NR 507 ACTUAL 2025-2026 MIDTERM EXAM
NEWEST TESTBANK WITH 200 QUESTIONS AND
VERIFIED ANSWERS ASSURED SUCCESS
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Terms in this set (180)
-Mediated IgE
-inflammation due to mast cell degranultion
Hypersensitivity: Type 1
-anaphylactic, asthma, hay fever
treatment: epinephrine
-Cytotoxic reaction: tissue specific
-binds to the antigen ON THE CELL SURFACE
Hypersensitivity: Type 2
-macrophages are primary effectors cells involved
cause tissue damage or alter function
, -Graves Disease (hyperthyroidism) -alters function not
destroy
-blood transfusions reaction- transfused erythrocytes
Hypersensitivity 2
are destroyed by agglutination or lysis
-drug allergies
-hemolytic anemia
-Not organ specific
-antibody binds to soluble antigen OUTSIDE THE
Hypersensitivity: type 3 CELL SURFACE that was released into the blood or
bodily fluids, and the complex is then deposited in the
tissues
-rheumatoid arthritis
Hypersensitivity: Type 3 -Systemic Lupus Erythematosus (SLE)
-Raynaud's
Hives (urticaria) Hypersensitivity Type 1
First responders to innate Neutrophils
the immune system
Allergic contact dermatitis Hypersensitivity Type IV
Type 2 cytotoxic -IgG IgM
hypersensitivity mediated -macrophages are primary effort cells
by: -
Serum Sickness type 3 hypersensitivity
-is more of a delayed immune response.
Hypersensitivity IV,
-mediated T-cells attack tissue directly (no antibodies)
-diseases in which the body makes antibodies
Autoimmune directed against its own tissues
-
-Genetic; inherited
Primary immunodeficiency -result of single gene defects
-B and T cell deficiencies
secondary -acquired
immunodeficiency
NEWEST TESTBANK WITH 200 QUESTIONS AND
VERIFIED ANSWERS ASSURED SUCCESS
Practice questions for this set
Learn
Studied 7 terms
Nice work. You’re doing brilliantly.
Continue studying in Learn
Terms in this set (180)
-Mediated IgE
-inflammation due to mast cell degranultion
Hypersensitivity: Type 1
-anaphylactic, asthma, hay fever
treatment: epinephrine
-Cytotoxic reaction: tissue specific
-binds to the antigen ON THE CELL SURFACE
Hypersensitivity: Type 2
-macrophages are primary effectors cells involved
cause tissue damage or alter function
, -Graves Disease (hyperthyroidism) -alters function not
destroy
-blood transfusions reaction- transfused erythrocytes
Hypersensitivity 2
are destroyed by agglutination or lysis
-drug allergies
-hemolytic anemia
-Not organ specific
-antibody binds to soluble antigen OUTSIDE THE
Hypersensitivity: type 3 CELL SURFACE that was released into the blood or
bodily fluids, and the complex is then deposited in the
tissues
-rheumatoid arthritis
Hypersensitivity: Type 3 -Systemic Lupus Erythematosus (SLE)
-Raynaud's
Hives (urticaria) Hypersensitivity Type 1
First responders to innate Neutrophils
the immune system
Allergic contact dermatitis Hypersensitivity Type IV
Type 2 cytotoxic -IgG IgM
hypersensitivity mediated -macrophages are primary effort cells
by: -
Serum Sickness type 3 hypersensitivity
-is more of a delayed immune response.
Hypersensitivity IV,
-mediated T-cells attack tissue directly (no antibodies)
-diseases in which the body makes antibodies
Autoimmune directed against its own tissues
-
-Genetic; inherited
Primary immunodeficiency -result of single gene defects
-B and T cell deficiencies
secondary -acquired
immunodeficiency
