NU 545-805 Unit 1 Test Bank 6.
7.
Questions
8. What process causes heat and redness to occur during the inflammatory
process?
a. Vasodilation of blood vessels
b. Platelet aggregation
CHAPTER 7 c. Decreased capillary permeability
d. Endothelial cell contraction
1. Which action is a purpose of the inflammatory process?
a. To provide specific responses toward antigens 9. What does activation of the classical pathway begin with?
b. To lyse cell membranes of microorganisms a. Viruses
c. To prevent infection of the injured tissue b. Antigen-antibody complexes
d. To create immunity against subsequent tissue injury c. Mast cells
d. Macrophages
2. How do surfactant proteins A through D provide innate resistance?
a. Initiate the complement cascade. 10. What plasma protein system forms a fibrinous meshwork at an
b. Promote phagocytosis. inflamed site?
c. Secrete mucus. a. Complement
d. Synthesize lysosomes. b. Coagulation
c. Kinin
3. Which secretion is a first line of defense against pathogen invasion that d. Fibrinolysis
involves antibacterial and antifungal fatty acids, as well as lactic acid?
a. Optic tears 11. Which component of the plasma protein system tags
b. Oral saliva pathogenic microorganisms for destruction by neutrophils and
c. Sweat gland perspiration macrophages?a. Complement cascade
d. Sebaceous gland sebum b. Coagulation system
c. Kinin system
4. Which bacterium grows in the intestines after prolonged antibiotic d. Immune system
therapy?
a. Lactobacillus 12. What is the vascular effect of histamine released from mast cells?
b. Candida albicans a. Platelet adhesion
c. Clostridium difficile b. Initiation of the clotting cascade
d. Helicobacter pylori c. Vasodilation
d. Increased endothelial adhesiveness
5. What causes the edema that occurs during the inflammatory process?
a. Vasodilation of blood vessels
b. Increased capillary permeability
c. Endothelial cell expansion
d. Emigration of neutrophils
,13. What is an outcome of the complement cascade? 18. What effect does the process of histamine binding to the histamine-2
a. Activation of the clotting cascade (H2) receptor have on inflammation?
b. Prevention of the spread of infection to adjacent tissues a. Inhibition
c. Inactivation of chemical mediators such as histamine b. Activation
d. Lysis of bacterial cell membranes c. Acceleration
d. Termination
14. What is the function of opsonization related to the complement
cascade? 19. Frequently when H1 and H2 receptors are located on the same cells,
a. To tag pathogenic microorganisms for destruction by neutrophils and they act in what fashion?
macrophages a. Synergistically
b. To process pathogenic microorganisms so that activated lymphocytes b. Additively
can be created for acquired immunity c. Antagonistically
c. To destroy glycoprotein cell membranes of pathogenic microorganisms d. Agonistically
d. To promote anaphylatoxic activity, resulting in mast cell degranulation
20. Some older adults have impaired inflammation and wound healing
15. In the coagulation (clotting) cascade, the intrinsic and the extrinsic because of which problem?
pathways converge at which factor? a. Circulatory system cannot adequately perfuse tissues.
a. XII b. Complement and chemotaxis are deficient.
b. VII c. Underlying chronic illness(es) exists.
c. X d. Number of mast cells is insufficient.
d. V
21. Which chemical mediator derived from mast cells retracts
16. Which chemical interacts among all plasma protein systems by endothelial cells to increase vascular permeability and to cause leukocyte
degrading blood clots, activating complement, and activating the Hageman adhesion toendothelial cells?
factor? a. Chemokines
a. Kallikrein b. Prostaglandin E
b. Histamine c. Platelet-activating factor
c. Bradykinin d. Bradykinin
d. Plasmin
22. What is the inflammatory effect of nitric oxide (NO)?
17. How does the chemotactic factor affect the inflammatory process? a. Increases capillary permeability, and causes pain
a. By causing vasodilation around the inflamed area b. Increases neutrophil chemotaxis and platelet aggregation
b. By stimulating smooth muscle contraction in the inflamed area c. Causes smooth muscle contraction and fever
c. By directing leukocytes to the inflamed area d. Decreases mast cell function, and decreases platelet aggregation
d. By producing edema around the inflamed area
, 23. What is the correct sequence in phagocytosis? 28. Which cytokine is produced and released from virally infected host
a. Engulfment, recognition, fusion, destruction cells?
