TESTBANK o
g
RUBIN'S PATHOLOGY: go
CLINICOPATHOLOGIC
go
FOUNDATIONS OF MEDICINE
go go go
7th Edition By David S. Strayer, Emanuel Rubin
go go go go go go go
TESTBANK o
g
,Test goBank goRubin's goPathology: goClinicopathologic goFoundations goof goMedicine go7th g o Edition
Table goof goContents:
Chapter go1: goCell goAdaptation, goInjury goand goDeath
Chapter go2: goInflammation
Chapter go3: goRepair, goRegeneration goand goFibrosis
Chapter go4: goImmunopathology
Chapter go5: goNeoplasia
Chapter go6: goDevelopmental goand goGenetic goDiseases
Chapter go7: goHemodynamic goDisorders
Chapter go8: goEnvironmental goand goNutritional goPathology
Chapter go9: goInfectious goand goParasitic goDiseases
Section goII: goPathogenesis goof goSystemic goConditions goExpandable gosection
Chapter go10: goAging
Chapter go11: goSystemic goAutoimmune goDiseases
Chapter go12: goSepsis
Chapter go13: goObesity goand goDiabetes goMellitus
Chapter go14: goThe goPathology goof goPregnancy
Chapter go15: goThe goAmyloidoses
Section goIII: goDiseases goof goIndividual goOrgan goSystemsExpandable gosection
Chapter go16: goBlood goVessels
Chapter go17: goThe goHeart
Chapter go18: goThe goRespiratory goSystem
Chapter go19: goThe goGastrointestinal goTract
Chapter go20: goThe goLiver goand goBiliary goSystem
Chapter go21: goThe goPancreas
Chapter go22: goThe goKidney
Chapter go23: goThe goLower goUrinary goTract goand goMale goReproductive goSystem
Chapter go24: goThe goFemale goReproductive goSystem goand goPeritoneum
Chapter go25: goThe goBreast
Chapter go26: goHematopathology
Chapter go27: goThe goEndocrine goSystem
Chapter go28: goThe goSkin
Chapter go29: goThe goHead goand goNeck
Chapter go30: goBones, goJoints goand goSoft goTissue
Chapter go31: goSkeletal goMuscle goand goPeripheral goNervous goSystem
Chapter go32: goThe goCentral goNervous goSystem
Chapter go33: goThe goEye
Chapter go34: goForensic goPathology
,Rubin's goPathology: goClinicopathologic goFoundations goof goMedicine
goChapter go1: goCell goAdaptation, goInjury goand goDeath
Ischemia goand goother gotoxic goinjuries goincrease gothe goaccumulation goof gointracellular gocalcium goas goa
1. goresult goof:
A) release goof gostored gocalcium gofrom gothe gomitochondria.
B) improved gointracellular govolume goregulation.
C) decreased goinflux goacross gothe gocell gomembrane.
D) attraction goof gocalcium goto gofatty goinfiltrates.
The gopatient gois gofound goto gohave goliver godisease, goresulting goin gothe goremoval goof goa golobe goof gohis
2. goliver. goAdaptation goto gothe goreduced gosize goof gothe goliver goleads goto go_ of gothe goremaining
goliver gocells.
A) metaplasia
B) organ goatrophy
C) compensatory gohyperplasia
D) physiologic gohypertrophy
A goperson goeating gopeanuts gostarts gochoking goand gocollapses. goHis goairway goobstruction gois
gopartially gocleared, gobut gohe goremains gohypoxic gountil gohe goreaches gothe gohospital. goThe
3. goprolonged gocell gohypoxia gocaused goa gocerebral goinfarction goand goresulting _ goin gothe gobrain.
A) caspase goactivation
B) coagulation gonecrosis
C) rapid gophagocytosis
D) protein gop53 godeficiency
Bacteria goand goviruses gocause gocell godamage goby , gowhich gois gounique gofrom gothe
4. gointracellular godamage gocaused goby goother goinjurious goagents.
A) disrupting gothe gosodium/potassium goATPase gopump
B) interrupting gooxidative gometabolism goprocesses
C) replicating goand goproducing gocontinued goinjury
D) decreasing goprotein gosynthesis goand gofunction
The gopatient gohas goa goprolonged gointerruption goin goarterial goblood goflow goto gohis goleft gokidney,
5. gocausing gohypoxic gocell goinjury goand gothe gorelease goof gofree goradicals. goFree goradicals godamage
gocells goby:
A) destroying gophospholipids goin gothe gocell gomembrane.
B) altering gothe goimmune goresponse goof gothe gocell.
