Davis Advantage for Pathophysiology 3rd Edition () – Test Bank All Chapters 1-46 with Questions, Answers, and Rationales

DAVIS qwADVANTAGE qwFOR qwPATHOPHYSIOLOGY3rd EDITION qwCAPRIOTTI qwTEST
qwBANK




Chapter 1, The Cell in Health and Illnes
TEST BANK FOR DAVIS ADVANTAGE FOR PATHOPHYSIOLOGY 3rd EDITION BY
CAPRIOTTI


Multiple qwChoice
Identify qwthe qwchoice qwthat qwbest qwcompletes qwthe qwstatement qwor qwanswers qwthe qwquestion.

q w 1. q w Which qwstatement qwregarding qwthe qwsodium–potassium qwpump qwis qwcorrect?
1. The qwcell’s qwplasma qwmembrane qwis qwmore qwsoluble qwto qwsodium qwions qwthan qwpotassium qwions.
2. The qwconcentration qwof qwsodium qwions qwshould qwbe qwhigher qwinside qwthe qwcell qwcompartment.
3. The qwconcentration qwof qwpotassium qwions qwshould qwbe qwhigher qwoutside
the qwcell qwcompartment.
qw

4. The qwactive qwtransport qwinvolves qwpumping qwout qwthree qwsodium qwions qwand
pumping qwin qwtwo qwpotassium qwions.
qw


q w 2. qwIn qwthe qwabsence qwof qwoxygen, qwwhich qwcellular qwfunction qwcreates qwthe qwsame qwamount qwof
qwenergy qwas qwis qwcreated qwin qwthe qwpresence qwof qwoxygen?
1. Dissipation qwof qwpyruvic qwacid
2. Initiation qwof qwthe qwcitric qwacid qwcycle
3. Activation qwof qwacetyl-coenzyme qwA
4. Creation qwof qwacidosis qwvia qwlactic qwacid
q w3. q w How qwmany qwadenosine qwtriphosphates qw(ATPs) qware qwproduced qwin qwaerobic qwenergy qwmetabolism?
1. q w q w 2
2. q w q w 3
3. q w q w 34 4. q w q w 53

4. Which qwcell qworganelles qwdiffer qwin qwtheir qwnumber qwaccording qwto qwthe qwcell’s
qw energy qwneeds?
1. Ribosomes
2. Mitochondria
3. Ribonucleic qwacids
4. Deoxyribonucleic qwacids

, DAVIS qwADVANTAGE qwFOR qwPATHOPHYSIOLOGY3rd EDITION qwCAPRIOTTI qwTEST
qwBANK
5. Which qwoption qwbest qwsupports qwthe qwreason qwmore qwenergy qwis qwproduced qwwhen qwa qwperson qwis
exercising?
qw

1. Exercise qwcauses qwan qwincrease qwin qwthe qwsynthesis qwof qwprotein.
2. There qwis qwan qwincrease qwin qwthe qwproduction qwof qwpyruvic qwacid qwin qwthe qwcells.
3. The qwconversion qwof qwpyruvic qwacid qwto qwlactic qwacid qwis qwincreased qwby qwexercise.
4. Muscle qwcells qwhave qwmore qwmitochondria qwto qwmeet qwenergy qwdemands.
6. When qwdoes qwribosomal qwprotein qwsynthesis qwcease?
1. During qwendoplasmic qwreticulum qwstress
2. During qwthe qwsynthesis qwof qwadenosine qwtriphosphate qw(ATP)
3. During qwa qwsevere qwhypoxic qwstate
4. During qwthe qwprocessing qwof qw prohormone

7. Which qwcellular qworganelles qware qwresponsible qwfor qwpropelling qwmucus qwand qwinhaled qwdebris qwout
of qwthe qwlungs?
qw

1. Cilia
2. Microfilaments
3. Secretory qwvesicles

, DAVIS qwADVANTAGE qwFOR qwPATHOPHYSIOLOGY3rd EDITION qwCAPRIOTTI qwTEST
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4. q w Endoplasmic qwreticula

qw 8. q w Which qware qwthe qwkey qwproteins qwin qwthe qwcontractile qwunits qwof qwthe qwmuscle qwcells?
1. Actin qwand qwmyosin
2. Prohormone qwand qwtubulin
3. Tubulin qwand qwactin
4. Myosin qwand qwprohormone

qw 9. q wWhich qwdeficiency qwcauses qwTay–Sachs qwdisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal qwenzymes
10. qwWhich qwis qwa qwcharacteristic qwof qwadrenoleukodystrophy?
1. Accumulation qwof qwganglioside
2. Cessation qwof qwribosomal qwprotein qwsynthesis
3. Acceleration qwof qwcellular qwproteasome qwactivity
4. Accumulation qwof qwlong-chain qwfatty qwacids qwin qwthe qwnervous qwsystem