b. Fusion, engulfment, recognition, destruction a. IL-1
c. Recognition, engulfment, fusion, destruction b. IL-10
d. Engulfment, fusion, recognition, destruction c. TNF
d. IFN
24. When considering white blood cell differentials, acute inflammatory
reactions are related to elevations of which leukocyte? 29. Which manifestation of inflammation is systemic?
a. Monocytes a. Formation of exudates
b. Eosinophils b. Fever and leukocytosis
c. Neutrophils c. Redness and heat
d. Basophils d. Pain and edema
25. In the later stages of an inflammatory response, which phagocytic cell is 30. The acute inflammatory response is characterized by fever that is
predominant? produced by the hypothalamus being affected by what?
a. Neutrophils a. Endogenous pyrogens
b. Monocytes b. Bacterial endotoxin
c. Chemokines c. Antigen-antibody complexes
d. Eosinophils d. Exogenous pyrogens
26. In regulating vascular mediators released from mast cells, the role of 31. What occurs during the process of repair after tissue damage?
eosinophils is to release what? a. Nonfunctioning scar tissue replaces destroyed tissue.
a. Arylsulfatase B, which stimulates the formation of B lymphocytes b. Regeneration occurs; the original tissue is replaced.
b. Histaminase, which limits the effects of histamine during acute c. Resolution occurs; tissue is regenerated.
inflammation d. Epithelialization replaces destroyed tissue.
c. Lysosomal enzymes, which activate mast cell degranulation during acute
inflammation 32. What is the role of fibroblasts during the reconstructive phase of
d. Immunoglobulin E, which defends the body against parasites wound healing?
a. Generate new capillaries from vascular endothelial cells around the
27. What is a role of a natural killer (NK) cells? wound.
a. Initiation of the complement cascade b. Establish connections between neighboring cells and contract their
b. Elimination of malignant cells fibers.
c. Binding tightly to antigens c. Synthesize and secrete collagen and the connective tissue proteins.
d. Proliferation after immunization with antigen d. Provide enzymes that débride the wound bed of dead cells.
7.
Questions
8. What process causes heat and redness to occur during the inflammatory
process?
a. Vasodilation of blood vessels
b. Platelet aggregation
CHAPTER 7 c. Decreased capillary permeability
d. Endothelial cell contraction
1. Which action is a purpose of the inflammatory process?
a. To provide specific responses toward antigens 9. What does activation of the classical pathway begin with?
b. To lyse cell membranes of microorganisms a. Viruses
c. To prevent infection of the injured tissue b. Antigen-antibody complexes
d. To create immunity against subsequent tissue injury c. Mast cells
d. Macrophages
2. How do surfactant proteins A through D provide innate resistance?
a. Initiate the complement cascade. 10. What plasma protein system forms a fibrinous meshwork at an
b. Promote phagocytosis. inflamed site?
c. Secrete mucus. a. Complement
d. Synthesize lysosomes. b. Coagulation
c. Kinin
3. Which secretion is a first line of defense against pathogen invasion that d. Fibrinolysis
involves antibacterial and antifungal fatty acids, as well as lactic acid?
a. Optic tears 11. Which component of the plasma protein system tags
b. Oral saliva pathogenic microorganisms for destruction by neutrophils and
c. Sweat gland perspiration macrophages?a. Complement cascade
d. Sebaceous gland sebum b. Coagulation system
c. Kinin system
4. Which bacterium grows in the intestines after prolonged antibiotic d. Immune system
therapy?
a. Lactobacillus 12. What is the vascular effect of histamine released from mast cells?
b. Candida albicans a. Platelet adhesion
c. Clostridium difficile b. Initiation of the clotting cascade
d. Helicobacter pylori c. Vasodilation
d. Increased endothelial adhesiveness
5. What causes the edema that occurs during the inflammatory process?
a. Vasodilation of blood vessels
b. Increased capillary permeability
c. Endothelial cell expansion
d. Emigration of neutrophils
,13. What is an outcome of the complement cascade? 18. What effect does the process of histamine binding to the histamine-2
a. Activation of the clotting cascade (H2) receptor have on inflammation?
