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NURS 2245: Exam 2 Exam Study Guide

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©BRIGHTSTARS EXAM SOLUTIONS 11/16/2024 10:16 AM 1 | P a g e NURS 2245: Exam 2 Exam Study Guide. Dysrhythmias originating in the atria - answerpremature atrial contractions paroxysmal supra ventricular tachycardia atrial flutter atrial fibrillation premature atrial contractions - answerrate: depends upon underlying rhythm rhythm: usually regular except for PAC pacemaker site: ectopic sites in atria P waves: occurs earlier than expected PRI: varies dependent on foci of impulse QRS: usually normal paroxysmal supra ventricular tachycardia - answerrate: 150-250 bpm rhythm: regular pacemaker site: atria (outside SA node) P waves: often buried in preceding T wave PRI: usually normal QRS: usually normal adenosine (adenocard): action - answerslows conduction through AV node interrupts re-entry will not convert a flutter or a-fib **given rapidly: IVP, followed with saline flush ©BRIGHTSTARS EXAM SOLUTIONS 11/16/2024 10:16 AM 2 | P a g e adenosine: S/E - answerbrief period asystole flushing HA lightheadedness dizziness nausea SOB rarely: chest pain atrial flutter - answerrate: atrial, 250-350 bpm ventricular rate: varies rhythm: usually regular pacemaker site: atrial (outside SA node) P waves: F waves are present PRI: usually normal QRS: usually normal Atrial Fibrillation (A-Fib) - answerrate: atrial rate: 350-750 bpm ventricular: varies rhythm: irregularly irregular pacemaker site: atrial (outside of SA node) P waves: none discernible; f waves present PRI: none QRS: normal mechanism of A-fib - answerstructural &/or electrophysiologic abnormalities alter atrial tissue promoting abnormal impulse formation caused by diverse mechanisms ©BRIGHTSTARS EXAM SOLUTIONS 11/16/2024 10:16 AM 3 | P a g e mechanisms: not completely understood AF is the common rhythm seen - the end result so to speak Atrial Fibrillation (A-Fib) extra cardiac factors - answerHTN obesity sleep apnea hyperthyroidism alcohol drugs atrial structure abnormalities - answerfibrosis dilation ischemia hypertrophy atrial electrical abnormalities - answerincrease: heterogeneity automaticity decrease: conduction action potential duration abnormal: Ca ++ handling Treatment of A-fib and a-flutter - answerrate control prevent thromboembolism rhythm control by: chemical

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©BRIGHTSTARS EXAM SOLUTIONS

11/16/2024 10:16 AM


NURS 2245: Exam 2 Exam Study Guide.


Dysrhythmias originating in the atria - answer✔premature atrial contractions
paroxysmal supra ventricular tachycardia
atrial flutter
atrial fibrillation

premature atrial contractions - answer✔rate: depends upon underlying rhythm
rhythm: usually regular except for PAC
pacemaker site: ectopic sites in atria
P waves: occurs earlier than expected
PRI: varies dependent on foci of impulse
QRS: usually normal

paroxysmal supra ventricular tachycardia - answer✔rate: 150-250 bpm
rhythm: regular
pacemaker site: atria (outside SA node)
P waves: often buried in preceding T wave
PRI: usually normal
QRS: usually normal

adenosine (adenocard): action - answer✔slows conduction through AV node
interrupts re-entry
will not convert a flutter or a-fib
**given rapidly: IVP, followed with saline flush


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, ©BRIGHTSTARS EXAM SOLUTIONS

11/16/2024 10:16 AM

adenosine: S/E - answer✔brief period asystole
flushing
HA
lightheadedness
dizziness
nausea
SOB
rarely: chest pain

atrial flutter - answer✔rate: atrial, 250-350 bpm
ventricular rate: varies
rhythm: usually regular
pacemaker site: atrial (outside SA node)
P waves: F waves are present
PRI: usually normal
QRS: usually normal

Atrial Fibrillation (A-Fib) - answer✔rate: atrial rate: 350-750 bpm
ventricular: varies
rhythm: irregularly irregular
pacemaker site: atrial (outside of SA node)
P waves: none discernible; f waves present
PRI: none
QRS: normal

mechanism of A-fib - answer✔structural &/or electrophysiologic abnormalities alter atrial tissue
promoting abnormal impulse formation
caused by diverse mechanisms


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, ©BRIGHTSTARS EXAM SOLUTIONS

11/16/2024 10:16 AM

mechanisms: not completely understood
AF is the common rhythm seen - the end result so to speak

Atrial Fibrillation (A-Fib) extra cardiac factors - answer✔HTN
obesity
sleep apnea
hyperthyroidism
alcohol
drugs

atrial structure abnormalities - answer✔fibrosis
dilation
ischemia
hypertrophy

atrial electrical abnormalities - answer✔increase:
heterogeneity
automaticity


decrease:
conduction
action potential duration


abnormal: Ca ++ handling

Treatment of A-fib and a-flutter - answer✔rate control
prevent thromboembolism
rhythm control by:
chemical


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, ©BRIGHTSTARS EXAM SOLUTIONS

11/16/2024 10:16 AM

direct current cardioversion
catheter ablation

rate control - answer✔beta blockers: LOL
calcium channel blockers: verapamil, diltiazem
digoxin
amiodarone

Diltiazem (Cardizem): action - answer✔inhibits movement of Ca across cardiac and arterial
muscle
slows cardiac conduction
decreases contractility
dilates coronary arteries

Diltiazem (Cardizem): uses - answer✔control rapid ventricular response, Afl, Afib, SVT

administration of Diltiazem - answer✔IV infusion then oral

Interventions for Diltiazem - answer✔monitor VS
cardiac monitor
stop if:

medications: prevent thromboembolism - answer✔vitamin K agonist: warfarin (Coumadin)
heparin or enoxaparin (Lovenox)
Direct thrombin inhibitor: Dabigatran (Pradaxa), rivaroxaban (Xarelto), apixaban (Eliquis)

Vitamin K agonist: Warfarin (Coumadin) - answer✔target INR: 2.0 to 3.0
unless age > 75: 1.6-2.5
antidote:

heparin or enoxaparin (Lovenox) - answer✔until therapeutic warfarin OR when surgical
procedures
Target PTT:
Antidote:

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