NSG 170 Metabolism - Diabetes || with Errorless Answers.
Type 1 Diabetes Mellitus correct answers - Must get insulin from an outside source. ***
BIGGEST difference between Type 1 and Type 2 Diabetes is that in Type 1 there is an
ABSENCE of endogenous (self made) insulin!
- Formally known as juvenile-onset or insulin-dependent diabetes.
- 5-10% of all people with diabetes
- Generally affects people under age 40 (but can occur in any age)
- Autoimmune disorder: body develops antibodies against insulin and/or pancreatic B cells that
produce insulin. Results in not enough insulin to survive.
SYMPTOMS: onset rapid / ACUTE
- diabetic ketoacidosis
* Polyuria (excessive urination)
* Polydipsia (excessive thirst)
* Polyphagia (excessive hunger)
- Weight loss
- Weakness
- Fatigue
**Remember the 3 P's!
- Genetic link
-Idiopathic diabetes
, - Latent autoimmune diabetes in adults (LADA)
Etiology and Pathophysiology of Type 1 Diabetes Mellitus:
- Autoimmune destruction of β-cells
- Total absence of insulin
- Manifestations develop when pancreas can no longer produce insulin—then rapid onset with
ketoacidosis
**All Type 1 Diabetics will eventually need insulin
Type 2 Diabetes Mellitus correct answers - Pancreas continues to produce SOME endogenous
insulin BUT not enough OR body does not use insulin effectively
- Non-insulin dependent diabetes
- Formally knows as adult-onset diabetes (AODM) or non-insulin-dependent diabetes (NIDDM)
- Most prevalent type 90-95% of all diabetics
SYMPTOMS: Nonspecific (may present Type 1 symptoms)
- Fatigue
- Recurrent infection
- Recurrent vaginal yeast or candidal infection
- Prolonged wound healing
- Visual changes
Type 1 Diabetes Mellitus correct answers - Must get insulin from an outside source. ***
BIGGEST difference between Type 1 and Type 2 Diabetes is that in Type 1 there is an
ABSENCE of endogenous (self made) insulin!
- Formally known as juvenile-onset or insulin-dependent diabetes.
- 5-10% of all people with diabetes
- Generally affects people under age 40 (but can occur in any age)
- Autoimmune disorder: body develops antibodies against insulin and/or pancreatic B cells that
produce insulin. Results in not enough insulin to survive.
SYMPTOMS: onset rapid / ACUTE
- diabetic ketoacidosis
* Polyuria (excessive urination)
* Polydipsia (excessive thirst)
* Polyphagia (excessive hunger)
- Weight loss
- Weakness
- Fatigue
**Remember the 3 P's!
- Genetic link
-Idiopathic diabetes
, - Latent autoimmune diabetes in adults (LADA)
Etiology and Pathophysiology of Type 1 Diabetes Mellitus:
- Autoimmune destruction of β-cells
- Total absence of insulin
- Manifestations develop when pancreas can no longer produce insulin—then rapid onset with
ketoacidosis
**All Type 1 Diabetics will eventually need insulin
Type 2 Diabetes Mellitus correct answers - Pancreas continues to produce SOME endogenous
insulin BUT not enough OR body does not use insulin effectively
- Non-insulin dependent diabetes
- Formally knows as adult-onset diabetes (AODM) or non-insulin-dependent diabetes (NIDDM)
- Most prevalent type 90-95% of all diabetics
SYMPTOMS: Nonspecific (may present Type 1 symptoms)
- Fatigue
- Recurrent infection
- Recurrent vaginal yeast or candidal infection
- Prolonged wound healing
- Visual changes
