Surgicall Nursingl Il Guidel |l Fortisl (Latestl
2026/l 2027l Update)l 100%l Verifiedl
Questionsl &l Answersl |l Gradel A
Q:l hypertensionl s/s
Answer:
SILENTl KILLERl (oftenl asymptomatic),l headache,l drowsiness,l MI,l visionl changes,l
stroke,l kidneyl failure,l dizziness,l blurredl vision,l blackouts,l irritability,l angina,l dyspnea,l orl
fatigue.
Q:l treatmentl forl htn
Answer:
Thel goalsl ofl treatmentl are
l -l Reductionl ofl highl bloodl pressure
-l Long-terml controll tol decreasel thel riskl ofl stroke,l heartattack,l lossl ofl vision,l andl
kidneyl disease.
Smokingl cessation,l weightl reduction,l sodiuml restriction,l alcoholl restriction,l exercise,l al
low-fatl diet,l stressl control,l Antihypertensivel therapy
Q:l primaryl htn
Answer:
unknownl cause
Q:l secondaryl htn
,Answer:
Elevatedl BPl withl al specificl cause
Q:l Primaryl HTNl riskl factors
Answer:
unknownl causel 90-95%
-l agel
afterl 50
-l etohl
males:l 2l drinksl perl day
female:l 1l drinkl perl day
-l dm
-l elevatedl seruml lipids
^l levelsl ofl cholesteroll +l triglyceridesl arel primaryl rfl forl atherosclerosis
hyperlipidemial morel commonl inl peoplel withl htn
-l ethnicity
blacks
-l ^l sml diet
-l familyl hx
-l gender
menl
womenl afterl 64
-l obesity
-l sedentaryl lifestyle
-l poverty
-l stress
-l smokers
^l riskl forl cvd
Q:l Secondaryl HTNl riskl factors
Answer:
-l cirrhosis
-l narrowingl ofl thel aorta
-l drugl related
,estrogenl replacementl therapy,l birthl controll pills,l corticosteroids,l nonsteroidall
antinflammatoryl drugs
l (ex:l cyclooxygenasel 2-l inhibitors),l snsl stimulantsl (coke,l monoaminel oxidase)
-l endocrinel disorders
ex:l cushingl syndrome,l thyroidl disorders,l pheochromocytoma
-l neurologicall disorder
ex:l brainl tumors,l quadriplegia,l traumaticl brainl injury
-l pregnancyl inducedl HTN,
-l renall diseasel
ex:l renall arteryl stenosis,l glomerulonephritis
-l sleepl apnea
Q:l classificationl ofl htn
Answer:
normall -l
SBPl (mmHg)l <120l /l DBPl (mmHg)l <80
elevatedl -
SBPl (mmHg)l <120-129l /l DBPl (mmHg)l <80
htnl stagel 1l -
SBPl (mmHg)l <130-139l /l DBPl (mmHg)l <80-89
htnl stagel 2l -
SBPl (mmHg)l <140l /l DBPl (mmHg)l <90
Q:l complicationsl ofl htn
Answer:
-l canl causel damagel tol arteries,l makingl theml lessl elastic.l Thisl placesl anl increasedl
workloadl onl thel heart.l Thisl mayl causel myocardiall infarction,l leftl ventricularl
hypertrophy,l andl congestivel heartl failure.
-l smalll vessell damagel tol thel brainl disruptsl circulationl andl mayl leadl tol transientl
ischemicl attacksl (TIAs)l andl stroke.
, -l mayl causel damagel tol thel smalll vesselsl ofl thel kidneyl andl mayl leadl tol kidneyl failure.
-l damagesl thel arteriesl ofl thel eye,l causingl thel formationl ofl clotsl orl occurrencel ofl
hemorrhagel thatl mayl leadl tol blurredl visionl orl blindness.
Q:l sodiuml intakel forl htn:
Answer:
nol morel thanl 1,500-2,000l milligramsl (mg).
Q:l Arteriosclerosis
Answer:
hardeningl ofl thel arteries
Etiology:
thickeningl ofl thel arteryl wallsl thatl progressesl tol hardeningl asl calciuml depositsl form.
Q:l Atherosclerosis
Answer:
arteryl narrowingl duel tol lipids.
Etiology:
lipidsl arel depositedl withinl thevessell wallsl andl combinel withl cells,l fibrin,l andl celldebrisl
tol forml plaques.
Q:l Diabetesl mellitusl speedl developmentl ofl both
Answer:
Arteriosclerosisl andl Atherosclerosis.
Q:l Hypertensionl isl al majorl factorl in