TEST BANK
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF
MEDICINE 7TH EDITION
DAVID S. STRAYER, EMANUEL RUBIN
,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
Table of Contents:
Chapter 1: Cell Adaptation, Injury and Death
Chapter 2: Inflammation
Chapter 3: Repair, Regeneration and Fibrosis
Chapter 4: Immunopathology
Chapter 5: Neoplasia
Chapter 6: Developmental and Genetic Diseases
Chapter 7: Hemodynamic Disorders
Chapter 8: Environmental and Nutritional Pathology
Chapter 9: Infectious and Parasitic Diseases
Section II: Pathogenesis of Systemic Conditions Expandable section
Chapter 10: Aging
Chapter 11: Systemic Autoimmune Diseases
Chapter 12: Sepsis
Chapter 13: Obesity and Diabetes Mellitus
Chapter 14: The Pathology of Pregnancy
Chapter 15: The Amyloidoses
Section III: Diseases of Individual Organ SystemsExpandable section
Chapter 16: Blood Vessels
Chapter 17: The Heart
Chapter 18: The Respiratory System
Chapter 19: The Gastrointestinal Tract
Chapter 20: The Liver and Biliary System
Chapter 21: The Pancreas
Chapter 22: The Kidney
Chapter 23: The Lower Urinary Tract and Male Reproductive System
Chapter 24: The Female Reproductive System and Peritoneum
Chapter 25: The Breast
Chapter 26: Hematopathology
Chapter 27: The Endocrine System
Chapter 28: The Skin
Chapter 29: The Head and Neck
Chapter 30: Bones, Joints and Soft Tissue
Chapter 31: Skeletal Muscle and Peripheral Nervous System
Chapter 32: The Central Nervous System
Chapter 33: The Eye
Chapter 34: Forensic Pathology
,Rubin's mmPathology: mmClinicopathologic
mmFoundations mmof mmMedicineChapter mm1: mmCell
mmAdaptation, mmInjury mmand mmDeath
Ischemia mmand mmother mmtoxic mminjuries mmincrease mmthe mmaccumulation mmof mmintracellular
mmcalcium mmas mma mmresult
1. of:
A) release mmof mmstored mmcalcium mmfrom mmthe mmmitochondria.
B) improved mmintracellular mmvolume mmregulation.
C) decreased mminflux mmacross mmthe mmcell mmmembrane.
D) attraction mmof mmcalcium mmto mmfatty mminfiltrates.
The mmpatient mmis mmfound mmto mmhave mmliver mmdisease, mmresulting mmin mmthe mmremoval mmof
mma mmlobe mmof mmhis mmliver.
2. Adaptation mmto mmthe mmreduced mmsize mmof mmthe mmliver mmleads mmto mm_ of mmthe mmremaining
mmliver mmcells.
A) metaplasia
B) organ mmatrophy
C) compensatory mmhyperplasia
D) physiologic mmhypertrophy
A mmperson mmeating mmpeanuts mmstarts mmchoking mmand mmcollapses. mmHis mmairway
mmobstruction mmis mmpartially mmcleared, mmbut mmhe mmremains mmhypoxic mmuntil mmhe
mmreaches mmthe mmhospital. mmThe mmprolonged mmcell mmhypoxia
3. caused mma mmcerebral mminfarction mmand mmresulting _ mmin mmthe mmbrain.
A) caspase mmactivation
B) coagulation mmnecrosis
C) rapid mmphagocytosis
D) protein mmp53 mmdeficiency
Bacteria mmand mmviruses mmcause mmcell mmdamage mmby, mmwhich mmis mmunique mmfrom mmthe
mmintracellular
4. damage mmcaused mmby mmother mminjurious mmagents.
A) disrupting mmthe mmsodium/potassium mmATPase mmpump
B) interrupting mmoxidative mmmetabolism mmprocesses
C) replicating mmand mmproducing mmcontinued mminjury
D) decreasing mmprotein mmsynthesis mmand mmfunction
The mmpatient mmhas mma mmprolonged mminterruption mmin mmarterial mmblood mmflow mmto mmhis mmleft
mmkidney, mmcausing
5. hypoxic mmcell mminjury mmand mmthe mmrelease mmof mmfree mmradicals. mmFree mmradicals mmdamage
mmcells mmby:
A) destroying mmphospholipids mmin mmthe mmcell mmmembrane.
, B) altering mmthe mmimmune mmresponse mmof mmthe mmcell.
C) disrupting mmcalcium mmstorage mmin mmthe mmcell.
D) inactivation mmof mmenzymes mmand mmmitochondria.
