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NURS 5315 Advanced Pathophysiology
UTA Exam 1 | Study Guide
NURS 5315 Advanced Pathophysiology
NURS
UTA5315
ExamAdvanced
1 _ StudyPathophysiology
Guide (1).pdfNURS
UTA5315
ExamAdvanced
1 _ StudyPathophysiology
Guide (1).pdf UTA Exam 1 _ Study Guide (1).pdf
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What can Reactive Oxygen Species cause?
Heart disease, Alzheimers, Parkinsons, Amyotrophic Lateral Sclerosis (ALS), CV disease,
HTN, HLD, DM, ischemic heart disease, HF, OSA. Lipid perioxidation, damage proteins,
fragment DNA, less protein synthesis, chromatin destruction, damage mitochondria
What is the body's defense against ROS?
Antioxidants (Vitamin E, Vitamin C, cysteine, glutathione, albumin, ceruloplasmin, transferrin)
How are free radicals produced?
1. Normal cellular respiration
2. Absorption of extreme energy sources (radiation, UV light)
3. Metabolism of exogenous chemicals, drugs, and pesticides
4. Transition of metals
5. Nitric oxide acting like a chemical mediator and a free radical
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action potential
Process of conducting an impulse. Activates the neuron --> the neuron depolarizes --> then
repolarizes
Threshold potential
Point at which depolarization must reach in order to initiate an action potential
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Hypokalemia and action potentials
HYPERpolarized (more negative, ex. -100). Less excitable. Decreased neuromuscular
excitability: weakness, smooth muscle atony, paresthesia, cardiac dysrhythmias
Hyperkalemia and action potentials
HYPOpolarized (more positive, ex: closer to 0). More excitable. Peaked T waves.
When resting membrane potential=threshold potential, it is BAD = cardiac standstill,
paresthesia, paralysis
Hypocalcemia and action potentials
Increased permeability to Na+. More excitable. Tetany, hyperreflexia, circumoral paresthesia,
seizures, dysrhythmias.
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