C) disrupting gocalcium gostorage goin gothe gocell.
, D) inactivation goof goenzymes goand gomitochondria.
6. Injured gocells gohave goimpaired goflow goof gosubstances gothrough gothe gocell gomembrane goas goa goresult goof:
A) increased gofat goload.
B) altered gopermeability.
C) altered goglucose goutilization.
D) increased gosurface goreceptors.
7. Reversible goadaptive gointracellular goresponses goare goinitiated goby:
A) stimulus gooverload.
B) genetic gomutations.
C) chemical gomessengers.
D) mitochondrial goDNA.
8. Injured gocells gobecome govery goswollen goas goa goresult goof:
A) increased gocell goprotein gosynthesis.
B) altered gocell govolume goregulation.
C) passive goentry goof gopotassium gointo gothe gocell.
D) bleb goformation goin gothe goplasma gomembrane.
A godiabetic gopatient gohas goimpaired gosensation, gocirculation, goand gooxygenation goof gohis gofeet. goHe
gosteps goon goa gopiece goof goglass, gothe gowound godoes gonot goheal, goand gothe goarea gotissue gobecomes
9. gonecrotic. goThe gonecrotic gocell godeath gois gocharacterized goby:
A) rapid goapoptosis.
B) cellular gorupture.
C) shrinkage goand gocollapse.
D) chronic goinflammation.
A go99-year-old gowoman gohas goexperienced gothe godecline goof gocell gofunction goassociated gowith goage.
10. goA gogroup goof gotheories goof gocellular goaging gofocus goon goprogrammed:
A) changes gowith gogenetic goinfluences.
B) elimination goof gocell goreceptor gosites.
C) insufficient gotelomerase goenzyme.
D) DNA gomutation goor gofaulty gorepair.
An go89-year-old gofemale gopatient gohas goexperienced gosignificant godecreases goin goher gomobility
goand gostamina goduring goa go3-week gohospital gostay gofor gothe gotreatment goof goa gofemoral gohead
gofracture. goWhich goof gothe gofollowing gophenomena gomost golikely goaccounts gofor gothe gopatients
11. godecrease goin gomuscle gofunction gothat gounderlies goher goreduced gomobility?
A) Impaired gomuscle gocell gometabolism goresulting gofrom gometaplasia
g
RUBIN'S PATHOLOGY: go
CLINICOPATHOLOGIC
go
FOUNDATIONS OF MEDICINE
go go go
7th Edition By David S. Strayer, Emanuel Rubin
go go go go go go go
TESTBANK o
g
,Test goBank goRubin's goPathology: goClinicopathologic goFoundations goof goMedicine go7th g o Edition
Table goof goContents:
Chapter go1: goCell goAdaptation, goInjury goand goDeath
Chapter go2: goInflammation
Chapter go3: goRepair, goRegeneration goand goFibrosis
Chapter go4: goImmunopathology
Chapter go5: goNeoplasia
Chapter go6: goDevelopmental goand goGenetic goDiseases
Chapter go7: goHemodynamic goDisorders
Chapter go8: goEnvironmental goand goNutritional goPathology
Chapter go9: goInfectious goand goParasitic goDiseases
Section goII: goPathogenesis goof goSystemic goConditions goExpandable gosection
Chapter go10: goAging
Chapter go11: goSystemic goAutoimmune goDiseases
Chapter go12: goSepsis
Chapter go13: goObesity goand goDiabetes goMellitus
Chapter go14: goThe goPathology goof goPregnancy
Chapter go15: goThe goAmyloidoses
Section goIII: goDiseases goof goIndividual goOrgan goSystemsExpandable gosection
Chapter go16: goBlood goVessels
Chapter go17: goThe goHeart
Chapter go18: goThe goRespiratory goSystem
Chapter go19: goThe goGastrointestinal goTract
Chapter go20: goThe goLiver goand goBiliary goSystem
Chapter go21: goThe goPancreas
Chapter go22: goThe goKidney
Chapter go23: goThe goLower goUrinary goTract goand goMale goReproductive goSystem
Chapter go24: goThe goFemale goReproductive goSystem goand goPeritoneum
Chapter go25: goThe goBreast
Chapter go26: goHematopathology
Chapter go27: goThe goEndocrine goSystem
Chapter go28: goThe goSkin
Chapter go29: goThe goHead goand goNeck
Chapter go30: goBones, goJoints goand goSoft goTissue
Chapter go31: goSkeletal goMuscle goand goPeripheral goNervous goSystem
Chapter go32: goThe goCentral goNervous goSystem
Chapter go33: goThe goEye
Chapter go34: goForensic goPathology
,Rubin's goPathology: goClinicopathologic goFoundations goof goMedicine
goChapter go1: goCell goAdaptation, goInjury goand goDeath
Ischemia goand goother gotoxic goinjuries goincrease gothe goaccumulation goof gointracellular gocalcium goas goa
1. goresult goof:
A) release goof gostored gocalcium gofrom gothe gomitochondria.