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF
MEDICINE 7TH EDITION
DAVID S. STRAYER, EMANUEL RUBIN
,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
Table of Contents:
Chapter 1: Cell Adaptation, Injury and Death
Chapter 2: Inflammation
Chapter 3: Repair, Regeneration and Fibrosis
Chapter 4: Immunopathology
Chapter 5: Neoplasia
Chapter 6: Developmental and Genetic Diseases
Chapter 7: Hemodynamic Disorders
Chapter 8: Environmental and Nutritional Pathology
Chapter 9: Infectious and Parasitic Diseases
Section II: Pathogenesis of Systemic Conditions Expandable section
Chapter 10: Aging
Chapter 11: Systemic Autoimmune Diseases
Chapter 12: Sepsis
Chapter 13: Obesity and Diabetes Mellitus
Chapter 14: The Pathology of Pregnancy
Chapter 15: The Amyloidoses
Section III: Diseases of Individual Organ SystemsExpandable section
Chapter 16: Blood Vessels
Chapter 17: The Heart
Chapter 18: The Respiratory System
Chapter 19: The Gastrointestinal Tract
Chapter 20: The Liver and Biliary System
Chapter 21: The Pancreas
Chapter 22: The Kidney
Chapter 23: The Lower Urinary Tract and Male Reproductive System
Chapter 24: The Female Reproductive System and Peritoneum
Chapter 25: The Breast
Chapter 26: Hematopathology
Chapter 27: The Endocrine System
Chapter 28: The Skin
Chapter 29: The Head and Neck
Chapter 30: Bones, Joints and Soft Tissue
Chapter 31: Skeletal Muscle and Peripheral Nervous System
Chapter 32: The Central Nervous System
Chapter 33: The Eye
Chapter 34: Forensic Pathology
,Rubin's mmPathology: mmClinicopathologic
mmFoundations mmof mmMedicineChapter mm1: mmCell
mmAdaptation, mmInjury mmand mmDeath
Ischemia mmand mmother mmtoxic mminjuries mmincrease mmthe mmaccumulation mmof mmintracellular
mmcalcium mmas mma mmresult
1. of:
A) release mmof mmstored mmcalcium mmfrom mmthe mmmitochondria.
B) improved mmintracellular mmvolume mmregulation.
C) decreased mminflux mmacross mmthe mmcell mmmembrane.
D) attraction mmof mmcalcium mmto mmfatty mminfiltrates.
The mmpatient mmis mmfound mmto mmhave mmliver mmdisease, mmresulting mmin mmthe mmremoval mmof
mma mmlobe mmof mmhis mmliver.
2. Adaptation mmto mmthe mmreduced mmsize mmof mmthe mmliver mmleads mmto mm_ of mmthe mmremaining
mmliver mmcells.
A) metaplasia
B) organ mmatrophy
C) compensatory mmhyperplasia
D) physiologic mmhypertrophy
A mmperson mmeating mmpeanuts mmstarts mmchoking mmand mmcollapses. mmHis mmairway
mmobstruction mmis mmpartially mmcleared, mmbut mmhe mmremains mmhypoxic mmuntil mmhe
mmreaches mmthe mmhospital. mmThe mmprolonged mmcell mmhypoxia
3. caused mma mmcerebral mminfarction mmand mmresulting _ mmin mmthe mmbrain.
A) caspase mmactivation
B) coagulation mmnecrosis
C) rapid mmphagocytosis
D) protein mmp53 mmdeficiency
Bacteria mmand mmviruses mmcause mmcell mmdamage mmby, mmwhich mmis mmunique mmfrom mmthe
mmintracellular
4. damage mmcaused mmby mmother mminjurious mmagents.
A) disrupting mmthe mmsodium/potassium mmATPase mmpump
B) interrupting mmoxidative mmmetabolism mmprocesses
C) replicating mmand mmproducing mmcontinued mminjury
D) decreasing mmprotein mmsynthesis mmand mmfunction
The mmpatient mmhas mma mmprolonged mminterruption mmin mmarterial mmblood mmflow mmto mmhis mmleft
mmkidney, mmcausing
5. hypoxic mmcell mminjury mmand mmthe mmrelease mmof mmfree mmradicals. mmFree mmradicals mmdamage
mmcells mmby:
A) destroying mmphospholipids mmin mmthe mmcell mmmembrane.
, B) altering mmthe mmimmune mmresponse mmof mmthe mmcell.
C) disrupting mmcalcium mmstorage mmin mmthe mmcell.
D) inactivation mmof mmenzymes mmand mmmitochondria.
