type suptypes cause age lesion spicific thing trattment tast
Topical steroid are the mainstay of N o biomarker for the diagnosis of
-itchy erythema involves the face(cheeks)
the infant is irritable , restless at night due to treatment atopic dermatitis
-the extensor surfaces of the extremities
Infantile atopic dermatitis infancy,( 2months___ 2 years)
-The diaper area is usually spared ,
sever pruritus -Low potency steroid on (face and groin) swab of infected skin
-The course chronically relapsing and remitting -Medium potency on (trunk and extremities) radioallergosorbent(R AS T ) testing
-
-Strong steroid on (palmoplantar areas) Serum IgE .
-flexural areas of elbows , knees, wrist ,ankle
and the neck -Oral steroid in sever atopic dermatitis.
Atopic dermatitis (AD) Childhood atopic dermatitis Childhood (2-12 years)
-Persistent rubbing and scratching lead to
Subtopic 1
-Anti-histamin help in controlling the itching
same
lichenification.
( dry skin)
-Ear lobe eczema
-flexural involvement
-eczema of the nipple
-front and sides of neck, eyelids , face , feet -Immune modulator : pimecrolimus and
-eczema around the margins of the mouth
and hands tacrolimus inhibit inflammatory cytokine
Adult atopic dermatitis adult age -hyper pigmented, pruritus is sever
(cheilitis)
transcription in activated T-cell.
same
*black people , follicular eczema
-excoriation and lichenification common. -Cyclosporine in sever case refractory case
characterized by (discrete follicular
-30%develop atopic hand eczema
papules)
•The acute phase:
-Nickel: artificial jewellery, Hair pins.
-pruritus, erythema, edema, and vesicles
watches. button
-confined to the area of direct exposure.
-Paraphenylene diamine: hair dye
a cell-mediated (type IV) •chronic disease:
Allergic contact dermatitis (ACD) - delayed type hypersensitivity reaction caused Some people are susceptible -lichenified erythematous plaques with
-Chromium:cement, painter Avoidance is the mainstay of treat
Patch test is diagnostic
20% -Rubber:gloves ,Condom nent for ACD.
by skin contact with an environmental allergen. hyperkeratosis and fissuring
-Synthetic fiber:,acrylic and polyesters cloths.
-spread beyond the areas of direct exposure.
-formaldehyde: shampoo and cosmetics
·The hands, feet, and face (including the
-Topical medication: neomycin, tar.
eyelids)are some of the common sites forACD
successful treatment of contact dermatitis:
contact dermatitis identification of suhstances causing CD its
-a non-immunologic inflammation of the skin -Most irritant eczemas occur on( the hand) ·Examples of ICD:
avoidance.
caused by contact with a chemical, physical, or -Strong irritant: blisters, erosions, And ulcers -house wife dermatitis.
Irritant contact dermatitis (ICD) biologic agent. all people are suspected and pain rather than itching.. -cleaners. Hair dresser, food handler health
-Acute reaction require (wet soaks) or
Patch test isn't diagnostic
80% (compress) and topical steroid and antibiotic
eczema
-No hypersensitivity involved -mild irritant: produce (cumulative worker
cream or lotion.
-the reaction is immediate dermatitis)erythema and micro-vesiculation. -infant napkin dermatit
-In sever cases :oral steroid &oral antibiotic if
secondary infection.
contact with certain plant chemicals contain
Phytophotodermatitis (psoralin) can cause skin inflammation when less common ___________________________
exposed to sunlight.
• infant :self limited form
-1)low potency topical corticosteroids
(hydrocortisone 1% cream orlotion for a few
lesions: days), followed by2) topical imidazoles (2%
scalp,ears,face,nasolabial,eyebrows,uppereyelid, ketoconazole cream,lotion, or 1% shampo0),
posturicular area,presternal chest, uppertrunk, -Seborrhoeic dermatitis in fold axilla, groin, • Adults: tend to have chronic and recurrent
•The etiology is unclear but there are associations flares occur when sebaceous glands are most neck and intertriginous areas submamary seen in middle and elderly disease
seborrheic dermatitis with Malassezia yeasts, sebum secretion and active (firstfew months of life, and post
-sharply demarcated, yellow to red to brown,
specially obese woman -the patients should be informed that the aim
oftreatment will be to control rather than cure
composition,and certain drugs. puberty) -scalp of infant called cradle cap
greasy or bran-like scaling patches and plaques. -scaling on eye browse seborrheic blepharitis the disease.
-presence ot bright red and -Scalp seborrheic dermatitis treated by
macerated ill defined edge with sever itching (shampoos containing selenium sulfide,
imidazoles ,ketoconazole shampoo)
-oral antifungals should be reserved for severe
and refractory cases
-round lesions distributed commonly on the
extensor surtace of extremities . -topical steroid
--------------- Nummular eczema unknown Occurs in all age group common 55-65 years.
- coin- shape studded with vesicles on an
______
-Immune modulator topically applied.
_____________
erythematous base
-unknown cause.
deep seated sago like vesicles on the palms and
-slightly higher in atopy. common in young adults, rare in children and
----------- pompholyx -related to stress it may follow drug eruption. old age
lateral aspect of fingers and soles bilateral
symmetrical
---------- highy potency topical steroid ---------
aspirin and oral cintraceptive
tenderness, edema, dry scaly or lichenifid risk factors:
treat the underline cause : leg elevation
occurs on lower legs because of underlying hyperpigmentation, and varicose veins. Above genetics ,obesity ,female gender, pregnancy , -----------
-------------- stasis eczema insufficientvenous drainage.
it is common among middle'aged women.
medial malleolus. prolonged standing, surgery, trauma, and
compression,
treatment of infection, and dermatitis.
