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1
CCT EXAM - CARDIAC MEDICATIONS
QUESTIONS AND CORRECT DETAILED
ANSWERS 2025-2026 (VERIFIED ANSWERS)
||ALREADY GRADED A+||NEWEST VERSION
Verapamil ANS: antianginal, antihypertensive, antiarrhythmic -- smooth muscle
relaxation, widening of blood vessels to increase oxygen delivery to heart. Decreases
conduction through the AV node.
ACE inhibitors ANS: Block conversion of angiotensin I to angiotensin II -->
vasodilation, fluid excretion, reduced aldosterone production
ARBs ANS: inhibit the activity of ang II at specific receptors in blood vessels -->
vasodilation, fluid excretion, reduced aldosterone production
Therapeutic uses of cardiac glycosides ANS: CHF:
-increased inotropy
-increased EF
-reduced preload
-reduced pulmonary congestion
Arrhythmias:
o
o
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-reduced conduction through AV node
-reduced ventricular rate in A-fib and A-flutter
Effect of cardiac glycosides ANS: bind to the Na-K-ATP pump, causing a buildup of
intracellular sodium, which leads to a subsequent increase in the intracellular concentration
of calcium. --> increases force of contraction, lower HR
Type I Antiarrhythmics ANS: sodium channel blockers: inhibit the movement of
sodium across cellular membranes (slows conduction velocity and prolongs the refractory
period)
Type Ia antiarrythmics ANS: -Moderate sodium channel blockade, increased refractory
period
-commonly used to treat supraventricular and ventricular tachyarrhythmias
i.e. quinidine, procainamide, disopyramide
Type Ib antiarrythmics ANS: -weak sodium channel blockade, decreased refractory
period
-commonly used to treat ventricular arrhythmias
o
o
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© 2024 TestTrackers o Resources & Updates: [Testtrackers - Stuvia US]
o Customer Support: o Your Success is Our Mission!
[+254707240657]
1
CCT EXAM - CARDIAC MEDICATIONS
QUESTIONS AND CORRECT DETAILED
ANSWERS 2025-2026 (VERIFIED ANSWERS)
||ALREADY GRADED A+||NEWEST VERSION
Verapamil ANS: antianginal, antihypertensive, antiarrhythmic -- smooth muscle
relaxation, widening of blood vessels to increase oxygen delivery to heart. Decreases
conduction through the AV node.
ACE inhibitors ANS: Block conversion of angiotensin I to angiotensin II -->
vasodilation, fluid excretion, reduced aldosterone production
ARBs ANS: inhibit the activity of ang II at specific receptors in blood vessels -->
vasodilation, fluid excretion, reduced aldosterone production
Therapeutic uses of cardiac glycosides ANS: CHF:
-increased inotropy
-increased EF
-reduced preload
-reduced pulmonary congestion
Arrhythmias:
o
o
Thank You for Choosing Us!
© 2024 TestTrackers o Resources & Updates: [Testtrackers - Stuvia US]
o Customer Support: o Your Success is Our Mission!
[+254707240657]
, Testtrackers
2
-reduced conduction through AV node
-reduced ventricular rate in A-fib and A-flutter
Effect of cardiac glycosides ANS: bind to the Na-K-ATP pump, causing a buildup of
intracellular sodium, which leads to a subsequent increase in the intracellular concentration
of calcium. --> increases force of contraction, lower HR
Type I Antiarrhythmics ANS: sodium channel blockers: inhibit the movement of
sodium across cellular membranes (slows conduction velocity and prolongs the refractory
period)
Type Ia antiarrythmics ANS: -Moderate sodium channel blockade, increased refractory
period
-commonly used to treat supraventricular and ventricular tachyarrhythmias
i.e. quinidine, procainamide, disopyramide
Type Ib antiarrythmics ANS: -weak sodium channel blockade, decreased refractory
period
-commonly used to treat ventricular arrhythmias
o
o
Thank You for Choosing Us!
© 2024 TestTrackers o Resources & Updates: [Testtrackers - Stuvia US]
o Customer Support: o Your Success is Our Mission!
[+254707240657]
