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Pathophysiology
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NURS 5315 Advanced Pathophysiology
UTA Exam 1 | Q&A 2026
NURS 5315 Advanced Pathophysiology
NURS
UTA 5315
ExamAdvanced
1 _ Q&A 2026
Pathophysiology
(1).pdf NURS
UTA 5315
ExamAdvanced
1 _ Q&A 2026
Pathophysiology
(1).pdf UTA Exam 1 _ Q&A 2026 (1).pdf
,NURS 5315 Advanced Pathophysiology UTA Exam 1 2026
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Terms (138)
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What can Reactive Oxygen Species cause?
Heart disease, Alzheimers, Parkinsons, Amyotrophic Lateral Sclerosis (ALS), CV disease,
HTN, HLD, DM, ischemic heart disease, HF, OSA. Lipid perioxidation, damage proteins,
fragment DNA, less protein synthesis, chromatin destruction, damage mitochondria
What is the body's defense against ROS?
Antioxidants (Vitamin E, Vitamin C, cysteine, glutathione, albumin, ceruloplasmin, transferrin)
NURS 5315 Advanced Pathophysiology UTA Exam 1 2026
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How are free radicals produced?
1. Normal cellular respiration
2. Absorption of extreme energy sources (radiation, UV light)
3. Metabolism of exogenous chemicals, drugs, and pesticides
4. Transition of metals
5. Nitric oxide acting like a chemical mediator and a free radical
NURS 5315 Advanced Pathophysiology UTA Exam 1 2026
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action potential
Process of conducting an impulse. Activates the neuron --> the neuron depolarizes --> then
repolarizes
Threshold potential
Point at which depolarization must reach in order to initiate an action potential
Hypokalemia and action potentials
HYPERpolarized (more negative, ex. -100). Less excitable. Decreased neuromuscular
excitability: weakness, smooth muscle atony, paresthesia, cardiac dysrhythmias
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