PTH 617 Exam 1
- exaggerated or inappropriate immune response
- increased response to the presence of an antigen referred to as an allergen
- causes tissue damage - ANS-hypersensitivity disorders
- individual is more susceptible to opportunistic infections
- decrease ability to protect self from proliferation of cancer cells - ANS-problems of
immunodeficiency
- neutralize toxins from bacterial infections
- mark antigen
- agglutination
- complement fixation sets off chain reaction
- initiate inflammation - ANS-destruction of antigen steps
- patterned input from sense organs in the skin produce pain
- spatial and temporal impulses into the CNS that result in the brain interpreting the stimulus -
ANS-pattern theory of pain
- separate nerve ending for various senses
- pain receptors and pain pathways
- when stimulated would always and only produce pain - ANS-specificity model of pain
- through release of cytokine stim the proliferation of B cells and their release of antibodies
- role in activation of killer T cells
- stim activity of macrophages - ANS-helper T cells role
-refer to another and conclude care
-refer to another and continue care
-receive referral from other HCP who concludes care
-receive referral from other HCP who is continuing care - ANS-referral types (4)
1st, 2nd, 3rd degree sprains, intraarticular ligaments heal poorly, release of matrix
metalloproteinases causes turnover of extracellular matrix - ANS-ligament injury
56 identified - ANS-autoimmune diseases
A delta fibers, immediate, localized, high threshold pressure and temperature - ANS-fast pain
pathway (neo)
a multiple system output activated by the brain based on perceived threat - ANS-pain
, a state of complete physical, mental, and social well being and not merely as the absence of
disease or infirmity - ANS-health (WHO definition)
accumulating macrophages, lymphocytes, and plasma cells - ANS-cardinal sign of chronic
inflammation
activated B cells divide to form plasma cells and memory cells - ANS-humoral response 2
activated when exposed to antigen/epitope, stim/released by IL2, secreted by T4 helpers,
search out and destroy antigen - ANS-cytotoxic (killer) T lymphocytes
actual movement of signal - ANS-transmission
afferent translating the stimulus into an impulse - ANS-transduction
aging - ANS-cell senescence
all cases of condition among those at risk of developing at one point in time - ANS-prevalence
allergic reactions - ANS-eosinophils and basophils
altered structure or number of chromosomes, single mutations of a gene, multiple gene
mutations interacting with environment - ANS-genetic factors (3 mechanisms of cell injury)
an interruption in the continuity of a tissue - ANS-injury
anaphylactic (allergies), hypersensitivity to a foreign substance cause body to overproduce
antibody IgE - ANS-type I hypersensitivity
anemia - ANS-iron deficiency
annulus is fibro cartilaginous, sik has peripheral blood supply but not inner, outer has neural
supply, inner aneural - ANS-disk
anoxia or hypoxia, loss of aerobic metabolism and decreased ATP synthesis, cell swells due to
accumulation of ions and fluid - ANS-results of ischemia
anti bacterial and anti viral, complement, neutralize toxins - ANS-IgG
antibodies - IgG, IgM, IgE, IgA, IgD
antigen recognition molecules - ANS-immunoglobulins
any substance or organism that is capable of setting off an immune response - ANS-antigen
- exaggerated or inappropriate immune response
- increased response to the presence of an antigen referred to as an allergen
- causes tissue damage - ANS-hypersensitivity disorders
- individual is more susceptible to opportunistic infections
- decrease ability to protect self from proliferation of cancer cells - ANS-problems of
immunodeficiency
- neutralize toxins from bacterial infections
- mark antigen
- agglutination
- complement fixation sets off chain reaction
- initiate inflammation - ANS-destruction of antigen steps
- patterned input from sense organs in the skin produce pain
- spatial and temporal impulses into the CNS that result in the brain interpreting the stimulus -
ANS-pattern theory of pain
- separate nerve ending for various senses
- pain receptors and pain pathways
- when stimulated would always and only produce pain - ANS-specificity model of pain
- through release of cytokine stim the proliferation of B cells and their release of antibodies
- role in activation of killer T cells
- stim activity of macrophages - ANS-helper T cells role
-refer to another and conclude care
-refer to another and continue care
-receive referral from other HCP who concludes care
-receive referral from other HCP who is continuing care - ANS-referral types (4)
1st, 2nd, 3rd degree sprains, intraarticular ligaments heal poorly, release of matrix
metalloproteinases causes turnover of extracellular matrix - ANS-ligament injury
56 identified - ANS-autoimmune diseases
A delta fibers, immediate, localized, high threshold pressure and temperature - ANS-fast pain
pathway (neo)
a multiple system output activated by the brain based on perceived threat - ANS-pain
, a state of complete physical, mental, and social well being and not merely as the absence of
disease or infirmity - ANS-health (WHO definition)
accumulating macrophages, lymphocytes, and plasma cells - ANS-cardinal sign of chronic
inflammation
activated B cells divide to form plasma cells and memory cells - ANS-humoral response 2
activated when exposed to antigen/epitope, stim/released by IL2, secreted by T4 helpers,
search out and destroy antigen - ANS-cytotoxic (killer) T lymphocytes
actual movement of signal - ANS-transmission
afferent translating the stimulus into an impulse - ANS-transduction
aging - ANS-cell senescence
all cases of condition among those at risk of developing at one point in time - ANS-prevalence
allergic reactions - ANS-eosinophils and basophils
altered structure or number of chromosomes, single mutations of a gene, multiple gene
mutations interacting with environment - ANS-genetic factors (3 mechanisms of cell injury)
an interruption in the continuity of a tissue - ANS-injury
anaphylactic (allergies), hypersensitivity to a foreign substance cause body to overproduce
antibody IgE - ANS-type I hypersensitivity
anemia - ANS-iron deficiency
annulus is fibro cartilaginous, sik has peripheral blood supply but not inner, outer has neural
supply, inner aneural - ANS-disk
anoxia or hypoxia, loss of aerobic metabolism and decreased ATP synthesis, cell swells due to
accumulation of ions and fluid - ANS-results of ischemia
anti bacterial and anti viral, complement, neutralize toxins - ANS-IgG
antibodies - IgG, IgM, IgE, IgA, IgD
antigen recognition molecules - ANS-immunoglobulins
any substance or organism that is capable of setting off an immune response - ANS-antigen
