Neuro Final
Study online at https://quizlet.com/_d7ds8x
1. What do cranial neural crest
nerves develop
from?
2. Name the neu- Chiari Malformation
roanatomic ab- Tethered Cord
normalities or Hydrocephalus
complications as- Syrinx (syringomyelia and syringobulbia)
sociated with
spina bifida?
3. What type of Somatotopic
organization ex-
plains why the (NOTE central cord: FIRST lose pain and temp bilat in the UEs- at the level due to
UE's have more damage to anterior white commissure) then progress to UMN signs in bilat UEs
UMN signs than
LE's in central
cord syndrome?
4. What type of axoaxonic
synapse is most
likely involved in
presynaptic inhi-
bition at the neu-
romuscular junc-
tion
, Neuro Final
Study online at https://quizlet.com/_d7ds8x
5. The reason for Oligodendrocytes (MS) vs Schwann cells(GB). Schwann cells can reform and
the difference secrete Nerve Growth Factor
in prognosis be-
tween Guillain
Barre and MS
6. Death of the neu- Retrograde transneuronal degeneration (1st order neuron died because of dam-
rons in the DRG age to second order neuron)
that occurs in re-
sponse to dam- (Note: orthograde would be damaging DRG because of anterolateral damage)
age of the antero-
lateral tract in the
spinal cord would
be considered
7. Term for the dis- Wernicke's aphasia
order in which
your patient
speaks fluently
but cannot car-
ry on a conver-
sation or follow
commands
8. Lesion location Brainstem above the red nucleus (decorticate posturing)
and term for
when a comatose
patient has his
UE's flexed and
LE's extended
9.
Study online at https://quizlet.com/_d7ds8x
1. What do cranial neural crest
nerves develop
from?
2. Name the neu- Chiari Malformation
roanatomic ab- Tethered Cord
normalities or Hydrocephalus
complications as- Syrinx (syringomyelia and syringobulbia)
sociated with
spina bifida?
3. What type of Somatotopic
organization ex-
plains why the (NOTE central cord: FIRST lose pain and temp bilat in the UEs- at the level due to
UE's have more damage to anterior white commissure) then progress to UMN signs in bilat UEs
UMN signs than
LE's in central
cord syndrome?
4. What type of axoaxonic
synapse is most
likely involved in
presynaptic inhi-
bition at the neu-
romuscular junc-
tion
, Neuro Final
Study online at https://quizlet.com/_d7ds8x
5. The reason for Oligodendrocytes (MS) vs Schwann cells(GB). Schwann cells can reform and
the difference secrete Nerve Growth Factor
in prognosis be-
tween Guillain
Barre and MS
6. Death of the neu- Retrograde transneuronal degeneration (1st order neuron died because of dam-
rons in the DRG age to second order neuron)
that occurs in re-
sponse to dam- (Note: orthograde would be damaging DRG because of anterolateral damage)
age of the antero-
lateral tract in the
spinal cord would
be considered
7. Term for the dis- Wernicke's aphasia
order in which
your patient
speaks fluently
but cannot car-
ry on a conver-
sation or follow
commands
8. Lesion location Brainstem above the red nucleus (decorticate posturing)
and term for
when a comatose
patient has his
UE's flexed and
LE's extended
9.
