MCB 100 - QUIZ #9 EXAM 2 WITH QUESTIONS AND COR
Place cthe cfollowing cstatements cin cthe cchronological corder cthat cbest cdescribes cthe csequence co
cappearance cof cacne.
- cP. cacnes cmetabolizes cglycerol cin csebum, cforming cfree cfatty cacids.
- cOverproduction cof csebum cleads cto can caccumulation cin cthe cfollicle.
- cFree cfatty cacids cstimulate cthe crelease cof cproinflammatory ccytokines cwithin cthe cfollicle. c
- cThe cblood cvessels csurrounding cthe cfollicle cdilate cand cbecome cmore cpermeable.
- cImmune ccells csuch cas cneutrophils cand cphagocytes cmove cinto cthe cfollicle cto ctry cto celiminate
- cThe cbuildup cof cdead ccells, cbacteria, cand cbody cfluids ccauses ca craised cpustule cto cappear con
cCORRECT cANS✔✔1. cOverproduction cof csebum cleads cto can caccumulation cin cthe cfollicle.
2. cP. cacnes cmetabolizes cglycerol cin csebum, cforming cfree cfatty cacids.
3. cFree cfatty cacids cstimulate cthe crelease cof cproinflammatory ccytokines cwithin cthe cfollicle.
4. cThe cblood cvessels csurrounding cthe cfollicle cdilate cand cbecome cmore cpermeable.
5. cImmune ccells csuch cas cneutrophils cand cphagocytes cmove cinto cthe cfollicle cto ctry cto celiminate
6. cThe cbuildup cof cdead ccells, cbacteria, cand cbody cfluids ccauses ca craised cpustule cto cappear co
Which ccomponent cof ca cGram-negative ccell cwall ccan ccause cfever, cvasodilation, cinflammation, cs
A. cLipid cA
B. cPeptidoglycan
C. cThe cperiplasm
D. cThe cinner cmembrane c- cCORRECT cANS✔✔A
n cthe ccase cof cgastrointestinal cdisease, chow ccan can cintoxication cbe cdistinguished cfrom can cinfe
A. cAn cintoxication cis cusually capparent cshortly cafter cingestion, cwhile can cinfection ctakes clonger.
B. cIntoxications cmay cbe ccaused cby cendotoxin.
C. cInfections care ccaused cby cheat-stable cagents.
D. cAn cintoxication cusually ccauses ca cfever, cwhile can cinfection cdoes cnot. c- cCORRECT cANS✔✔
Most cmicrobes cthat creside cin cthe cintestines care cfacultative cor cobligate canaerobes. cDescribe cth
canaerobes cin cthe cpresence cof cand calso cin cthe cabsence cof cmolecular coxygen.
A. cFacultative canaerobes crequire ca csmall camount cof cmolecular coxygen.
B. cFacultative canaerobes cgrow cequally cwell cin cthe cpresence cof cand cin cthe cabsence cof cmolec
C. cFacultative canaerobes ccan cgrow cwithout cmolecular coxygen, cbut cgrow cfaster cin cits cpresence
D. cFacultative canaerobes crequire cthe cabsence cof cmolecular coxygen. c- cCORRECT cANS✔✔C
An cappropriately cstained csmear cwas cprepared cof ca csputum cspecimen cobtained cfrom cthe csick
cUsing coil-immersion cmagnification con cyour cmicroscope, cwhat cwould cyou cobserve cthat cwould c
, A. cA csputum cspecimen cfrom cLance cwas ccultured cfor cpossible cgrowth cof cMycobacterium ctuber
B. cLance's cblood cwas cdrawn cand cexamined cfor cthe cpresence cof cMycobacterium ctuberculosis.
C. cAn cX-ray cfilm cof cLance's clungs cwas ctaken.
D. cA csmall camount cof ccell cwall cmaterial cfrom cMycobacterium ctuberculosis cwas cinjected cinto cL
cCORRECT cANS✔✔D
How cis ctuberculosis ctransmitted?
