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Nur 3200 Exam 3 Prep | Study Guide | Questions And Answers | Nursing Exam Prep

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Prepare for NUR 3200 Exam 3 with a focused study resource designed to support effective nursing exam preparation and targeted review. This material provides practice questions covering important NUR 3200 concepts, helping you reinforce course knowledge, strengthen recall, and practice selecting the best answer in an exam-style setting. Use the questions to review essential nursing principles, patient care, assessment, safety, communication, clinical judgment, professional practice, and other relevant course concepts while identifying areas that may need additional attention. The resource is useful for structured study sessions, independent practice, and final review before Exam 3. Work through the questions to test your understanding, revisit challenging concepts, and develop greater confidence with nursing-style multiple-choice questions. Pair this study resource with your course notes, lectures, and assigned materials for a well-rounded preparation strategy as you get ready for the NUR 3200 Exam 3.

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NUR 3200 EXAM 3 PREP | STUDY GUIDE |
QUESTIONS AND ANSWERS | NURSING
EXAM PREP| 100% VERIFIED | A+
GUARANTEE
Updated Questions and Answers | 100% Verified Exam Prep

,Sclerosis Hardening or thickening of a body part


Sclerodactyly Hardening or thickening of the skin, particularly the hands (can cause fingers to turn
inward, like a grip)


Ankylosing Fusion of vertebrae bodies, particularly in the posterior back, that changes the shape of
the bones to become more squarelike


Arthropathy Disease of the joints (i.e., arthritis)


Arthritis Inflammation of the joints


Polyarticular Involving 5 or more joints (i.e., polyarthritis= pain in 5+ joints of the body)


Symmetric Both sides match


Asymmetric Both sides do not match- Disease process effects one side more than the other, or only
affects one side


Rheumatoid/ism/tic Various conditions characterized by inflammation, pain (particularly in joints or fibrous
tissue), and stiffness


Autoimmune Our own antibodies are attacking our body's cells, because they no longer recognize
them as self


Rheumatoid (Rheumatic) Arthritis Genetic + environmental exposure
Autoimmune
Antigens attack collagen
Phagocytic enzymes destroy joint cartilage


Rheumatoid Arthritis Vulnerable Populations More common in women (possibly some connection with estrogen) and aging


Rheumatoid Arthritis Joint Manifestations Symmetric and polyarticular symptoms in freely moving joints
Rheumatoid nodules
Reduced range of motion
Pain and stiffness in morning after waking
Swelling and thickening of synovial space causes ulnar deviation and swan neck

, Rheumatoid Arthritis Diagnosis Rheumatoid factor (self-produced antibody)
Increased erythrocyte sedimentation rate


Rheumatoid Arthritis Treatment Bee-venom


Systemic Lupus Erythematosus (SLE) Genetic + environmental exposure
Autoimmune
Early onset (15-44)
Autoantibodies and immune complexes form with B-cell hyperactivity that produce
autoantibodies (ANA)
Nuclear of antibodies is attacked


SLE Triggers Hormones, environment, UV light


SLE Vulnerable Populations Females
African Americans, Hispanics, Asians


SLE Manifestations Butterfly rash on face
Sun sensitivity
Glomerulonephritis
"The Great Immitator"


SLE Diagnosis Increased ANA, but is is not as simple as a lab test - must meet 4/11 criteria
Anti-DNA test


SLE Treatment Prevent progressive loss of organ function


Systemic Sclerosis/Sclerodema Autoimmune disease of connective tissue
Excessive collagen deposition in skin and internal organs
Fibrosis thickens skin


Systemic Sclerosis Vulnerable Populations Females

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