NURS 611 ADVANCED 2 PATHOPHYSIOLOGY EXAM
20206 UPDATE QUESTIONS AND CORRECT
VERIFIED ANSWERS ALREADY GRADED A+
IgE - ANS-type I hypersensitivity reactions are mediated by antigen-specific _______ and
products of tissue mast cells
alloimmunity - ANS-blood transfusion reactions are an example of _______________
eosinophils - ANS-the leukocyte activated during an IgE-mediated, or type I hypersensitivity
reaction
antigens on the cell surface - ANS-antibodies bind to this as the mechanism of action that
results in type II hypersensitivity reactions
complement-mediated cell lysis - ANS-when mismatched blood is administered causing an ABO
incompatibility, the eyrthrocytes are destroyed by ____________
natural killer cells - ANS-target cells are destroyed by what type of cells in a type II
hypersensitivity reaction
modulation - ANS-graves disease is an example of which type II hypersensitivity reaction
type III hypersensitivity reactions - ANS-this type of hypersensitivity reaction is a result of
antibodies binding to soluble antigens that were released into body fluids and the immune
complexes being deposited in the tissues
,type IV hypersensitivity - ANS-this type of hypersensitivity reaction causes lymphokine-
producing Th1 cells directly attacking and destroying cellular targets
raynaud's phenomenon - ANS-type III hypersensitivity reaction that is due to immune
complexes that are deposited in capillary beds, blocking circulation.
Lupus - ANS-tissue damage disease caused by the deposition of circulating immune complexes
containing an antibody against the host DNA
A and B - ANS-a person with type O blood is likely to have high titers of which anti-antibodies?
IV - ANS-urticaria is a manifestation of which type of hypersensitivity reaction?
Graves disease - ANS-result of autoantibodies binding to TSH receptor sites
it contains no antigens - ANS-O blood is considered the universal donor because ______
affecting the receptor for TSH, causing neonatal hyperthyroidism - ANS-graves disease is an
autoimmune disease that results in which maternal antibody?
t lymphocytes and macrophages - ANS-a positive PPD is the result of ________ and
____________
poison ivy, neomycin, nickle, detergents - ANS-potential causes for type IV hypersensitivity
bacteria - ANS-phagocytosis involves neutrophils actively attacking, engulfing, and destroying
these microorganisms
, viruses - ANS-what doe natural killer cells actively attack
exotoxins - ANS-proteins released during bacteria growth
CD4+ Th cells, macrophages, and natural killer cells - ANS-primary target cells of HIV
CD4+ Th cells - ANS-AIDS produces a striking decrease in the number of which cells?
cortisol - ANS-helper t1 cells suppress this hormone during a stress response
hyopalbuminemia - ANS-this causes impaired circulation of epinephrine and norepinephrine
during a central stress response
endogenous pyrogens - ANS-considering the hypothalamus, a fever is produced by ______
_______acting directly on the hypothalamus
adaptation - ANS-exhaustion occurs if stress continues when which stage of general adaptation
syndrome is not successful?
alarm stage - ANS-stage of general adaptation syndrome that activates that stimulates the
hypothalamus
adrenal cortex - ANS-released stress-induced cortisol results in the stimulation of
gluoneogenesis by affecting this structure
cortisol - ANS-this hormone increases the formation of glucose form amino acids and free fatty
acids
20206 UPDATE QUESTIONS AND CORRECT
VERIFIED ANSWERS ALREADY GRADED A+
IgE - ANS-type I hypersensitivity reactions are mediated by antigen-specific _______ and
products of tissue mast cells
alloimmunity - ANS-blood transfusion reactions are an example of _______________
eosinophils - ANS-the leukocyte activated during an IgE-mediated, or type I hypersensitivity
reaction
antigens on the cell surface - ANS-antibodies bind to this as the mechanism of action that
results in type II hypersensitivity reactions
complement-mediated cell lysis - ANS-when mismatched blood is administered causing an ABO
incompatibility, the eyrthrocytes are destroyed by ____________
natural killer cells - ANS-target cells are destroyed by what type of cells in a type II
hypersensitivity reaction
modulation - ANS-graves disease is an example of which type II hypersensitivity reaction
type III hypersensitivity reactions - ANS-this type of hypersensitivity reaction is a result of
antibodies binding to soluble antigens that were released into body fluids and the immune
complexes being deposited in the tissues
,type IV hypersensitivity - ANS-this type of hypersensitivity reaction causes lymphokine-
producing Th1 cells directly attacking and destroying cellular targets
raynaud's phenomenon - ANS-type III hypersensitivity reaction that is due to immune
complexes that are deposited in capillary beds, blocking circulation.
Lupus - ANS-tissue damage disease caused by the deposition of circulating immune complexes
containing an antibody against the host DNA
A and B - ANS-a person with type O blood is likely to have high titers of which anti-antibodies?
IV - ANS-urticaria is a manifestation of which type of hypersensitivity reaction?
Graves disease - ANS-result of autoantibodies binding to TSH receptor sites
it contains no antigens - ANS-O blood is considered the universal donor because ______
affecting the receptor for TSH, causing neonatal hyperthyroidism - ANS-graves disease is an
autoimmune disease that results in which maternal antibody?
t lymphocytes and macrophages - ANS-a positive PPD is the result of ________ and
____________
poison ivy, neomycin, nickle, detergents - ANS-potential causes for type IV hypersensitivity
bacteria - ANS-phagocytosis involves neutrophils actively attacking, engulfing, and destroying
these microorganisms
, viruses - ANS-what doe natural killer cells actively attack
exotoxins - ANS-proteins released during bacteria growth
CD4+ Th cells, macrophages, and natural killer cells - ANS-primary target cells of HIV
CD4+ Th cells - ANS-AIDS produces a striking decrease in the number of which cells?
cortisol - ANS-helper t1 cells suppress this hormone during a stress response
hyopalbuminemia - ANS-this causes impaired circulation of epinephrine and norepinephrine
during a central stress response
endogenous pyrogens - ANS-considering the hypothalamus, a fever is produced by ______
_______acting directly on the hypothalamus
adaptation - ANS-exhaustion occurs if stress continues when which stage of general adaptation
syndrome is not successful?
alarm stage - ANS-stage of general adaptation syndrome that activates that stimulates the
hypothalamus
adrenal cortex - ANS-released stress-induced cortisol results in the stimulation of
gluoneogenesis by affecting this structure
cortisol - ANS-this hormone increases the formation of glucose form amino acids and free fatty
acids