid-stimulating Low. The hyperfunction of the thyroid gland leads to
hormone (TSH) in individuals with suppression of TSH because of the normal negative
Graves disease is usually: feedback mechanism.
25. Palpation of the neck of a per- Diffusely enlarged.
son diagnosed with Graves dis-
ease would detect a thyroid that
is:
26. The effects of the syndrome of in- Dilution & water retention
appropriate antidiuretic hormone
(SIADH) secretion include solute:
Maryville NURS 611 Exam 3
AND VERIFIED SOLUTIONS/CORRECT ANSWERS|GET 100%
Study online at https://quizlet.com/_h357ma
ACCURATE!!
1. What are clinical manifestations of Constipation, decreased heat rate, and lethargy
hypothyroidism?
2. Thyroid-stimulating hormone Negative feedback. Feedback systems provide precise
(TSH) is released to stimulate thy- monitoring and control of the cellular environment.
roid hormone (TH) and is inhibited Negative feedback occurs because the changing chemi-
when plasma levels of TH are ade- cal, neural, or endocrine response to a stimulus negates
quate. This is an example of: the initiating change that triggered the release of the
hormone.
3. Lipid-soluble hormone receptors Inside the plasma membrane in the cytoplasm.
are located:
4. The releasing hormones that are Portal hypophyseal blood vessels.
made in the hypothalamus travel
to the anterior pituitary via the:
5. Which mineral is needed for thy- Iodide.
roid-stimulating hormone (TSH) to
stimulate the secretion of thyroid
hormone (TH)?
6. What effect does hyperphos- Decreases serum calcium.
phatemia have on other elec-
trolytes?
7. Insulin transports which elec- Potassium.
trolyte in the cell?
8. Which second messenger is stim- Cyclic adenosine monophosphate (cAMP). Sec-
ulated by epinephrine binding to a ond-messenger molecules are the initial link between
β-adrenergic receptor? the first signal (hormone) and the inside of the cell.
For example, the binding of epinephrine to a β adren-
, Maryville NURS 611 Exam 3
Study online at https://quizlet.com/_h357ma
ergic-receptor subtype activates (through a stimulatory
G protein) the enzyme, adenylyl cyclase. Adenylyl cy-
clase catalyzes the conversion of adenosine triphosphate
(ATP) to the second messenger, 3', and 5'-cAMP.
9. Regulation of the release of cat- Neural. The release of hormones occurs either in re-
echolamines from the adrenal sponse to an alteration in the cellular environment
medulla is an example of which or in the process of maintaining a regulated level of
type of regulation? certain hormones or certain substances. Several dif-
ferent mechanisms, one of which is neural control
(e.g., stress-induced release of catecholamines from the
adrenal medulla), regulate the release of hormones.
10. Which hormone does the second Angiotensin II. Ca++ is considered an important second
messenger calcium (Ca++) bind to messenger that facilitates the binding of a hormone
activate phospholipase C through (e.g., norepinephrine, angiotensin II) to a surface re-
a G protein? ceptor, activating the enzyme phospholipase C through
a G protein inside the plasma membrane.
11. The control of calcium in cells is Acts as a second messenger. In addition to being an
important because it: important ion that participates in a multitude of cellular
actions, Ca++ is considered an important second mes-
senger.
12. Where is antidiuretic hormone Hypothalamus; renal tubular cells. Once synthesized in
(ADH) synthesized, and where the hypothalamus, ADH acts on the vasopressin 2 (V2)
does it act? receptors of the renal duct cells to increase their perme-
ability.
13. How does a faulty negative-feed- Excessive hormone production results from a failure to
back mechanism result in a hor- turn off the system. Negative- feedback systems are
monal imbalance? important in maintaining hormones within physiolog-
ic ranges. The lack of negative-feedback inhibition on
, Maryville NURS 611 Exam 3
Study online at https://quizlet.com/_h357ma
hormonal release often results in pathologic conditions.
Excessive hormone production, which is the result of
the failure to turn off the system, can cause various
hormonal imbalances and related conditions.
14. A deficiency of which chemical Iodine. The only cause of hypothyroidism from among
may result in hypothyroidism? the provided options is a deficiency of endemic iodine.
15. What imbalance lessens the rate The overall effect of parathyroid hormone (PTH)is to in-
of secretion of parathyroid hor- crease serum calcium and to decrease serum phosphate
mone concentration.
16. Which condition may result from Hypothyroidism. If the tumor exerts sufficient pressure,
pressure exerted by a pituitary tu- then thyroid and adrenal hypofunction may occur be-
mor? cause of lack of thyroid-stimulating hormone (TSH) and
adrenocorticotropic hormone (ACTH). These result in the
symptoms of hypothyroidism and hypocortisolism.
17. Which substance is a water-solu- Insulin. Peptide or protein hormones, such as insulin,
ble protein hormone? pituitary, hypothalamic, and parathyroid, are water sol-
uble and circulate in free (unbound) forms.
18. Which of the following is a Cortisol. Cortisol and adrenal androgens are lipid-solu-
lipid-soluble hormone? ble hormones and are primarily bound to a carrier or
transport protein in circulation.
19. Most protein hormones are trans- Free in an unbound, water-soluble form. Peptide or pro-
ported in the bloodstream and tein hormones, such as insulin, pituitary, hypothalamic,
are: and parathyroid, are water soluble and circulate in free
(unbound) forms.
20. When insulin binds its receptors
on muscle cells, an increase in glu-
, Maryville NURS 611 Exam 3
Study online at https://quizlet.com/_h357ma
cose uptake by the muscle cells is Direct. Direct effects are the obvious changes in cell
the result. This is an example of function that specifically result from the stimulation by
what type of effect by a hormone? a particular hormone.
21. Graves disease develops from Thyroid-stimulating immunoglobulin that causes the
a(n): overproduction of thyroid hormones. The pathologic
features of Graves disease indicates that normal reg-
ulatory mechanisms are overridden by abnormal im-
munologic mechanisms that result in the stimulation of
excessive TH.
22. Pathologic changes associated High levels of circulating thyroid-stimulating im-
with Graves disease include: munoglobulins. The only option that correctly describes
the changes associated with Graves disease identi-
fies high levels of circulating thyroid-stimulating im-
munoglobulins that are found in more than 95% of
individuals diagnosed with the disease.
23. The signs of thyrotoxic crisis in- Hyperthermia and tachycardia
clude:
24. The level of thyroid-stimulating Low. The hyperfunction of the thyroid gland leads to
hormone (TSH) in individuals with suppression of TSH because of the normal negative
Graves disease is usually: feedback mechanism.
25. Palpation of the neck of a per- Diffusely enlarged.
son diagnosed with Graves dis-
ease would detect a thyroid that
is:
26. The effects of the syndrome of in- Dilution & water retention
appropriate antidiuretic hormone
(SIADH) secretion include solute:
