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BIOD 210/ BIOD210 Module 7: (Latest 2025/ 2026 Update) Genetics | Questions & Answers| Grade A| 100% Correct (Verified Solutions)- Portage Learning

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BIOD 210/ BIOD210 Module 7: (Latest 2025/ 2026 Update) Genetics | Questions & Answers| Grade A| 100% Correct (Verified Solutions)- Portage Learning QUESTION The fused "Philadelphia chromosome" in myelogenous leukemia is from a mutated _______ gene. Answer: BCR-ABL QUESTION Match the terms Answer: Driver: mutations that impart some sort of growth advantage Stem cell: Undifferentiated cells Passenger: mutations that do not contribute to the phenotype Metastatic: defects in the cells restraints that no longer keep them localized Tumor: solid mass growth QUESTION Using the development of colon cancer as an example, explain how the process of tumorigenesis can occur in distinct stages through the accumulation of mutations. Answer: in the first stage of colon cancer a polyp will grow and is considered benign due to a defect in the APC gene. In the second stage the kras gene causes issues in the adenoma . the kras gene becomes active and cell growth is uncontrolled and manifests a larger polyp. Then in the third stage the adenoma becomes a carcinoma which is malignant and the carcinoma is able to move and invade other tissues of different parts of the body. QUESTION Most scientists believe that the biggest driver of the cancer phenotype is the inability of the cell to: Answer: Repair QUESTION Cells that have matured to provide a specific function in the body are called ___ Answer: Differentiated cells QUESTION

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BIODl 210/l BIOD210l Modulel 7:l (Latestl
2025/l 2026l Update)l Geneticsl |l Questionsl
&l Answers|l Gradel A|l 100%l Correctl
(Verifiedl Solutions)-l Portagel Learning

Q:l Thel fusedl "Philadelphial chromosome"l inl myelogenousl leukemial isl
froml al mutatedl _______l gene.l


Answer:
BCR-ABL




Q:l Matchl thel termsl

Answer:
Driver:l mutationsl thatl impartl somel sortl ofl growthl advantagel

Steml cell:l Undifferentiatedl cells

Passenger:l mutationsl thatl dol notl contributel tol thel phenotype

Metastatic:l defectsl inl thel cellsl restraintsl thatl nol longerl keepl theml localized

Tumor:l solidl massl growth

, Q:l Usingl thel developmentl ofl colonl cancerl asl anl example,l explainl howl
thel processl ofl tumorigenesisl canl occurl inl distinctl stagesl throughl thel
accumulationl ofl mutations.l


Answer:
inl thel firstl stagel ofl colonl cancerl al polypl willl growl andl isl consideredl
benignl duel tol al defectl inl thel APCl gene.l Inl thel secondl stagel thel krasl
genel causesl issuesl inl thel adenomal .l thel krasl genel becomesl activel andl
celll growthl isl uncontrolledl andl manifestsl al largerl polyp.l Thenl inl thel thirdl
stagel thel adenomal becomesl al carcinomal whichl isl malignantl andl thel
carcinomal isl ablel tol movel andl invadel otherl tissuesl ofl differentl partsl ofl
thel body.




Q:l Mostl scientistsl believel thatl thel biggestl driverl ofl thel cancerl phenotypel
isl thel inabilityl ofl thel celll to:l


Answer:
Repair




Q:l Cellsl thatl havel maturedl tol providel al specificl functionl inl thel bodyl arel
calledl ___l


Answer:
Differentiatedl cells




Q:l Givel threel examplesl ofl differentiatedl cellsl thatl stilll divide.l

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