Modulel 4:l BIODl 101/l BIOD101l (Latestl
2025/l 2026l Update)l Essentiall Biologyl Il
W/Labl |l Questionsl &l Answers|l Gradel A|l
100%l Correctl (Verifiedl Solutions)-l
Portagel Learning
Q:l Whatl isl thel rolel ofl thel centriolel duringl thel celll cycle?
Answer:
Al centriolel isl al short,l cylindricall proteinl filamentl composedl ofl tubulinl thatl
playsl al rolel inl thel formationl ofl thel mitoticl spindle
Q:l Whyl isl thel celll cyclel controll systeml important?
Answer:
Thel celll cyclel controll systeml isl essentiall forl normall cellularl growthl andl
development,l whichl isl achievedl byl controllingl thel timingl andl ratel ofl thel
celll cycle
Q:l Truel orl False:l Thel celll cyclel controll systeml isl regulatedl byl (1)l
moleculesl calledl cyclinsl andl cyclin-dependentl kinasesl andl (2)l cellularl
signals.
Answer:
, True
Q:l Describel eachl ofl thel majorl checkpointsl ofl thel celll cycle.
Answer:
Thel G1l checkpointl isl consideredl thel majorl checkpointl ofl thel cyclel
because,l oncel thel celll proceedsl throughl thisl checkpoint,l itl hasl committedl
tol division.
Next,l isl thel G2l checkpoint.l Thisl checkpointl servesl asl al mechanisml tol
evaluatel newlyl duplicatedl DNAl beforel mitoticl divisionl occurs.l Essentially,l
thisl checkpointl focusesl onl ensuringl thel qualityl andl completionl ofl DNAl
replication
Finally,l thel Ml checkpointl isl alsol knownl asl thel spindlel checkpointl andl
occursl duringl anaphase.l Thisl isl becausel thel mainl purposel ofl thisl
checkpointl isl tol ensurel thatl thel spindlesl arel correctlyl attachedl tol thel
kinetochorel regionsl ofl thel sisterl chromatids
Q:l Truel orl False:l Celll cyclel dysregulationl isl thel solel causel ofl cancerl
celll development.
Answer:
False;l Cancerl cellsl canl developl froml causesl otherl thanl celll cyclel
dysregulationl thatl resultsl inl uncontrolledl growth.l Cancerl cellsl mayl developl
andl secretel theirl ownl growthl factors,l losel thel abilityl tol respondl tol celll
surfacel signals,l orl mayl evenl developl internall signalingl abnormalitiesl thatl
allowl immortall growth
2025/l 2026l Update)l Essentiall Biologyl Il
W/Labl |l Questionsl &l Answers|l Gradel A|l
100%l Correctl (Verifiedl Solutions)-l
Portagel Learning
Q:l Whatl isl thel rolel ofl thel centriolel duringl thel celll cycle?
Answer:
Al centriolel isl al short,l cylindricall proteinl filamentl composedl ofl tubulinl thatl
playsl al rolel inl thel formationl ofl thel mitoticl spindle
Q:l Whyl isl thel celll cyclel controll systeml important?
Answer:
Thel celll cyclel controll systeml isl essentiall forl normall cellularl growthl andl
development,l whichl isl achievedl byl controllingl thel timingl andl ratel ofl thel
celll cycle
Q:l Truel orl False:l Thel celll cyclel controll systeml isl regulatedl byl (1)l
moleculesl calledl cyclinsl andl cyclin-dependentl kinasesl andl (2)l cellularl
signals.
Answer:
, True
Q:l Describel eachl ofl thel majorl checkpointsl ofl thel celll cycle.
Answer:
Thel G1l checkpointl isl consideredl thel majorl checkpointl ofl thel cyclel
because,l oncel thel celll proceedsl throughl thisl checkpoint,l itl hasl committedl
tol division.
Next,l isl thel G2l checkpoint.l Thisl checkpointl servesl asl al mechanisml tol
evaluatel newlyl duplicatedl DNAl beforel mitoticl divisionl occurs.l Essentially,l
thisl checkpointl focusesl onl ensuringl thel qualityl andl completionl ofl DNAl
replication
Finally,l thel Ml checkpointl isl alsol knownl asl thel spindlel checkpointl andl
occursl duringl anaphase.l Thisl isl becausel thel mainl purposel ofl thisl
checkpointl isl tol ensurel thatl thel spindlesl arel correctlyl attachedl tol thel
kinetochorel regionsl ofl thel sisterl chromatids
Q:l Truel orl False:l Celll cyclel dysregulationl isl thel solel causel ofl cancerl
celll development.
Answer:
False;l Cancerl cellsl canl developl froml causesl otherl thanl celll cyclel
dysregulationl thatl resultsl inl uncontrolledl growth.l Cancerl cellsl mayl developl
andl secretel theirl ownl growthl factors,l losel thel abilityl tol respondl tol celll
surfacel signals,l orl mayl evenl developl internall signalingl abnormalitiesl thatl
allowl immortall growth
