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NURS5315 EXAM 3 QUESTIONS WITH 100 VERIFIED ANSWERS

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NURS5315 EXAM 3 QUESTIONS WITH 100 VERIFIED ANSWERS Left atrial, pulmonary, pulmonary - Answer-Increase in LVEDP in HFpEF increases the (chamber) pressure and causes an increase in the pressure in the circulation leading to edema. Normal, decreased - Answer-The contractility of the heart is in HFpEF with filling of the ventricles. Left-Sided HF - Answer-synonymous with HFrEF Right-Sided HF - Answer-Right ventricle is unable to adequately pump blood forward to the pulmonary circulation Left-sided HF - Answer-most common cause of right sided HF due to increased work load on right ventricle r/t pulmonary back flow Right-sided HF - Answer-dependent edema; jugular vein distention; enlarged liver & spleen; fluid retention, right atrial hypertrophy Right-sided HF - Answer-May be caused by RV MI, cardiomyopathies, or pulmonic valvular disease Pulmonary edema, pulmonary - Answer-Ultimately, a person with HFrEF will experience secondary to the back of blood and high pressures into the circulation Pulmonary edema - Answer-Clinical manifestations include dyspneea, orthopnea, cough with frothy sputum, fatigue, decreased urine output, and edema. Pulmonary edema - Answer-In assessment you will observe cyanosis, pulmonary rales, pleural effusions, and an S3 gallop. Hypotension or HTN may be seen. HFpEF - Answer-EF is normal, stroke volume is normal, cardiac output is normal but there is pulmonary congestion HFpEF - Answer-Most commonly caused by HTN-induced hypertrophy or myocardial ischemia which result in remodeling HFpEF - Answer-Results from the inability of the myocytes to actively pump calcium from the cytosol which impairs ventricular relaxation (diastole) Afib - Answer-most common arrhythmia Anticoagulants - Answer-Prescribed to afib patients due to high risk of stroke cardiovascular, MI, pericardial, hyper, low - Answer-The onset of Afib may be triggered by surgery, acute , thyrotoxicosis, disease, thyroidism, pulmonary pathologies, magnesium, and caffeine intake. Rapid Ventricular Response - Answer-RVR 300 - Answer-Atrial rate is typically bpm in Afib Ventricular - Answer-Response that is important to observe in patients with Afib Controlled Afib - Answer-Afib with absence of RVR Reduced cardiac output - Answer-Reason for dyspnea, dizziness, and fatigue in afib P waves - Answer-Afib EKG wil show a rapid, irregular rhythm without . Left and Right - Answer-Two main coronary arteries that branch off of the aorta Left anterior descending artery, left marginal artery, left circumflex artery - Answer-Left coronary artery branches off into these 3 arteries Right coronary artery, right marginal artery, posterior descending artery (Interventricular Artery) - Answer-Right coronary artery branches off into these 3 arteries Left anterior descending artery - Answer-Supplies blood to the anterior 2/3rds of the interventricular septum, anterior papillary muscles, and the anterior surface of the left ventricle Left Marginal Artery - Answer-Supplies blood to the anterior side of the left ventricle obtuse marginal artery - Answer-aka Left marginal artery Left Circumflex Artery - Answer-Supplies blood to the lateral and posterior walls of the left ventricle and the left atrium Right Coronary Artery - Answer-Supplies blood to the SA and AV nodes in the right atrium and both ventricles, however it supplies more blood to the right atrium. right marginal artery - Answer-Supplies blood to the anterior and