CANC 380 - MODULE 1 REVIEW QUESTIONS AND CORRECT
DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY
GRADED A+||BRAND NEW
How are tumours formed? Ans✓✓✓When DNA in chromosomes
undergo a change, causing cell proliferation, angiogenesis and
metastasis
Cell proliferation Ans✓✓✓Uncontrollable growth and reproduction of a
tumour
Angiogenesis Ans✓✓✓Formation of new blood vessels allowing for the
tumour to live and grow.
How does a cell become cancerous Ans✓✓✓Genetic factors, exposure
to viruses (such as HIV, hepatitis B, C and D, HPV and Epstein-Barr),
exposure to toxic chemicals, radiation, sun and unhealthy behaviours
such as alcohol, tobacco and a diet rich in fat.
Methods to eliminate cancer Ans✓✓✓Surgery, Radiation,
Chemotherapy.
Chemotherapy Ans✓✓✓Administering systemic medicinal substances
to destroy the cancerous cells or prevent them from spreading.
Radiation Ans✓✓✓Exposing the tumour to radioactive rays =,
preventing the diseased cells from multiplying.
, Carcinoma Ans✓✓✓Greek word for crab. Used by Greek physician
Hippocrates to refer to tumours - describes cancer because of the finger-
like spreading projections from a tumour.
Oncos Ans✓✓✓Greek word for swelling. Still used to describe
oncologists which are cancer specialist doctors.
Development of Chemotherapy Ans✓✓✓chemical weapons contributed
to the development.
How does cancer arise in terms of genetics and epigenetics?
Ans✓✓✓Originates from mutations in oncogenes and tumour
suppressors.
Oncogenes Ans✓✓✓Signal the cell to grow - normally dormant. One
mutation in this gene is enough to activate the gene, telling the cell to
keep growing and dividing.
Tumour suppressor genes Ans✓✓✓Stop a cell from growing, unless
conditions are favourable. One gene become deactivated and a section of
DNA containing the other gene is lost, allowing for tumour cell growth.
Genome sequencing Ans✓✓✓determine which genes have been
mutated, predict which drugs would be most effective - resulting in
personalized medicine.
DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY
GRADED A+||BRAND NEW
How are tumours formed? Ans✓✓✓When DNA in chromosomes
undergo a change, causing cell proliferation, angiogenesis and
metastasis
Cell proliferation Ans✓✓✓Uncontrollable growth and reproduction of a
tumour
Angiogenesis Ans✓✓✓Formation of new blood vessels allowing for the
tumour to live and grow.
How does a cell become cancerous Ans✓✓✓Genetic factors, exposure
to viruses (such as HIV, hepatitis B, C and D, HPV and Epstein-Barr),
exposure to toxic chemicals, radiation, sun and unhealthy behaviours
such as alcohol, tobacco and a diet rich in fat.
Methods to eliminate cancer Ans✓✓✓Surgery, Radiation,
Chemotherapy.
Chemotherapy Ans✓✓✓Administering systemic medicinal substances
to destroy the cancerous cells or prevent them from spreading.
Radiation Ans✓✓✓Exposing the tumour to radioactive rays =,
preventing the diseased cells from multiplying.
, Carcinoma Ans✓✓✓Greek word for crab. Used by Greek physician
Hippocrates to refer to tumours - describes cancer because of the finger-
like spreading projections from a tumour.
Oncos Ans✓✓✓Greek word for swelling. Still used to describe
oncologists which are cancer specialist doctors.
Development of Chemotherapy Ans✓✓✓chemical weapons contributed
to the development.
How does cancer arise in terms of genetics and epigenetics?
Ans✓✓✓Originates from mutations in oncogenes and tumour
suppressors.
Oncogenes Ans✓✓✓Signal the cell to grow - normally dormant. One
mutation in this gene is enough to activate the gene, telling the cell to
keep growing and dividing.
Tumour suppressor genes Ans✓✓✓Stop a cell from growing, unless
conditions are favourable. One gene become deactivated and a section of
DNA containing the other gene is lost, allowing for tumour cell growth.
Genome sequencing Ans✓✓✓determine which genes have been
mutated, predict which drugs would be most effective - resulting in
personalized medicine.