B) improved gointracellular govolume goregulation.
C) decreased goinflux goacross gothe gocell gomembrane.
D) attraction goof gocalcium goto gofatty goinfiltrates.
The gopatient gois gofound goto gohave goliver godisease, goresulting goin gothe goremoval goof goa golobe goof gohis
2. goliver. goAdaptation goto gothe goreduced gosize goof gothe goliver goleads goto go_ of gothe goremaining
goliver gocells.
A) metaplasia
B) organ goatrophy
C) compensatory gohyperplasia
D) physiologic gohypertrophy
A goperson goeating gopeanuts gostarts gochoking goand gocollapses. goHis goairway goobstruction gois
gopartially gocleared, gobut gohe goremains gohypoxic gountil gohe goreaches gothe gohospital. goThe
3. goprolonged gocell gohypoxia gocaused goa gocerebral goinfarction goand goresulting _ goin gothe gobrain.
A) caspase goactivation
B) coagulation gonecrosis
C) rapid gophagocytosis
D) protein gop53 godeficiency
Bacteria goand goviruses gocause gocell godamage goby , gowhich gois gounique gofrom gothe
4. gointracellular godamage gocaused goby goother goinjurious goagents.
A) disrupting gothe gosodium/potassium goATPase gopump
B) interrupting gooxidative gometabolism goprocesses
C) replicating goand goproducing gocontinued goinjury
D) decreasing goprotein gosynthesis goand gofunction
The gopatient gohas goa goprolonged gointerruption goin goarterial goblood goflow goto gohis goleft gokidney,
5. gocausing gohypoxic gocell goinjury goand gothe gorelease goof gofree goradicals. goFree goradicals godamage
gocells goby:
A) destroying gophospholipids goin gothe gocell gomembrane.
B) altering gothe goimmune goresponse goof gothe gocell.
C) disrupting gocalcium gostorage goin gothe gocell.
, D) inactivation goof goenzymes goand gomitochondria.
6. Injured gocells gohave goimpaired goflow goof gosubstances gothrough gothe gocell gomembrane goas goa goresult goof:
A) increased gofat goload.
B) altered gopermeability.
C) altered goglucose goutilization.
D) increased gosurface goreceptors.
7. Reversible goadaptive gointracellular goresponses goare goinitiated goby:
A) stimulus gooverload.
B) genetic gomutations.
C) chemical gomessengers.
D) mitochondrial goDNA.
8. Injured gocells gobecome govery goswollen goas goa goresult goof:
A) increased gocell goprotein gosynthesis.
B) altered gocell govolume goregulation.
C) passive goentry goof gopotassium gointo gothe gocell.
D) bleb goformation goin gothe goplasma gomembrane.
A godiabetic gopatient gohas goimpaired gosensation, gocirculation, goand gooxygenation goof gohis gofeet. goHe
gosteps goon goa gopiece goof goglass, gothe gowound godoes gonot goheal, goand gothe goarea gotissue gobecomes
9. gonecrotic. goThe gonecrotic gocell godeath gois gocharacterized goby:
A) rapid goapoptosis.
B) cellular gorupture.
C) shrinkage goand gocollapse.
D) chronic goinflammation.
A go99-year-old gowoman gohas goexperienced gothe godecline goof gocell gofunction goassociated gowith goage.
10. goA gogroup goof gotheories goof gocellular goaging gofocus goon goprogrammed:
A) changes gowith gogenetic goinfluences.
B) elimination goof gocell goreceptor gosites.
C) insufficient gotelomerase goenzyme.
D) DNA gomutation goor gofaulty gorepair.
An go89-year-old gofemale gopatient gohas goexperienced gosignificant godecreases goin goher gomobility
goand gostamina goduring goa go3-week gohospital gostay gofor gothe gotreatment goof goa gofemoral gohead
gofracture. goWhich goof gothe gofollowing gophenomena gomost golikely goaccounts gofor gothe gopatients
11. godecrease goin gomuscle gofunction gothat gounderlies goher goreduced gomobility?
A) Impaired gomuscle gocell gometabolism goresulting gofrom gometaplasia