Diabetes (NCA) Exam Questions With Complete Solutions
diabetes
When the ability to produce or to use the hormone insulin is
impaired. Results in abnormal metabolism of carbohydrates and
elevated levels of glucose in the blood and urine
classifications
Type 1
Type 2
Gestational Diabetes (GDM)
gestational diabetes
Develops during pregnancy
Should be reclassified after delivery
High risk of developing type 2 later
disease of pancreas
Chronic pancreatitis can lead to the development of diabetes
infection
drug induced
Diabetics that develops as a result of the use of medications
pre-diabetes
,Impaired glucose tolernace
normal physiology
Insulin secreted
Glucose enters cell/metabolized
Stored as glycogen in liver, muscle
Fat stored in adipose tissue
Protein synthesized into amino acids
Plasma glucose within normal limits
Between 70 and 100 mg/dL
diabetes physiology
Insufficient/absent insulin
hyperglycemia
fluid shift
Polyuria
Polydypsia
Glycogenesis
Polyphasia
hyperglycemia
Blood glucose exceeds renal threshold
fluid shift
, Intracellular to extracellular
glycogenesis
Fats and protein metabolized
type 1
Younger client, <20
Sudden onset
Beta cell destruction (no insulin)
Normal/weight loss
type 1 complications
Hyperglycemia: DKA
Hypoglycemia:
HHNK
type 2
Older client
Gradual onset
Insulin resistance
Beta cell dysfunction
Obesity
type 2 acute complications
Hyperglycemia
Hypoglycemia
HHNK
diabetes
When the ability to produce or to use the hormone insulin is
impaired. Results in abnormal metabolism of carbohydrates and
elevated levels of glucose in the blood and urine
classifications
Type 1
Type 2
Gestational Diabetes (GDM)
gestational diabetes
Develops during pregnancy
Should be reclassified after delivery
High risk of developing type 2 later
disease of pancreas
Chronic pancreatitis can lead to the development of diabetes
infection
drug induced
Diabetics that develops as a result of the use of medications
pre-diabetes
,Impaired glucose tolernace
normal physiology
Insulin secreted
Glucose enters cell/metabolized
Stored as glycogen in liver, muscle
Fat stored in adipose tissue
Protein synthesized into amino acids
Plasma glucose within normal limits
Between 70 and 100 mg/dL
diabetes physiology
Insufficient/absent insulin
hyperglycemia
fluid shift
Polyuria
Polydypsia
Glycogenesis
Polyphasia
hyperglycemia
Blood glucose exceeds renal threshold
fluid shift
, Intracellular to extracellular
glycogenesis
Fats and protein metabolized
type 1
Younger client, <20
Sudden onset
Beta cell destruction (no insulin)
Normal/weight loss
type 1 complications
Hyperglycemia: DKA
Hypoglycemia:
HHNK
type 2
Older client
Gradual onset
Insulin resistance
Beta cell dysfunction
Obesity
type 2 acute complications
Hyperglycemia
Hypoglycemia
HHNK