Complication:legulcer malignancies
by fatema okoff
Topical steroid are the mainstay of N o biomarker for the diagnosis of
-itchy erythema involves the face(cheeks)
the infant is irritable , restless at night due to treatment atopic dermatitis
-the extensor surfaces of the extremities
Infantile atopic dermatitis infancy,( 2months___ 2 years)
-The diaper area is usually spared ,
sever pruritus -Low potency steroid on (face and groin) swab of infected skin
-The course chronically relapsing and remitting -Medium potency on (trunk and extremities) radioallergosorbent(R AS T ) testing
-
-Strong steroid on (palmoplantar areas) Serum IgE .
-flexural areas of elbows , knees, wrist ,ankle
and the neck -Oral steroid in sever atopic dermatitis.
Atopic dermatitis (AD) Childhood atopic dermatitis Childhood (2-12 years)
-Persistent rubbing and scratching lead to
Subtopic 1
-Anti-histamin help in controlling the itching
same
lichenification.
( dry skin)
-Ear lobe eczema
-flexural involvement
-eczema of the nipple
-front and sides of neck, eyelids , face , feet -Immune modulator : pimecrolimus and
-eczema around the margins of the mouth
and hands tacrolimus inhibit inflammatory cytokine
Adult atopic dermatitis adult age -hyper pigmented, pruritus is sever
(cheilitis)
transcription in activated T-cell.
same
*black people , follicular eczema
-excoriation and lichenification common. -Cyclosporine in sever case refractory case
characterized by (discrete follicular
-30%develop atopic hand eczema
papules)
•The acute phase:
-Nickel: artificial jewellery, Hair pins.
-pruritus, erythema, edema, and vesicles
watches. button
-confined to the area of direct exposure.
-Paraphenylene diamine: hair dye
a cell-mediated (type IV) •chronic disease:
Allergic contact dermatitis (ACD) - delayed type hypersensitivity reaction caused Some people are susceptible -lichenified erythematous plaques with
-Chromium:cement, painter Avoidance is the mainstay of treat
Patch test is diagnostic
20% -Rubber:gloves ,Condom nent for ACD.
by skin contact with an environmental allergen. hyperkeratosis and fissuring
-Synthetic fiber:,acrylic and polyesters cloths.
-spread beyond the areas of direct exposure.
-formaldehyde: shampoo and cosmetics
·The hands, feet, and face (including the
-Topical medication: neomycin, tar.
eyelids)are some of the common sites forACD
successful treatment of contact dermatitis:
contact dermatitis identification of suhstances causing CD its
-a non-immunologic inflammation of the skin -Most irritant eczemas occur on( the hand) ·Examples of ICD:
avoidance.
caused by contact with a chemical, physical, or -Strong irritant: blisters, erosions, And ulcers -house wife dermatitis.
Irritant contact dermatitis (ICD) biologic agent. all people are suspected and pain rather than itching.. -cleaners. Hair dresser, food handler health
-Acute reaction require (wet soaks) or
Patch test isn't diagnostic
80% (compress) and topical steroid and antibiotic
eczema
-No hypersensitivity involved -mild irritant: produce (cumulative worker
cream or lotion.
-the reaction is immediate dermatitis)erythema and micro-vesiculation. -infant napkin dermatit
-In sever cases :oral steroid &oral antibiotic if
secondary infection.
contact with certain plant chemicals contain
Phytophotodermatitis (psoralin) can cause skin inflammation when less common ___________________________
exposed to sunlight.
• infant :self limited form
-1)low potency topical corticosteroids
(hydrocortisone 1% cream orlotion for a few
lesions: days), followed by2) topical imidazoles (2%
scalp,ears,face,nasolabial,eyebrows,uppereyelid, ketoconazole cream,lotion, or 1% shampo0),
posturicular area,presternal chest, uppertrunk, -Seborrhoeic dermatitis in fold axilla, groin, • Adults: tend to have chronic and recurrent
•The etiology is unclear but there are associations flares occur when sebaceous glands are most neck and intertriginous areas submamary seen in middle and elderly disease
seborrheic dermatitis with Malassezia yeasts, sebum secretion and active (firstfew months of life, and post
-sharply demarcated, yellow to red to brown,
specially obese woman -the patients should be informed that the aim
oftreatment will be to control rather than cure
composition,and certain drugs. puberty) -scalp of infant called cradle cap
greasy or bran-like scaling patches and plaques. -scaling on eye browse seborrheic blepharitis the disease.
-presence ot bright red and -Scalp seborrheic dermatitis treated by
macerated ill defined edge with sever itching (shampoos containing selenium sulfide,
imidazoles ,ketoconazole shampoo)
-oral antifungals should be reserved for severe
and refractory cases
-round lesions distributed commonly on the
extensor surtace of extremities . -topical steroid
--------------- Nummular eczema unknown Occurs in all age group common 55-65 years.
- coin- shape studded with vesicles on an
______
-Immune modulator topically applied.
_____________
erythematous base
-unknown cause.
deep seated sago like vesicles on the palms and
-slightly higher in atopy. common in young adults, rare in children and
----------- pompholyx -related to stress it may follow drug eruption. old age
lateral aspect of fingers and soles bilateral
symmetrical
---------- highy potency topical steroid ---------
aspirin and oral cintraceptive
tenderness, edema, dry scaly or lichenifid risk factors:
treat the underline cause : leg elevation
occurs on lower legs because of underlying hyperpigmentation, and varicose veins. Above genetics ,obesity ,female gender, pregnancy , -----------
-------------- stasis eczema insufficientvenous drainage.
it is common among middle'aged women.
medial malleolus. prolonged standing, surgery, trauma, and
compression,
treatment of infection, and dermatitis.
Complication:legulcer malignancies
by fatema okoff
