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NR511 Final Exam Study Guide (Latest-2022, Version-2) / NR 511 Final Exam Study Guide / NR511 Week 8 Final Exam Study Guide: Differential Diagnosis and Primary Care Practicum: Chamberlain College of Nursing |Latest and Updated Guide| $15.49   Add to cart

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NR511 Final Exam Study Guide (Latest-2022, Version-2) / NR 511 Final Exam Study Guide / NR511 Week 8 Final Exam Study Guide: Differential Diagnosis and Primary Care Practicum: Chamberlain College of Nursing |Latest and Updated Guide|

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NR511 Final Exam Study Guide (Latest-2022, Version-1) / NR 511 Final Exam Study Guide / NR511 Week 8 Final Exam Study Guide: Differential Diagnosis and Primary Care Practicum: Chamberlain College of Nursing |Latest and Updated Guide|

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  • February 22, 2022
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  • 2021/2022
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1

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, Poststreptococcal glomerulonephritis is a serious complication of impetigo (ecthyma).
This condition develops within 1–2 weeks following infection. Poststreptococcal
glomerulonephritis manifests with edema, hypertension, fever, and hematuria.

The diagnosis of impetigo often can be made on the basis of clinical manifestations.
A Gram stain and culture of pus or exudate is recommended to identify whether S.
aureus and/or a beta-hemolytic Streptococcus is the cause. However, treatment may be
initiated without these studies in patients with typical clinical presentations.

Bullous and nonbullous impetigo can be treated with either topical or oral therapy.
Topical therapy is used for patients with limited skin involvement whereas oral therapy is
recommended for patients with numerous lesions. Unlike impetigo, ecthyma should
always be treated with oral therapy.

Benefits of topical therapy include fewer side effects and lower risk for contributing to
bacterial resistance compared with oral therapy. Topical choices to treat impetigo
include the following medications for 5 days.
• Mupirocin three times daily
• Retapamulin twice daily

Extensive impetigo and ecthyma should be treated with an antibiotic effective for both S.
aureus and streptococcal infections unless cultures reveal only streptococci. Dicloxacillin
and cephalexin are appropriate treatments. A 7-day course of oral antibiotic treatment is
recommended. If only streptococci are detected in extensive impetigo or ecthyma, oral
penicillin is the preferred therapy.

MRSA impetigo can be treated with doxycycline, clindamycin, or trimethoprim-
sulfamethoxazole (Bactrim). Crusted lesions can be washed gently. Children can return
to school 24 hours after beginning an effective antimicrobial therapy. Draining lesions
should be kept covered.

Quiz:Sally, aged 25, presents with impetigo that has been diagnosed as infected with
staphylococcus. The clinical presentation is pruritic tender, red vesicles surrounded
by erythema with a rash that is ulcerating. She has not been adequately treated
recently. Which type of impetigo is this?
a. Bullous impetigo
b. Staphylococcal scalded skin syndrome (SSSS)
c. Nonbullous impetigo
d. Ecthyma

2. Staphylococcal Scalded Skin Syndrome
Caused by Staphylococcus aureus, it’s a variant of bullous impetigo:Epidermal necrosis
caused by bacterial exotoxins, resulting in the epithelial layer peeling off in large,
sheetlike pieces; mimics scalded-skin thermal burn.This serious infection is more
commonly seen in children and usually begins in the intertriginous areas.

3. Cellulitis

, Cellulitis is an acute infection as a result of bacterial entry via breaches in the skin
barrier. As the bacteria enter the subcutaneous tissues, their toxins are released which
causes an inflammatory response.

 Cellulitis and erysipelas is almost always a unilateral infection with the most
common site of infection being the lower extremities.
 Cellulitis involves the deeper dermis and subcutaneous fat.
 Cellulitis is observed most frequently among middle-aged individuals and older
adults.
 The vast majority of pathogens associated with cellulitis are from either
Streptococcus or Staphlococcus bacteria. The most common are beta-hemolytic
streptococci (groups A, B, C, G, and F), and S. aureus (gram +)
 Both erysipelas and cellulitis manifest with areas of skin erythema, edema,
warmth and pain. Fever may be present. Additional manifestations of cellulitis
and erysipelas include lymphangitis and inflammation of regional lymph nodes.
Edema surrounding the hair follicles may lead to dimpling in the skin, creating an
appearance reminiscent of an orange peel texture called "peaud'orange".
 Cellulitis may present with or without purulence
 patients with cellulitis tend to have a more indolent course with development of
localized symptoms over a few days.
 Many patients with cellulitis have underlying such as tinea pedis, lymphedema,
and chronic venous insufficiency. In such patients, treatment should be directed
at both the infection and the predisposing condition if modifiable.
 Patients with cellulitis or erysipelas in the absence of abscess or purulent
drainage should be managed with empiric antibiotic therapy. Patients with
drainable abscess should undergo incision and drainage.
I. Describe an appropriate empiric antibiotic treatment plan for cellulitis

 should be managed with empiric therapy for infection due to beta-hemolytic
streptococci and methicillin-susceptible Staphylococcus aureus (MSSA) with:
• Cephalexin 500 mg four times daily (alternative for mild penicillin allergy)
• Clindamycin 300 mg to 450 mg four times daily (alternative for severe penicillin
allergy)

 Good choices for uncomplicated cases of cellulitis that are not associated with
human or animal bites include dicloxacillin or cephalexin for 10 to 14 days.
 If pt has severe PCN allergy rx erythromycin
 If caused by animal or human bite: amoxicillin-clavulanic acid (augmentin) for 2
weeks
The coverage for MRSA is achieved by adding to amoxicillin one of the following:
Bactrim DS twice daily
Doxycycline 100 mg twice daily
Minocycline 200 mg orally once, then 100 mg orally every 12 hours
If clindamycin is used, no additional MRSA coverage is needed.

Risk factors for community-associated methicillin-resistant S. aureus (CA-MRSA) include the
following.

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