Pathophysiology - Exam 3 - UTA (Urban)
QUESTIONS AND VERIFIED CORRECT
SOLUTIONS || 100% GUARANTEED PASS
BRAND NEW VERSION
Arteriosclerosis -ANSWER✅✅✅✅Thick and Hardened artery
HTN | Smoking | Diabetes
Intima is damaged and walls stiffen. Elasticity and compliance decreases.
Atherosclerosis -ANSWER✅✅✅✅Fatty Deposits
Plaques form | tissue inflamed | High amount of LDL's (low-density lipoproteins=more
fat than protein)
Hypertension -ANSWER✅✅✅✅the force of the blood against the artery walls is too
high.
Pre - Stage I - Stage II
Preload -ANSWER✅✅✅✅Volume of blood returning from the heart.
Normal volume = 4-6ml
Afterload -ANSWER✅✅✅✅Amount of blood ejected from the heart
Contractility -ANSWER✅✅✅✅How effectively the heart works to eject blood "toned"
Arterial Insufficiency -ANSWER✅✅✅✅Muscle tone and the state of the lumen are
compromised.
Aneurysms -ANSWER✅✅✅✅A ballooning and weakened area in an artery.
Venous Disorders -ANSWER✅✅✅✅Chronic Venous Insufficiency (CVI)
Deep Vein Thrombosis (DVT)
Valvular Disorders -ANSWER✅✅✅✅Gravity Winning - not necessarily pathogenic
Valve Incompetence - pathology invlolved
, Cardiogenic Shock -ANSWER✅✅✅✅Heart suddenly can't pump enough blood to
meet your body's needs
What side of the heart is the Arterial side? -ANSWER✅✅✅✅The LEFT.
OXYGENATED blood is going "out" via aorta to all arteries and tissues
What side of the heart is the Venous side? -ANSWER✅✅✅✅The RIGHT.
DEOXYGENATED blood if flowing "in" from all the veins and tissues.
Inotropic -ANSWER✅✅✅✅POSITIVELY or NEGATIVELY modifying the force or
speed of contraction of muscles.
pos- pumps faster
neg- pumps less
Systemic Vasodilation -ANSWER✅✅✅✅from anaphalaxis or Sepsis
Analyphalyaxis -ANSWER✅✅✅✅severe, potentially life-threatening allergic reaction.
It can occur within seconds or minutes of exposure to something you're allergic to.
Low blood pressure (hypotension)
A weak and rapid pulse
In a case of HIGH preload -ANSWER✅✅✅✅Volume too LARGE- heart is working too
hard.
CAUSES: Fluid Excess States
1. heart failure
2. iatrogenic fluid overload
3. hormonal imbalances (SIADH)
In a case of LOW preload -ANSWER✅✅✅✅Volume too SMALL - not enough to
sustain good perfusion
CAUSES:
Fluid volume deficit (bleeding, dehydration, hormonal imbalances like DI)
Systemic vasodilation (blood pools in periphery- sepsis, anaphylaxis)
Afterload problems -ANSWER✅✅✅✅Resistance - ANY resistance to forward flow
RV Afterload Pathologies -ANSWER✅✅✅✅Pulmonary Vascular Resistance
Too High due to: athero/arteriosclerosis of pulmonary vasculature (narrowed stiff
pulmonary artery) Stiff noncompliant lung tissue (lung disease)
If RV afterload too high, can result in RIGHT heart failure. COR PULMONALE
QUESTIONS AND VERIFIED CORRECT
SOLUTIONS || 100% GUARANTEED PASS
BRAND NEW VERSION
Arteriosclerosis -ANSWER✅✅✅✅Thick and Hardened artery
HTN | Smoking | Diabetes
Intima is damaged and walls stiffen. Elasticity and compliance decreases.
Atherosclerosis -ANSWER✅✅✅✅Fatty Deposits
Plaques form | tissue inflamed | High amount of LDL's (low-density lipoproteins=more
fat than protein)
Hypertension -ANSWER✅✅✅✅the force of the blood against the artery walls is too
high.
Pre - Stage I - Stage II
Preload -ANSWER✅✅✅✅Volume of blood returning from the heart.
Normal volume = 4-6ml
Afterload -ANSWER✅✅✅✅Amount of blood ejected from the heart
Contractility -ANSWER✅✅✅✅How effectively the heart works to eject blood "toned"
Arterial Insufficiency -ANSWER✅✅✅✅Muscle tone and the state of the lumen are
compromised.
Aneurysms -ANSWER✅✅✅✅A ballooning and weakened area in an artery.
Venous Disorders -ANSWER✅✅✅✅Chronic Venous Insufficiency (CVI)
Deep Vein Thrombosis (DVT)
Valvular Disorders -ANSWER✅✅✅✅Gravity Winning - not necessarily pathogenic
Valve Incompetence - pathology invlolved
, Cardiogenic Shock -ANSWER✅✅✅✅Heart suddenly can't pump enough blood to
meet your body's needs
What side of the heart is the Arterial side? -ANSWER✅✅✅✅The LEFT.
OXYGENATED blood is going "out" via aorta to all arteries and tissues
What side of the heart is the Venous side? -ANSWER✅✅✅✅The RIGHT.
DEOXYGENATED blood if flowing "in" from all the veins and tissues.
Inotropic -ANSWER✅✅✅✅POSITIVELY or NEGATIVELY modifying the force or
speed of contraction of muscles.
pos- pumps faster
neg- pumps less
Systemic Vasodilation -ANSWER✅✅✅✅from anaphalaxis or Sepsis
Analyphalyaxis -ANSWER✅✅✅✅severe, potentially life-threatening allergic reaction.
It can occur within seconds or minutes of exposure to something you're allergic to.
Low blood pressure (hypotension)
A weak and rapid pulse
In a case of HIGH preload -ANSWER✅✅✅✅Volume too LARGE- heart is working too
hard.
CAUSES: Fluid Excess States
1. heart failure
2. iatrogenic fluid overload
3. hormonal imbalances (SIADH)
In a case of LOW preload -ANSWER✅✅✅✅Volume too SMALL - not enough to
sustain good perfusion
CAUSES:
Fluid volume deficit (bleeding, dehydration, hormonal imbalances like DI)
Systemic vasodilation (blood pools in periphery- sepsis, anaphylaxis)
Afterload problems -ANSWER✅✅✅✅Resistance - ANY resistance to forward flow
RV Afterload Pathologies -ANSWER✅✅✅✅Pulmonary Vascular Resistance
Too High due to: athero/arteriosclerosis of pulmonary vasculature (narrowed stiff
pulmonary artery) Stiff noncompliant lung tissue (lung disease)
If RV afterload too high, can result in RIGHT heart failure. COR PULMONALE
