<0.9 ABI
- answers-LEAD
>1.3 abi
- answers-non compressable
0.4 ABI
- answers-limb threatening critical ishcemia
0.5 ABI
- answers-severe ischemia
0.6-0.8 abi
- answers-boarderline perfusion
1.0-1.3 ABI
- answers-normal, can compress
2 sided textile dressings>
- answers-HYDROPHOBIC = DRY WOUND>
HYDROPHILIC = WET WOUNDs
30% less function in what resulting in injury for aging population?
- answers-nerve receptors.
5 points for wounds?
- answers-1 Maintain moist wound base,
2 protect periwound skin from maceration and mechanical stripping
3 adjust topical therapy per exudate levels
4 anticipate education needed to patient/caregivers
5 clinical decision making with evidenced based practice.
60% of chronic wounds have?
- answers-BIOFILM! maxtrix sheilds
74% of venous ulcers?
- answers-being with a trigger: dermatitus, edema, burns, dry itching.
A1C for wound healing?
- answers-6.5-7.0, BG 140/154
,ABCDE?
- answers-Assymetry
Border
Color
Diameter
Evolution
abcess?
- answers-accumulation of pus or purulent drainage, raised/red
ABI = 0.5-0.8 ?
- answers-use modified 25-30mmHG. uniboot
absorption of topical treatments?
- answers-scrotum, hair follicles, face, forearm, palm of the hand, plantar foot.
acrylate terpolymer film?
- answers-cavilon! protects skin from MASD
acute lymb ischemia?
- answers-thrombus: pulessness, pain, pallor, paresthesia, paralysis, polar = cold.
Acute osteo?
- answers-Diabetics, increased bone perfusion, caused by immune compromise, neuropathy and
arterial disease.
acute skin failure?
- answers-hypo perfusion leads to tissue death, simultaneously to critical illness.
Acute wound fluid?
- answers-promotes healing = proloferative cytokines, growth factors
Acute/chronic disease with albumin and prealbumin?
- answers-contribute to inflammation, results in serum level of those proteins leak through
capillaries into the tissue = edema.
age and skin?
- answers-neonates and infants skin absorbption risj is higher. Older = prolonged skin responce.
age in wound healing?
- answers-thinning epidermis, reduced barrier function as well as increased comorbidities
aging leads to?
- answers-altered fibroblast function leads to dermal antrophy, prolonged collagen synthesis
time.
,Alginates, hydrofibers, copolyers?
- answers-wet gelling dressings for wet wounds or moderate wounds.
Allergic contact dermatitis?
- answers-well defined, macules, papules, vesicles and diffuse rash.
Angiography?
- answers-invasive contrast catheter, definative atomic eval.
angogenesis?
- answers-formation of new vaculature for wound bed.
antimicorbial cover dressings have to?
- answers-be directly in contact with wound bed to work.
antimicrobial foams helpful for?
- answers-Hypergranuation
antimicrobial topical dressing selection?
- answers-Silver (alginate), medical honey, Cardexomer Iodoine, gentine blue (hydrofera),
Cutamed (attract and bind bacteria).
Antimicrobial wound dressings>
- answers-cardexomer iodine, Honey, polyhexamethylel biguanide, methylene blue &gentian
violet, Silver and DACC (cutamed binder)
antiplatelet meds in venous issues?
- answers-aspirin= improves walking speed, decrease strokes and vascular events. Clopidogrel
(plavix) readuce vascular deaths in LEAD.
appearance of arterial wounds?
- answers-pale, necortic, punched out cookie cutter wounds. minimal drainage.
Arterial location of wounds?
- answers-Distal foot, nonhealing tramatic injury some times lower leg.
arterial pain?
- answers-cramping throbbing pain, worsened with activity and elevation. Relieved by rest and
dependancy.
Arterial wound characteristics?
- answers-tips of toes, distal foot, lower leg: Punched out well defined areas, pale looking or
necrotic wound bed, minimal drainage
, Arterial wound patient pain?
- answers-pain worsens by activity, rest makes if feel better until advanced disease then only
dependency lessons pain.
associated clinical findings of arterial wounds?
- answers-diminished pulses, Abnormal ABI, not triphasic waveforms, infection common but not
in your face apparent.
associated clinical findings of neuropathic wounds>
- answers-LOPS, foot deformities, callus (macerated or dry.)
associcated clinical findings of venous wounds>
- answers-edema, hemosiderin staining, venous stasus dermatitis, periwound maceration.
Asymptomatic LEAD?
- answers-lower resting ABI < 0.9, no claudication no prior vascular event.
Atrophie blanche>
- answers-smooth localized white atrophic skin.
atrophy?
- answers-thinning of under tissue
Autlytic, selective debridement?
- answers-removal of necrotic tissue with the body: WBC's. proteolytic, fibrinolytic and
collegenolytic enzymes
Autolytic debridement is>?
- answers-slow, inappropriate for infected wounds. most effective on slough.
autonomic neuropathy manifestation?
- answers-anhidrosis, callus, interdigital or plantar fissures, Onychomycosis, peripheral edema,
chartcot.
autonomic neuropathy?
- answers-loss of vasomotor control, impaired microvascular skin perfusion, abnormal dryness or
fissures, evidence of fungal infection, LE hair growth
Avoid all creams where?
- answers-in skin folds!
Avoid compression?
- answers-<0.5, ankle pressure below 70, toe pressure below 50.
baby skin stratum cornium?
- answers-not really functional, thinner. major heat loss and systemic absorption increased