posterior portions of the right ventricle. posterior descending artery - Answer-Supplies blood to the posterior 1/3 of the interventricular septum, the posterior wall of both ventricles, and the posteromedial papillary muscles. Right, right - Answer-The majority of persons are dominant in arterial blood supply due to the posterior descending artery arising form the coronary artery Left Anterior descending - Answer-Coronary artery occlusion most commonly occurs in the artery. Left atrium - Answer-the most posterior portion of the heart. When enlarged it can cause dysphagia r/t esophageal compression or hoarseness r/t laryngeal nerve compression Atherosclerosis - Answer-Cause of coronary artery disease LDLs - Answer-Increase in is associated with an increased risk of developing CAD due to its key role in the development of atherosclerosis Catecholamines - Answer-Nicotine causes the release of which cause vasoconstriction and HTN. Nitric - Answer-Diabetes Mellitus causes a decreased production of acid, a vasodilating agent hs-CRP - Answer-high-sensitivity C-reactive protein hs-CRP - Answer- is a marker of inflammation. Elevation in this lab is associated with a higher frisk for CAD. leptin, adiponectin - Answer-Hormones released by fat cells Increased, decrease - Answer- leptin levels are associated with autoimmunity and endothelial angiogenesis. Decreased, incressed - Answer- levels of Adiponectin are associated with cardiovascular risk. Adiponectin - Answer-a protein produced by adipose cells that inhibits inflammation and protects against insulin resistance, type 2 diabetes, and cardiovascular disease Lumen, ischemia - Answer-Ultimately, CAD will cause narrowing and may cause acute . CAD - Answer-The basis for stable angina Stable angina - Answer-chest pain r/t myocardial ischemia that occurs when a person is active or under severe stress Rest - Answer-Chest pain r/t stable angina is often relieved with . Prinzmetal angina - Answer-drop in blood flow through the coronary arteries caused by a vasospasm in the artery, not by atherosclerosis (usually) Increase, increase - Answer-Catecholamines the rate of depolarization in SA & AV node Action Potentials and the HR. Class 1 Antiarrhythmics - Answer-Sodium channel blockers Class 2 antiarrhythmics - Answer-Beta-blockers; reduces cAMP, slowing SA and AV node activity, increases PR interval; adverse effects include impotence, asthma exacerbation, sedation Pharmacology Cardio Class 3 antiarrhythmics - Answer-Amiodarone, ibutilide, dofetilide, sotalol; Potassium channel blockers; increase AP duration and QT interval Class 4 antiarrhythmics - Answer-Calcium channel blockers; verapamil and diltiazem; increase PR interval; cause constipation, flushing, and edema P wave - Answer-Represents atrial depolarization PR Interval - Answer-delay of AV node to allow filling of ventricles, usually 0.12-0.20 seconds QRS complex - Answer-represents ventricular depolarization - normal is .12 seconds QT interval - Answer-Represents mechanical contraction of the ventricles Prolonged QT interval - Answer-Occurs with a number of drugs including fluoroquinolones, diflucan, zofran, many psychiatric meds, Mg deficiency, and will increase the risk for ventricular arrhythmias T wave - Answer-Represents ventricular repolarization ST segment - Answer-Represents ventricular depolarization U wave - Answer-Present on EKG in hypokalemia or bradycardia T wave inversion - Answer-Indicator of myocardial ischemia on EKG ST elevation - Answer-Bad sign on EKG, indicates myocardial injury affecting the entire thickness of the cardiac wall

