NSG 3850 Exam 3 Actual Exam Questions & Verified Answers for Galen College of Nursing provides comprehensive exam-focused coverage of gastrointestinal, hepatobiliary, and liver-related nursing concepts. This resource focuses on gallstone formation, cholelithiasis, cholecystitis, liver disease, hepatitis, risk factors, diagnostic testing, clinical manifestations, immune responses, and important hepatitis terminology.
Topics covered include:
• Cholelithiasis — mechanisms of gallstone formation, including cholesterol supersaturation, crystal nucleation, bile stasis, and stone growth.
• Gallstone Risk Factors — prolonged fasting, rapid weight loss, oral contraceptive use, diabetes, spinal cord injury, total parenteral nutrition (TPN), and pregnancy.
• Cholesterol Crystals — understanding their role in initiating gallstone formation.
• Cholecystitis — inflammation of the gallbladder associated with the continued presence of gallstones.
• Acute Cholecystitis — progression of untreated inflammation, including possible gangrene, gallbladder rupture, and peritonitis.
• Chronic Cholecystitis — predisposing factors such as diabetes and obesity and potential long-term complications.
• Chronic Gallbladder Complications — biliary sepsis, scarring, calcification, porcelain gallbladder, and associated clinical concerns.
• Cholecystitis Diagnostics — ultrasound, CT, MRI, HIDA scanning, MRCP, and ERCP.
• Liver Disease — jaundice, impaired bilirubin metabolism, reduced clotting factors, hypoalbuminemia, portal hypertension, muscle wasting, ascites, impaired absorption of fat-soluble vitamins, and altered lipid metabolism.
• Liver Disease and Lipoprotein Metabolism — understanding how impaired liver function can contribute to dyslipidemias and elevated triglycerides.
• Hepatitis Terminology — understanding the “anti-” designation in relation to antibodies and immunity against specific hepatitis viruses.
• Hepatitis A and E — fecal-oral transmission and important infection-control considerations.
• Hepatitis A — incubation period, supportive management, avoidance of hepatotoxic substances, and clinical manifestations.
• Hepatitis A Manifestations — jaundice, abdominal pain, rashes, glomerulonephritis, and angioedema.
• Hepatitis Immunization — review of hepatitis viruses for which immunization is available, including hepatitis A and B.
• Hepatitis A Antibodies — IgM associated with acute infection and IgG associated with previous infection or immunity.
• Clinical Nursing Assessment — recognizing important signs, symptoms, risk factors, complications, diagnostic procedures, and disease-management concepts.
This NSG 3850 Exam 3 resource is designed for Galen College of Nursing students reviewing hepatobiliary and liver disorders, gallstone disease, cholecystitis, hepatitis, laboratory and diagnostic concepts, and related clinical manifestations for exam preparation.
Content preview
NSG 3850 EXAM 3
ACTUAL EXAM 2026 |
ALL QUESTIONS AND
CORRECT ANSWERS |
VERIFIED ANSWERS |
UPDATED VERSION
| GALEN COLLEGE OF
NURSING
,three phases: supersaturation of bile with cholesterol causing precipitation of
cholesterol, nucleation of crystals, hypermobility (stasis of bile) allowing stone
growth
cholelithiasis
T/F: crystals of cholesterol may ignite gallstone formation
true
T/F: excessive somatostatin stimulates the gallbladder
true
T/F: crystals of cholesterol may initiate gallstone formation
true
risk factors of this include prolonged fasting, rapid weight loss, oral
contraceptives, diabetes, spinal cord injury, TPN, pregnancy
cholelithiasis
inflammation of the gallbladder wall related to continued presence of gallstones
cholecystitis
, if untreated, escalates; gangrene may occur. can rupture gallbladder and cause
peritonitis
acute cholecystitis
predisposing factors: diabetes and obesity
chronic cholecystitis
can lead to complications like biliary sepsis and scarring/calcified (porcelain
gallbladder); causes higher risk for cancer.
chronic cholcystitis
diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP
cholecystitis
S/S: jaundice (from impaired bilirubin metabolism), decreased clotting
factors, hypoalbuminemia (from ascites/edema related to low serum oncotic
pressure), portal HTN, muscle wasting, ascites, impaired absorption of vit A, D,
E, K, hypertriglyceridemia
liver disease
can disrupt lipoprotein metabolism causing dyslipidemias.
liver disease