A. cSharing c"dirty" cneedles cbetween cIV cdrug cusers
B. cFecal/oral croute
C. cAerosol cdroplets creleased cby ca ccough cor csneeze
D. cDrinking cpasteurized cmilk c- cCORRECT cANS✔✔C
The cname cPropionibacterium cis cderived cfrom cthe cfact cthat cthe corganism cproduces cpropionic ca
cmetabolism. cWhich cof cthe cfollowing cstatements cabout cfermentative cmetabolism cis ctrue?
A. cFermentation cproduces csignificantly cless cATP cthan caerobic crespiration; chowever cit cgenerate
cin cglycolysis.
B. cAll cfermentative corganisms chave cthe cability cto cperform cethanol cfermentation.
C. cFermentation cdoes cnot cyield cany cATP cfor cthe ccell.
D. cFermentation cis cunfavorable cbecause cthe cproduction cof cacidic cend-products cprovides can cu
corganism's cgrowth. c- cCORRECT cANS✔✔A
Research chas cindicated cthat cduring cthe cinflammatory cresponse cto cP. cacnes, cboth cthe cclassica
ccomplement cpathways care cactivated. cWhich cof cthe cfollowing care coutcomes cof cactivating ccom
A. cInflammation
B. cGeneration cof cantibodies cagainst cP. cacnes
C. cActivation cof cT ccytotoxic ccells
D. cOpsonization
E. cCytolysis c- cCORRECT cANS✔✔A, cD, c& cE
Part cof cthe creason cfor cScott's cacne cis cthe coverproduction cof csebum. cScott's cinitial cinstinct cis c
cof csebum cwould cbe cthe cquickest croute cto celiminating chis cacne; chowever, csebum cactually cpla
cmaintaining cthe cskin cas ca cfirst cline cof cdefense cagainst cpathogens. cWhich cof cthe cfollowing cbe
csebum cas ca ccomponent cof cthe cfirst cline cof cdefense?
A. cSebum ccontains cenzymes cthat cinhibit cthe cgrowth cof cpathogens.
B. cSebum ccreates can canaerobic cenvironment, cthereby cpreventing cthe cgrowth cof cpathogenic co
C. cSebum chelps cto ckeep cthe cpH cof cthe cskin clow, cwhich cinhibits cthe cgrowth cof cmicroorganism
D. cSebum cforms ca cprotective clayer cover cthe cskin. c- cCORRECT cANS✔✔C c& cD
Dr. cSmith cassures cScott cthat calthough cinflammation cmay csound cscary, cthere care cseveral cposs
cScott's cmoderate cacne. cWhich cof cthe cfollowing cwould cbe cthe cmost cappropriate cfor cScott's cca
A. cEliminating cthe cintake cof cfatty cfoods
B. cTopical capplication cof csalicylic cacid
C. cA cprescription cfor cAccutane
D. cTopical ctreatment cwith ca cproduct ccontaining cbenzoyl cperoxide
E. cAn coral cantibiotic csuch cas cerythromycin
F. cClear cLight, ca cnonchemical, clight-based csystem c- cCORRECT cANS✔✔D, cE, c& cF
Acne cis cthe cresult cof cthe cinflammatory cresponse. cIn cthis ccase, cthe cinflammation cis cresulting cin
c(physically cand cemotionally) cfor cScott. cHowever, cinflammation cis ca ccritical ccomponent cof ca cno
Place cthe cfollowing cstatements cin cthe cchronological corder cthat cbest cdescribes cthe csequence co
cappearance cof cacne.
- cP. cacnes cmetabolizes cglycerol cin csebum, cforming cfree cfatty cacids.
- cOverproduction cof csebum cleads cto can caccumulation cin cthe cfollicle.
- cFree cfatty cacids cstimulate cthe crelease cof cproinflammatory ccytokines cwithin cthe cfollicle. c
- cThe cblood cvessels csurrounding cthe cfollicle cdilate cand cbecome cmore cpermeable.
- cImmune ccells csuch cas cneutrophils cand cphagocytes cmove cinto cthe cfollicle cto ctry cto celiminate
- cThe cbuildup cof cdead ccells, cbacteria, cand cbody cfluids ccauses ca craised cpustule cto cappear con
cCORRECT cANS✔✔1. cOverproduction cof csebum cleads cto can caccumulation cin cthe cfollicle.
