Maryville NURS 615 Exam 3
1. First line treatment for Acute gout attack < > colchicine
2. First line treatment for mild to moderate pain < > Ibuprofen and tylenol
3. Treatment for RA < > NSAIDs, glucocorticoid injections and low dose prednisone
4. OTC Treatment for back pain < > Start w/ NSAIDs
5. Treatment for Gout < > Allopurinol, febuxstat, & Colchicine
6. Low dose colchicine versus high dose colchicine < > low: 1.2 mg initially, 0.6 mg
one hour later
High: 0.6-1.2mg every hour until relief. Max 4-8mg
7. 1st line diuretic treatment for heart failure < > -Spironalactone or furosemide?
8. What to monitor for with allopurinol < > BUN, Crt, uric acid & renal tests
9. What to monitor for w/ ACEIs/ARBs < > Renal function tests and serum Crt
10. What can amiodarone cause < > Hyperthyroidism
11. What to monitor for w/ Canaglifozin < > hypovolemia
12. What to monitor for w/ spironolactone < > Potassium levels frequently
13. Education needed with long-term corticosteroid use < > -Never stop abruptly
-take prilosec or zantac to protect from GI bleeds
-Diabetics may need more insulin
-Monitor for signs of infection
14. Education needed for alpha-glucosidase inhibitors < > Must be taken w/ first bite
of food from each meal
15. When to take levothyroxine < > Take first thing in the morning on an emptly
stomach and no other food/meds within 30 min of it
1/5
, 16. MOA of CCB < > -Prevents the opening of calcium channels, reducing the effect of
calcium excitation, contraction, coupling of skeletal, smooth and cardiac muscle,
regulating aldosterone and pacemaker signal conduction
= decreased contraction strength
17. MOA of metformin < > Stimulates infulin release from pancreatic beta cells to
decrease blood glucose
18. MOA of diltazem < > Same as CCB, Most likely to affect myocardial contractility
and AV conduction
19. MOa of Sacubitril/valsartan < > Block angiotensin 2 receptor, reduce vascular
tone and enhance sodium and water clearance
20. MOa of cholestyramine (questran) < > exchange chloride ions for bile acids
promoting an increase in bile acid secretion
21. MOA of ibuprofen < > inhibit COX enzymes (cyclooxygenase)
-prostaglandin synthetase
-Inhibition of Cox 1 and 2 pathways decreases prostaglandin formation that causes pain
response and inflammation
2/5
1. First line treatment for Acute gout attack < > colchicine
2. First line treatment for mild to moderate pain < > Ibuprofen and tylenol
3. Treatment for RA < > NSAIDs, glucocorticoid injections and low dose prednisone
4. OTC Treatment for back pain < > Start w/ NSAIDs
5. Treatment for Gout < > Allopurinol, febuxstat, & Colchicine
6. Low dose colchicine versus high dose colchicine < > low: 1.2 mg initially, 0.6 mg
one hour later
High: 0.6-1.2mg every hour until relief. Max 4-8mg
7. 1st line diuretic treatment for heart failure < > -Spironalactone or furosemide?
8. What to monitor for with allopurinol < > BUN, Crt, uric acid & renal tests
9. What to monitor for w/ ACEIs/ARBs < > Renal function tests and serum Crt
10. What can amiodarone cause < > Hyperthyroidism
11. What to monitor for w/ Canaglifozin < > hypovolemia
12. What to monitor for w/ spironolactone < > Potassium levels frequently
13. Education needed with long-term corticosteroid use < > -Never stop abruptly
-take prilosec or zantac to protect from GI bleeds
-Diabetics may need more insulin
-Monitor for signs of infection
14. Education needed for alpha-glucosidase inhibitors < > Must be taken w/ first bite
of food from each meal
15. When to take levothyroxine < > Take first thing in the morning on an emptly
stomach and no other food/meds within 30 min of it
1/5
, 16. MOA of CCB < > -Prevents the opening of calcium channels, reducing the effect of
calcium excitation, contraction, coupling of skeletal, smooth and cardiac muscle,
regulating aldosterone and pacemaker signal conduction
= decreased contraction strength
17. MOA of metformin < > Stimulates infulin release from pancreatic beta cells to
decrease blood glucose
18. MOA of diltazem < > Same as CCB, Most likely to affect myocardial contractility
and AV conduction
19. MOa of Sacubitril/valsartan < > Block angiotensin 2 receptor, reduce vascular
tone and enhance sodium and water clearance
20. MOa of cholestyramine (questran) < > exchange chloride ions for bile acids
promoting an increase in bile acid secretion
21. MOA of ibuprofen < > inhibit COX enzymes (cyclooxygenase)
-prostaglandin synthetase
-Inhibition of Cox 1 and 2 pathways decreases prostaglandin formation that causes pain
response and inflammation
2/5
