CMSRN PRACTICE TEST 15 ACTUAL
QUESTIONS AND CORRECT ANSWERS
◉ specific location on the abdomen, approximately one-third of the
distance from the anterior superior iliac spine to the umbilicus,
where tenderness is often felt in cases of acute appendicitis.
Answer: mcburnys point
◉ s/s apendicitis
Answer: a history of anorexia and periumbilical or epigastric pain
nausea, RLQ pain ( McBurney's point), and vomiting
rebound tenderness and pain on percussion
Rovsing's sign (pain in the RLQ that occurs with palpation of LLQ)
obturator sign (abdominal pain that occurs when the hip is rotated)
psoas sign (abdominal pain that occurs when flexing the hip when
pressure is applied to the knee)
◉ hypoCa s/s, ekg changes
Answer: cardiovascular and neuro changes, prolonged ST segment
and QT interval
◉ cause of peaked T wave
,Answer: MI, hyperK
◉ cause of prominent U wave
Answer: hypoK
◉ widened T wave cause
Answer: hyperCa
◉ compartment syndrome s/s
Answer: pain, paresthesia, paralysis, pulselessness, poikilothermia
◉ most common s/s pulm embolism
Answer: sudden onset chest pain, followed by dyspnea, tachypnea,
possibly hemoptysis, tachycardia, restlessness, cyanosis,
apprehension
◉ dig tox early s/s
Answer: The most common early manifestations of toxicity include
gastrointestinal disturbances such as nausea, vomiting, and
anorexia. Headaches, fatigue, drowsiness, confusion, and nightmares
can also occur early. Ocular disturbances, personality changes, and
facial pain are also signs of toxicity but are not early manifestations.
,◉ Pneumocystis jiroveciI pneumonia (PCP) secondary to
Answer: HIV - this is an opportunistic fungal infx in
immunocompromised pts
◉ how long til streptococcal pharyngitis infection can develop to
acute streptococcal glomerulonephritis?
Answer: 7-14 days
◉ oxycodone adverse effects
Answer: Side and adverse effects include respiratory depression,
drowsiness, hypotension, constipation, urinary retention, nausea,
vomiting, and tremors.
◉ resp alkalosis s/s
Answer: Clinical manifestations include light-headedness, confusion,
lethargy, tachycardia, cardiac arrhythmias related to hypokalemia,
nausea/vomiting, epigastric pain, and circumoral or peripheral
paresthesia. caused by tachypnea
◉ Electrolyte disorders frequently associated with acute kidney
injury (AKI) and advanced CKD (particularly stages 4 and 5)
Answer: hypernatremia, hypercalcemia, hyperkalemia, and
hyperphosphatemia. Other conditions can include decreased
bicarbonate levels (i.e., metabolic acidosis).
, ◉ laboratory findings consistent with disseminated intravascular
coagulation (DIC)
Answer: Increased D-dimer, prolonged activated partial
thromboplastin time (aPTT), decreased platelet count
◉ DIC
Answer: Disseminated intravascular coagulation is a diffuse
accelerated response to a stimulus that activates the coagulation
cascade and produces clotting and thrombus formation, followed by
bleeding and hemorrhage throughout the circulatory system. There
is a rapid depletion of platelets and clotting factors and excess
production of fibrin degradation products, which act as
anticoagulants.
Signs and symptoms include petechia, purpura, ecchymosis,
hematomas, and prolonged oozing or bleeding from orifices,
venipuncture sites, or incisions. Increased D-dimer, prolonged
activated partial thromboplastin time (aPTT), decreased platelet
count
◉ earliest detectable s/s of ARDS
Answer: tachypnea. Other vital sign measurements reveal increased
blood pressure and tachycardia. Tachypnea is followed by increasing
respiratory effort and accessory muscle use, and cyanosis ensues.
Breath sounds may be clear or consist of fine inspiratory crackles or
QUESTIONS AND CORRECT ANSWERS
◉ specific location on the abdomen, approximately one-third of the
distance from the anterior superior iliac spine to the umbilicus,
where tenderness is often felt in cases of acute appendicitis.
Answer: mcburnys point
◉ s/s apendicitis
Answer: a history of anorexia and periumbilical or epigastric pain
nausea, RLQ pain ( McBurney's point), and vomiting
rebound tenderness and pain on percussion
Rovsing's sign (pain in the RLQ that occurs with palpation of LLQ)
obturator sign (abdominal pain that occurs when the hip is rotated)
psoas sign (abdominal pain that occurs when flexing the hip when
pressure is applied to the knee)
◉ hypoCa s/s, ekg changes
Answer: cardiovascular and neuro changes, prolonged ST segment
and QT interval
◉ cause of peaked T wave
,Answer: MI, hyperK
◉ cause of prominent U wave
Answer: hypoK
◉ widened T wave cause
Answer: hyperCa
◉ compartment syndrome s/s
Answer: pain, paresthesia, paralysis, pulselessness, poikilothermia
◉ most common s/s pulm embolism
Answer: sudden onset chest pain, followed by dyspnea, tachypnea,
possibly hemoptysis, tachycardia, restlessness, cyanosis,
apprehension
◉ dig tox early s/s
Answer: The most common early manifestations of toxicity include
gastrointestinal disturbances such as nausea, vomiting, and
anorexia. Headaches, fatigue, drowsiness, confusion, and nightmares
can also occur early. Ocular disturbances, personality changes, and
facial pain are also signs of toxicity but are not early manifestations.
,◉ Pneumocystis jiroveciI pneumonia (PCP) secondary to
Answer: HIV - this is an opportunistic fungal infx in
immunocompromised pts
◉ how long til streptococcal pharyngitis infection can develop to
acute streptococcal glomerulonephritis?
Answer: 7-14 days
◉ oxycodone adverse effects
Answer: Side and adverse effects include respiratory depression,
drowsiness, hypotension, constipation, urinary retention, nausea,
vomiting, and tremors.
◉ resp alkalosis s/s
Answer: Clinical manifestations include light-headedness, confusion,
lethargy, tachycardia, cardiac arrhythmias related to hypokalemia,
nausea/vomiting, epigastric pain, and circumoral or peripheral
paresthesia. caused by tachypnea
◉ Electrolyte disorders frequently associated with acute kidney
injury (AKI) and advanced CKD (particularly stages 4 and 5)
Answer: hypernatremia, hypercalcemia, hyperkalemia, and
hyperphosphatemia. Other conditions can include decreased
bicarbonate levels (i.e., metabolic acidosis).
, ◉ laboratory findings consistent with disseminated intravascular
coagulation (DIC)
Answer: Increased D-dimer, prolonged activated partial
thromboplastin time (aPTT), decreased platelet count
◉ DIC
Answer: Disseminated intravascular coagulation is a diffuse
accelerated response to a stimulus that activates the coagulation
cascade and produces clotting and thrombus formation, followed by
bleeding and hemorrhage throughout the circulatory system. There
is a rapid depletion of platelets and clotting factors and excess
production of fibrin degradation products, which act as
anticoagulants.
Signs and symptoms include petechia, purpura, ecchymosis,
hematomas, and prolonged oozing or bleeding from orifices,
venipuncture sites, or incisions. Increased D-dimer, prolonged
activated partial thromboplastin time (aPTT), decreased platelet
count
◉ earliest detectable s/s of ARDS
Answer: tachypnea. Other vital sign measurements reveal increased
blood pressure and tachycardia. Tachypnea is followed by increasing
respiratory effort and accessory muscle use, and cyanosis ensues.
Breath sounds may be clear or consist of fine inspiratory crackles or