b. Prevention of the spread of infection to adjacent tissues a. Inhibition
c. Inactivation of chemical mediators such as histamine b. Activation
d. Lysis of bacterial cell membranes c. Acceleration
d. Termination
14. What is the function of opsonization related to the complement
cascade? 19. Frequently when H1 and H2 receptors are located on the same cells,
a. To tag pathogenic microorganisms for destruction by neutrophils and they act in what fashion?
macrophages a. Synergistically
b. To process pathogenic microorganisms so that activated lymphocytes b. Additively
can be created for acquired immunity c. Antagonistically
c. To destroy glycoprotein cell membranes of pathogenic microorganisms d. Agonistically
d. To promote anaphylatoxic activity, resulting in mast cell degranulation
20. Some older adults have impaired inflammation and wound healing
15. In the coagulation (clotting) cascade, the intrinsic and the extrinsic because of which problem?
pathways converge at which factor? a. Circulatory system cannot adequately perfuse tissues.
a. XII b. Complement and chemotaxis are deficient.
b. VII c. Underlying chronic illness(es) exists.
c. X d. Number of mast cells is insufficient.
d. V
21. Which chemical mediator derived from mast cells retracts
16. Which chemical interacts among all plasma protein systems by endothelial cells to increase vascular permeability and to cause leukocyte
degrading blood clots, activating complement, and activating the Hageman adhesion toendothelial cells?
factor? a. Chemokines
a. Kallikrein b. Prostaglandin E
b. Histamine c. Platelet-activating factor
c. Bradykinin d. Bradykinin
d. Plasmin
22. What is the inflammatory effect of nitric oxide (NO)?
17. How does the chemotactic factor affect the inflammatory process? a. Increases capillary permeability, and causes pain
a. By causing vasodilation around the inflamed area b. Increases neutrophil chemotaxis and platelet aggregation
b. By stimulating smooth muscle contraction in the inflamed area c. Causes smooth muscle contraction and fever
c. By directing leukocytes to the inflamed area d. Decreases mast cell function, and decreases platelet aggregation
d. By producing edema around the inflamed area
, 23. What is the correct sequence in phagocytosis? 28. Which cytokine is produced and released from virally infected host
a. Engulfment, recognition, fusion, destruction cells?
b. Fusion, engulfment, recognition, destruction a. IL-1
c. Recognition, engulfment, fusion, destruction b. IL-10
d. Engulfment, fusion, recognition, destruction c. TNF
d. IFN
24. When considering white blood cell differentials, acute inflammatory
reactions are related to elevations of which leukocyte? 29. Which manifestation of inflammation is systemic?
a. Monocytes a. Formation of exudates
b. Eosinophils b. Fever and leukocytosis
c. Neutrophils c. Redness and heat
d. Basophils d. Pain and edema
25. In the later stages of an inflammatory response, which phagocytic cell is 30. The acute inflammatory response is characterized by fever that is
predominant? produced by the hypothalamus being affected by what?
a. Neutrophils a. Endogenous pyrogens
b. Monocytes b. Bacterial endotoxin
c. Chemokines c. Antigen-antibody complexes
d. Eosinophils d. Exogenous pyrogens
26. In regulating vascular mediators released from mast cells, the role of 31. What occurs during the process of repair after tissue damage?
eosinophils is to release what? a. Nonfunctioning scar tissue replaces destroyed tissue.
a. Arylsulfatase B, which stimulates the formation of B lymphocytes b. Regeneration occurs; the original tissue is replaced.
b. Histaminase, which limits the effects of histamine during acute c. Resolution occurs; tissue is regenerated.
inflammation d. Epithelialization replaces destroyed tissue.
c. Lysosomal enzymes, which activate mast cell degranulation during acute
inflammation 32. What is the role of fibroblasts during the reconstructive phase of
d. Immunoglobulin E, which defends the body against parasites wound healing?
a. Generate new capillaries from vascular endothelial cells around the
27. What is a role of a natural killer (NK) cells? wound.
a. Initiation of the complement cascade b. Establish connections between neighboring cells and contract their
b. Elimination of malignant cells fibers.
c. Binding tightly to antigens c. Synthesize and secrete collagen and the connective tissue proteins.
d. Proliferation after immunization with antigen d. Provide enzymes that débride the wound bed of dead cells.
