NSG 3850 exam 4 Questions & Answers | Latest Already
Graded A+ |Questions with Correct Answers 2026 latest
update!!
chronic cholcystitis
can lead to complications like biliary sepsis and scarring/calcified (porcelain
gallbladder); causes higher risk for cancer.
cholecystitis
diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP
liver disease
S/S: jaundice (from impaired bilirubin metabolism), decreased clotting
factors, hypoalbuminemia (from ascites/edema related to low serum
oncotic pressure), portal HTN, muscle wasting, ascites, impaired
absorption of vit A, D, E, K, hypertriglyceridemia
liver disease
can disrupt lipoprotein metabolism causing dyslipidemias.
"anti"
with hepatitis, what term means immunity or forming immunity to the
specific hep virus
,hep A and E
heps that are fecal oral route
Hep A
2-7 week incubation
hep A
treatment is supportive and avoiding hepatoyoxins
hep A
clinical manifestations: rashes, glomerulonephritis, angioedema, jaundice,
abd pain
A and B
can get immunized from these heps
IgG(previous infection) and IgM (acute infection)
antibodies the body makes against hep A
hep A
treatment is supportive and avoiding hepatotoxins like ETOH (alcohol)
, B, C, D
heps transmitted through sex, blood, contaminated needles. mother can
pass to baby
hep B
incubation period 2-6 months
hep B
hep that can lead to chronic liver infection
active hep infection
positive serologic markers HBsAg and HBeAg show what?
hep C
incubation period 2-26 weeks
hep A
can be asymptomatic. can cause fatigue, nausea, anorexia, jaundice
hep B
can cause rashes, serum sickness, angioedema, glomerulonephritis, abd pain,
fever
Graded A+ |Questions with Correct Answers 2026 latest
update!!
chronic cholcystitis
can lead to complications like biliary sepsis and scarring/calcified (porcelain
gallbladder); causes higher risk for cancer.
cholecystitis
diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP
liver disease
S/S: jaundice (from impaired bilirubin metabolism), decreased clotting
factors, hypoalbuminemia (from ascites/edema related to low serum
oncotic pressure), portal HTN, muscle wasting, ascites, impaired
absorption of vit A, D, E, K, hypertriglyceridemia
liver disease
can disrupt lipoprotein metabolism causing dyslipidemias.
"anti"
with hepatitis, what term means immunity or forming immunity to the
specific hep virus
,hep A and E
heps that are fecal oral route
Hep A
2-7 week incubation
hep A
treatment is supportive and avoiding hepatoyoxins
hep A
clinical manifestations: rashes, glomerulonephritis, angioedema, jaundice,
abd pain
A and B
can get immunized from these heps
IgG(previous infection) and IgM (acute infection)
antibodies the body makes against hep A
hep A
treatment is supportive and avoiding hepatotoxins like ETOH (alcohol)
, B, C, D
heps transmitted through sex, blood, contaminated needles. mother can
pass to baby
hep B
incubation period 2-6 months
hep B
hep that can lead to chronic liver infection
active hep infection
positive serologic markers HBsAg and HBeAg show what?
hep C
incubation period 2-26 weeks
hep A
can be asymptomatic. can cause fatigue, nausea, anorexia, jaundice
hep B
can cause rashes, serum sickness, angioedema, glomerulonephritis, abd pain,
fever
