NURS 611 (MARYVILLE)ACTUAL EXAM 4 2026
LATEST UPDATE QUESTIONS AND CORRECT
VERIFIED ANSWERS ALREADY GRADED A+
What vascular changes occur in cirrhosis? - ANS-new vascular channels form shunts, and blood
from the portal vein bypasses the liver, contributing to portal hypertension, metabolic
alterations, and toxin accumulation.
What is the most common clinical manifestation of portal hypertension? - ANS-esophageal
varices.
What is the most common manifestation of congestive splenomegaly? - ANS-thrombocytopenia
(decreased platelet count).
What is hepatic encephalopathy the result of? - ANS-from a combination of biochemical
alterations that affect neurotransmission. Liver dysfunction and colalteral vessles that shunt
blood around the liver to the systemic circulation permit neurotoxins and other harmful
substances absorbed from the GI tract to accumulate and cirulate freely to the brain.
What are the most hazardous substances with hepatic encephalopathy? - ANS-end-products of
intestinal protein digestion, particularly ammonia, which cannot be converted to urea by the
diseased liver.
What is the cardinal symptom of acute pancreatitis? - ANS-epigastric or midabdominal pain.
The pain mya radiate to the back because of the retroperitoneal location of the pancreas.
What accompanies the inflammatory response with acute pancreatitis? - ANS-fever and
leukocytosis.
, What are indications of complications with pancreatitis? - ANS-hypocalcemia, hyotension,
shock, tachypena and hypoxemia are indicative of Sepsis and ARDS.
What accounts for the most deaths from pancreatitis? - ANS-SIRS and MODS account for most
deaths with severe pancreatitis.
Transient hyperglycemia occurs during pancreatitis because - ANS-damaged A cells in the
pancreatic islets by release glucagon.
What is primary dysmenorrhea attributed to? - ANS-excessive endometrial prostaglandin
productions. Women with painful periods produce 10 times as much prostaglandin.
What is prostaglandin? - ANS-it is a potent myometrial stimulant and vasoconstrictor.
What do elevated levels of prostaglandin cause? - ANS-uterine hypercontractility, decreased
blood flow to the uterus, and increased nerve hypersensitivity, thus resulting in pain.
What does secondary dysmenorrhea result from? - ANS-disorders such as endometriosis (the
most common cause), endometritis (infection), pelvic inflammatory disease, obstructive
uterine or baginal anomalies, uterine fibroids, polyps, tumors, ovarian cysts, pelvic congestion
syndrome, or nonhormonal IUDs.
What is the cardinal feature of PCOS? - ANS-a hyperandrogenic state.
What aggravates the hyperandrogenic state of PCOS? - ANS-glucose intolerance/insulin
resistance (IR) and hyperinsulinemia often run parallel and aggravate the hyperandrogenic
state of PCOS.
LATEST UPDATE QUESTIONS AND CORRECT
VERIFIED ANSWERS ALREADY GRADED A+
What vascular changes occur in cirrhosis? - ANS-new vascular channels form shunts, and blood
from the portal vein bypasses the liver, contributing to portal hypertension, metabolic
alterations, and toxin accumulation.
What is the most common clinical manifestation of portal hypertension? - ANS-esophageal
varices.
What is the most common manifestation of congestive splenomegaly? - ANS-thrombocytopenia
(decreased platelet count).
What is hepatic encephalopathy the result of? - ANS-from a combination of biochemical
alterations that affect neurotransmission. Liver dysfunction and colalteral vessles that shunt
blood around the liver to the systemic circulation permit neurotoxins and other harmful
substances absorbed from the GI tract to accumulate and cirulate freely to the brain.
What are the most hazardous substances with hepatic encephalopathy? - ANS-end-products of
intestinal protein digestion, particularly ammonia, which cannot be converted to urea by the
diseased liver.
What is the cardinal symptom of acute pancreatitis? - ANS-epigastric or midabdominal pain.
The pain mya radiate to the back because of the retroperitoneal location of the pancreas.
What accompanies the inflammatory response with acute pancreatitis? - ANS-fever and
leukocytosis.
, What are indications of complications with pancreatitis? - ANS-hypocalcemia, hyotension,
shock, tachypena and hypoxemia are indicative of Sepsis and ARDS.
What accounts for the most deaths from pancreatitis? - ANS-SIRS and MODS account for most
deaths with severe pancreatitis.
Transient hyperglycemia occurs during pancreatitis because - ANS-damaged A cells in the
pancreatic islets by release glucagon.
What is primary dysmenorrhea attributed to? - ANS-excessive endometrial prostaglandin
productions. Women with painful periods produce 10 times as much prostaglandin.
What is prostaglandin? - ANS-it is a potent myometrial stimulant and vasoconstrictor.
What do elevated levels of prostaglandin cause? - ANS-uterine hypercontractility, decreased
blood flow to the uterus, and increased nerve hypersensitivity, thus resulting in pain.
What does secondary dysmenorrhea result from? - ANS-disorders such as endometriosis (the
most common cause), endometritis (infection), pelvic inflammatory disease, obstructive
uterine or baginal anomalies, uterine fibroids, polyps, tumors, ovarian cysts, pelvic congestion
syndrome, or nonhormonal IUDs.
What is the cardinal feature of PCOS? - ANS-a hyperandrogenic state.
What aggravates the hyperandrogenic state of PCOS? - ANS-glucose intolerance/insulin
resistance (IR) and hyperinsulinemia often run parallel and aggravate the hyperandrogenic
state of PCOS.