11. qwWhich qwstatement qwregarding qwendoplasmic qwreticulum qw(ER) qwstress qwis qwcorrect?
1. During qwER qwstress, qwproteins qware qwrapidly qwdegraded.
2. During qwER qwstress, qwlipids qwcannot qwtravel qwto qwtheir qwproper qwintracellular qwlocations.
3. During qwER qwstress, qwlong-chain qwfatty qwacids qwaccumulate qwin qwthe qwnervous qwsystem.
4. DuringqwER qwstress, qwnondeNgU
raR
deSdIsN
ubGsT
taB
nc.eC
s qwO
acMcumulate q w in qwtheqwcells.
12. qwA qwclient qwis qwdiagnosed qwwith qwtype qw1 qwdiabetes qwmellitus. qwAt qwa qwcellular qwlevel, qwwhich
qwfunction qwis qwlikely qwto qwbe qwinvolved?
1. Inabilityqwof qwribosomes qwto qwproduce qwa qwspecific qwtype qwof qwprotein
2. Incorrect qwprocessing qwof qwa qwprotein qwbyqwthe qwGolgi qwapparatus
3. Stagnation qwof qwa qwpreviously qwdynamic qwaction qwin qwmicrotubules
4. Obstruction qwof qwthe qwsmooth qwendoplasmic qwreticulum
13. qwA qwnewborn qwpatient qwexhibits qwcharacteristics qwof qwsevere qwphysical qwdeformities. qwWhich qwcellular
component qwis qwexamined qwto qwdetermine qwthe qwcause qwand qwprobability qwof qwthe qwdisease
qw

qwbeing qwgenetically qwtransferred?
1. Transfer qwRNA
2. Ribosomal qwRNA
3. Double qwhelix qwof qwDNA
4. Mitochondrial qwDNA

14. qwA qwhiker qwexperiences qwmuscle qwpain qwand qwacidosis qwwhile qwascending qwa qwmountain qwduring
qwa qwlong, qwsteep qwclimb. qwWhich qwis qwthe qwreason qwfor qwthese qwmanifestations?
1. Cellular qwhypoxia
2. Autolysis
3. Heterolysis
4. Cellular qwedema
15. qwWhich qwfactor qwprovides qwDNA qwthe qwunique qwmolecular qwability qwto qwreplicate?
1. The qwpairing qwof qwnitrogenous qwbases

, DAVIS q w ADVANTAGE q w FOR 3rd EDITION qw CAPRIOTTI
q w PATHOPHYSIOLOGY q w TEST




2. The q w presence q w of q w pyrimidine q w bases
3. The q w presence q w of q w nucleotides

4. The q w nitrogenous q w base q w and q w phosphate q w bond
How many q w nitrogenous q w bases q w compose q w a q w single q w codon?
16.
qw

1. 2
2. 3
3. 4
4. 5
Which q w components q w form q w the q w structure q w of q w DNA?
17.
qw
1.Nucleotides
2.Amino q w acids
3.Fatty q w acids
4.Phosphates
Which q w factor q w is q w essential q w in q w order q w for q w protein q w synthesis
18.
qw
q w to q w occur?

1. Free-standing q w ribosomes q w within q w the q w cell
2. Protein q w blueprint q w from q w the q w cell q w of q w the q w DNA
3. Specific q w information q w from q w the q w nucleus q w of q w the q w cell
4. Transfer q w RNA q w to q w move q w the q w protein q w out q w of q w the q w cell
Tetracycline q w antibiotic q w is q w prescribed q w for q w an q w adult q w client q w with W
19.
qw
q w chlamydia q w infection. qwhich q w is q w the q w mechanism q w of q w action q w of

q w the q w drug?
1. It q w prevents q w the q w replicatioNnUoR
f q w bSaIctNerGiaT. q w B.COM
2. It q w alters q w the q w configuration q w of q w bacterial q w cytoplasm.
3. It q w interferes q w with q w the q w function q w of q w bacterial q w ribosomes.
4. It q w inhibits q w the q w functions q w of q w bacterial q w mitochondria.

qw20. q w qw Where q w does q w the q w conversion q w of q w a q w prohormone q w into q w a q w hormone q w take
q w place?
1. In q w the q w ribosomes
2. In q w the q w Golgi q w apparatus

3. In q w the q w secretory q w granules

4. In q w the q w endoplasmic q w reticulum


qw21. q w Which q w is q w the q w cell’s q w “master q w mind”?
1. Nucleus
2. Ribosome
3. Golgi q w apparatus
4. Endoplasmic q w reticulum

Multiple q w Response
Identify q w one q w or q w more q w choices q w that q w best q w complete q w the q w statement q w or q w answer q w the
q w question.



qw22. q w Which q w statements q w regarding q w the q w microtubules q w are q w true? q w Select q w all q w that
q w apply.
1. Microtubules q w are q w solid.
2. Microtubules q w are q w flexible.
3. Microtubules q w are q w composed q w of q w tubulin.