NR 464 REVISION EXAM 2025 QUESTIONS WITH ANSWERS
GUARANTEE A+
✔✔Clinical manifestations of acute pancreatitis? - ✔✔-LUQ pain
-Sudden, deep piercing pain that is aggravated by eating
-Starts when recumbent
-Tachycardia, low BP
-Abdominal tenderness, crackles, distention
✔✔2 significant local complication of acute pancreatitis? - ✔✔1. *Pseudocyst*- need to
drain
2. *Abscess*- need prompt surgical drain
✔✔Nursing pain management for acute pancreatitis? - ✔✔-IV opioids
-Frequent position changes (side-lying with HOB elevated 45 degrees), and knees up to
abdomen
✔✔Best diet for acute pancreatitis after discharge? - ✔✔-High-carb with protein, low-fat
-Maybe fat-soluble vitamins
✔✔Most common causes of cirrhosis? - ✔✔-Most common are alcohol-induced liver
disease and chronic hepatitis B & C
-Extreme dieting, obesity
-Environmental or genetic factors
✔✔Early clinical manifestations of cirrhosis? - ✔✔-Fatigue, RUQ pain, palpable liver
✔✔Nursing care for edema and acites? - ✔✔-Assessment of skin breakdown and
abdominal appearance
-Minimize respiratory distress
✔✔Treatments to treat ascites? - ✔✔-Sodium restriction
-Albumin
-Diuretics
-Paracentesis
-TIPS (type of shunt)
✔✔Nursing care for paracentesis? - ✔✔-Empty bladder
-Monitor BP
-Manage drainage if any
-Assist with positioning
✔✔Balloon Tamponade: Sengstaken-Blakemore Tube - ✔✔-Controls hemorrhage by
mechanical compression of varices
, ✔✔Should you place NGT in patients with known or suspected varices? - ✔✔NO
✔✔Hepatic Encephalopathy (HE) - ✔✔-Ammonia accumulates in the liver and is not
converted to urea
-Ammonia crosses BBB
✔✔Clinical manifestations of Hepatic Encephalopathy (HE)? - ✔✔-Drowsiness
-Slurred speech
-Memory loss/confusion
-Agitation
-Can progress to coma
✔✔Treatment for Hepatic Encephalopathy (HE)? - ✔✔-Reduce ammonia formation
(lactulose, rifaximin)
-Treat precipitating cause
-Protect airway and skin, avoid CNS depressants
-Quiet, calm environment
✔✔Nutritional therapy for Hepatic Encephalopathy (HE)? - ✔✔-High-cal, high-carb,
moderate to low fat, protein restriction
-B-complex vitamins
✔✔Acute Kidney Injury (AKI) - ✔✔-Sudden loss of renal function
-Usually reversible
✔✔Hallmark findings of Pre-Renal causes of AKI? - ✔✔Oliguria, urine sodum <20
mEq/L and azotemia
✔✔What does RIFLE mnemonic stand for? - ✔✔Staging of AKI:
*R*isk
*I*njury
*F*ailure
*L*oss
*E*nd-Stage
✔✔Clinical manifestations of Graves disease? (3) - ✔✔-Opthalmopathy-abnormal eye
appearance of function
-Exophthalmos-increased fat deposits and fluid
-Goiter
✔✔Thyrotoxicosis (Thyrotoxic Crisis) - ✔✔-Excessive amounts hormones released
-Life-threatening emergency
-Results from stressors
-Thyroidectomy patients at risk
GUARANTEE A+
✔✔Clinical manifestations of acute pancreatitis? - ✔✔-LUQ pain
-Sudden, deep piercing pain that is aggravated by eating
-Starts when recumbent
-Tachycardia, low BP
-Abdominal tenderness, crackles, distention
✔✔2 significant local complication of acute pancreatitis? - ✔✔1. *Pseudocyst*- need to
drain
2. *Abscess*- need prompt surgical drain
✔✔Nursing pain management for acute pancreatitis? - ✔✔-IV opioids
-Frequent position changes (side-lying with HOB elevated 45 degrees), and knees up to
abdomen
✔✔Best diet for acute pancreatitis after discharge? - ✔✔-High-carb with protein, low-fat
-Maybe fat-soluble vitamins
✔✔Most common causes of cirrhosis? - ✔✔-Most common are alcohol-induced liver
disease and chronic hepatitis B & C
-Extreme dieting, obesity
-Environmental or genetic factors
✔✔Early clinical manifestations of cirrhosis? - ✔✔-Fatigue, RUQ pain, palpable liver
✔✔Nursing care for edema and acites? - ✔✔-Assessment of skin breakdown and
abdominal appearance
-Minimize respiratory distress
✔✔Treatments to treat ascites? - ✔✔-Sodium restriction
-Albumin
-Diuretics
-Paracentesis
-TIPS (type of shunt)
✔✔Nursing care for paracentesis? - ✔✔-Empty bladder
-Monitor BP
-Manage drainage if any
-Assist with positioning
✔✔Balloon Tamponade: Sengstaken-Blakemore Tube - ✔✔-Controls hemorrhage by
mechanical compression of varices
, ✔✔Should you place NGT in patients with known or suspected varices? - ✔✔NO
✔✔Hepatic Encephalopathy (HE) - ✔✔-Ammonia accumulates in the liver and is not
converted to urea
-Ammonia crosses BBB
✔✔Clinical manifestations of Hepatic Encephalopathy (HE)? - ✔✔-Drowsiness
-Slurred speech
-Memory loss/confusion
-Agitation
-Can progress to coma
✔✔Treatment for Hepatic Encephalopathy (HE)? - ✔✔-Reduce ammonia formation
(lactulose, rifaximin)
-Treat precipitating cause
-Protect airway and skin, avoid CNS depressants
-Quiet, calm environment
✔✔Nutritional therapy for Hepatic Encephalopathy (HE)? - ✔✔-High-cal, high-carb,
moderate to low fat, protein restriction
-B-complex vitamins
✔✔Acute Kidney Injury (AKI) - ✔✔-Sudden loss of renal function
-Usually reversible
✔✔Hallmark findings of Pre-Renal causes of AKI? - ✔✔Oliguria, urine sodum <20
mEq/L and azotemia
✔✔What does RIFLE mnemonic stand for? - ✔✔Staging of AKI:
*R*isk
*I*njury
*F*ailure
*L*oss
*E*nd-Stage
✔✔Clinical manifestations of Graves disease? (3) - ✔✔-Opthalmopathy-abnormal eye
appearance of function
-Exophthalmos-increased fat deposits and fluid
-Goiter
✔✔Thyrotoxicosis (Thyrotoxic Crisis) - ✔✔-Excessive amounts hormones released
-Life-threatening emergency
-Results from stressors
-Thyroidectomy patients at risk