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NURS5315 EXAM 3 QUESTIONS
WITH 100% VERIFIED ANSWERS

Left atrial, pulmonary, pulmonary - Answer-Increase in LVEDP in HFpEF increases the
(chamber) pressure and causes an increase in the pressure in
the circulation leading to edema.

Normal, decreased - Answer-The contractility of the heart is in HFpEF
with filling of the ventricles.

Left-Sided HF - Answer-synonymous with HFrEF

Right-Sided HF - Answer-Right ventricle is unable to adequately pump blood forward to
the pulmonary circulation

Left-sided HF - Answer-most common cause of right sided HF due to increased work
load on right ventricle r/t pulmonary back flow

Right-sided HF - Answer-dependent edema; jugular vein distention; enlarged liver &
spleen; fluid retention, right atrial hypertrophy

Right-sided HF - Answer-May be caused by RV MI, cardiomyopathies, or pulmonic
valvular disease

Pulmonary edema, pulmonary - Answer-Ultimately, a person with HFrEF will experience
secondary to the back of blood and high pressures
into the circulation

Pulmonary edema - Answer-Clinical manifestations include dyspneea, orthopnea,
cough with frothy sputum, fatigue, decreased urine output, and edema.

Pulmonary edema - Answer-In assessment you will observe cyanosis, pulmonary rales,
pleural effusions, and an S3 gallop. Hypotension or HTN may be seen.

HFpEF - Answer-EF is normal, stroke volume is normal, cardiac output is normal but
there is pulmonary congestion

HFpEF - Answer-Most commonly caused by HTN-induced hypertrophy or myocardial
ischemia which result in remodeling

HFpEF - Answer-Results from the inability of the myocytes to actively pump calcium
from the cytosol which impairs ventricular relaxation (diastole)

,Afib - Answer-most common arrhythmia

Anticoagulants - Answer-Prescribed to afib patients due to high risk of stroke

cardiovascular, MI, pericardial, hyper, low - Answer-The onset of Afib may be triggered
by surgery, acute , thyrotoxicosis,
disease, thyroidism, pulmonary pathologies, magnesium, and caffeine
intake.

Rapid Ventricular Response - Answer-RVR

300 - Answer-Atrial rate is typically > bpm in Afib

Ventricular - Answer-Response that is important to observe in patients with Afib

Controlled Afib - Answer-Afib with absence of RVR

Reduced cardiac output - Answer-Reason for dyspnea, dizziness, and fatigue in afib

P waves - Answer-Afib EKG wil show a rapid, irregular rhythm without
.

Left and Right - Answer-Two main coronary arteries that branch off of the aorta

Left anterior descending artery, left marginal artery, left circumflex artery - Answer-Left
coronary artery branches off into these 3 arteries

Right coronary artery, right marginal artery, posterior descending artery (Interventricular
Artery) - Answer-Right coronary artery branches off into these 3 arteries

Left anterior descending artery - Answer-Supplies blood to the anterior 2/3rds of the
interventricular septum, anterior papillary muscles, and the anterior surface of the left
ventricle

Left Marginal Artery - Answer-Supplies blood to the anterior side of the left ventricle

obtuse marginal artery - Answer-aka Left marginal artery

Left Circumflex Artery - Answer-Supplies blood to the lateral and posterior walls of the
left ventricle and the left atrium

Right Coronary Artery - Answer-Supplies blood to the SA and AV nodes in the right
atrium and both ventricles, however it supplies more blood to the right atrium.

, right marginal artery - Answer-Supplies blood to the anterior and posterior portions of
the right ventricle.

posterior descending artery - Answer-Supplies blood to the posterior 1/3 of the
interventricular septum, the posterior wall of both ventricles, and the posteromedial
papillary muscles.

Right, right - Answer-The majority of persons are dominant in arterial blood
supply due to the posterior descending artery arising form the coronary artery

Left Anterior descending - Answer-Coronary artery occlusion most commonly occurs in
the artery.

Left atrium - Answer-the most posterior portion of the heart. When enlarged it can cause
dysphagia r/t esophageal compression or hoarseness r/t laryngeal nerve compression

Atherosclerosis - Answer-Cause of coronary artery disease

LDLs - Answer-Increase in is associated with an increased risk of developing
CAD due to its key role in the development of atherosclerosis

Catecholamines - Answer-Nicotine causes the release of
which cause vasoconstriction and HTN.

Nitric - Answer-Diabetes Mellitus causes a decreased production of
acid, a vasodilating agent

hs-CRP - Answer-high-sensitivity C-reactive protein

hs-CRP - Answer- is a marker of inflammation. Elevation in this lab is
associated with a higher frisk for CAD.

leptin, adiponectin - Answer-Hormones released by fat cells

Increased, decrease - Answer- leptin levels are associated with
autoimmunity and endothelial angiogenesis.

Decreased, incressed - Answer- levels of Adiponectin are associated with
cardiovascular risk.

Adiponectin - Answer-a protein produced by adipose cells that inhibits inflammation and
protects against insulin resistance, type 2 diabetes, and cardiovascular disease

Lumen, ischemia - Answer-Ultimately, CAD will cause narrowing and
may cause acute .

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