Questions and Verified 100% Solutions (2026/2027) |A+
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S&S Hyperaldosteronism - Answers -HTN & Hypokalemia
TX of osteoporosis - Answers -Fosamax
Nasal Calcitonin
Meds to decreae osteoclastic activity
TX of Cushings - Answers -Spironolactone - Aldosterone Blocker
Remove tumor
Decrease steroid use
TX of Hypothyroidism - Answers -steroids: prednisone
Aldosterone: Florinef
Diet high in NaCl
Hypocortisolism & Hypoaldosteronism - Answers -Addison's
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,How does a person get HYPOCORTISOLISM - Answers -Autoimmune dz - autoantibodies attacks
ADRENAL GLANDS→ atrophy & hypofunction → CORTISOL'S not released
How does a person get HYPOALDSTERONISM - Answers -Malfunctioning of the PITUITARY
GLAND that doesn't release ACTH
S&S of HYPORTISOLISM - Answers -Hypoglycemia → weakness, fatigue, confusion.
Anorexia, N&V, Diarrhea →weight loss due to Adrenal Gland problem
Pancreas problems - Answers -DM I, II, DKA, HHNKS, metabolic problems, angiopathy,
Hypoglycemia
DM I pathos - Answers -hyperglycemia →glucosuria → polyuria →polydipsia →dehydration →
dry skin
no glucose → nutrition deficiency → weight loss while eating→ fatigue
no glucose → gluconeogenesis → ketoacidosis → acetone breath→ketonuria
Extreme case of DM I - Answers -DKA
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, Kussmaul respiration - Answers -fast deep breaths
S&S of DM I - Answers -Metabolic Acidosis
Diabetic Coma
Kussmaul Respiration → compensatory mechanism for riding CO2
DM II patho - Answers -obesity → insulin resistance bc fat cells are overwhelming → glucose
cannot be made for energy → hyperglycemia → insulin keeps getting secreted →
hyperinsulinemia → pancreas is tired → chronic organ damage, diabetic retinitis, fatigue, mild
polydipsia, polyuria
S&S of DM II - Answers -chronic organ damage, diabetic retinitis, fatigue, mild polydipsia,
polyuria
Long Term Diabetic Problems - Answers -Angtiopathy
Angiopathy - Answers -excessive glucose + excessive fat → damages to arteries
damages to nerves
damages to phagocytes
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