2. cP. cacnes cmetabolizes cglycerol cin csebum, cforming cfree cfatty cacids.
3. cFree cfatty cacids cstimulate cthe crelease cof cproinflammatory ccytokines cwithin cthe cfollicle.
4. cThe cblood cvessels csurrounding cthe cfollicle cdilate cand cbecome cmore cpermeable.
5. cImmune ccells csuch cas cneutrophils cand cphagocytes cmove cinto cthe cfollicle cto ctry cto celiminate
6. cThe cbuildup cof cdead ccells, cbacteria, cand cbody cfluids ccauses ca craised cpustule cto cappear co
Which ccomponent cof ca cGram-negative ccell cwall ccan ccause cfever, cvasodilation, cinflammation, cs
A. cLipid cA
B. cPeptidoglycan
C. cThe cperiplasm
D. cThe cinner cmembrane c- cCORRECT cANS✔✔A
n cthe ccase cof cgastrointestinal cdisease, chow ccan can cintoxication cbe cdistinguished cfrom can cinfe
A. cAn cintoxication cis cusually capparent cshortly cafter cingestion, cwhile can cinfection ctakes clonger.
B. cIntoxications cmay cbe ccaused cby cendotoxin.
C. cInfections care ccaused cby cheat-stable cagents.
D. cAn cintoxication cusually ccauses ca cfever, cwhile can cinfection cdoes cnot. c- cCORRECT cANS✔✔
Most cmicrobes cthat creside cin cthe cintestines care cfacultative cor cobligate canaerobes. cDescribe cth
canaerobes cin cthe cpresence cof cand calso cin cthe cabsence cof cmolecular coxygen.
A. cFacultative canaerobes crequire ca csmall camount cof cmolecular coxygen.
B. cFacultative canaerobes cgrow cequally cwell cin cthe cpresence cof cand cin cthe cabsence cof cmolec
C. cFacultative canaerobes ccan cgrow cwithout cmolecular coxygen, cbut cgrow cfaster cin cits cpresence
D. cFacultative canaerobes crequire cthe cabsence cof cmolecular coxygen. c- cCORRECT cANS✔✔C
An cappropriately cstained csmear cwas cprepared cof ca csputum cspecimen cobtained cfrom cthe csick
cUsing coil-immersion cmagnification con cyour cmicroscope, cwhat cwould cyou cobserve cthat cwould c
, A. cA csputum cspecimen cfrom cLance cwas ccultured cfor cpossible cgrowth cof cMycobacterium ctuber
B. cLance's cblood cwas cdrawn cand cexamined cfor cthe cpresence cof cMycobacterium ctuberculosis.
C. cAn cX-ray cfilm cof cLance's clungs cwas ctaken.
D. cA csmall camount cof ccell cwall cmaterial cfrom cMycobacterium ctuberculosis cwas cinjected cinto cL
cCORRECT cANS✔✔D
How cis ctuberculosis ctransmitted?
A. cSharing c"dirty" cneedles cbetween cIV cdrug cusers
B. cFecal/oral croute
C. cAerosol cdroplets creleased cby ca ccough cor csneeze
D. cDrinking cpasteurized cmilk c- cCORRECT cANS✔✔C
The cname cPropionibacterium cis cderived cfrom cthe cfact cthat cthe corganism cproduces cpropionic ca
cmetabolism. cWhich cof cthe cfollowing cstatements cabout cfermentative cmetabolism cis ctrue?
A. cFermentation cproduces csignificantly cless cATP cthan caerobic crespiration; chowever cit cgenerate
cin cglycolysis.
B. cAll cfermentative corganisms chave cthe cability cto cperform cethanol cfermentation.
C. cFermentation cdoes cnot cyield cany cATP cfor cthe ccell.
D. cFermentation cis cunfavorable cbecause cthe cproduction cof cacidic cend-products cprovides can cu
corganism's cgrowth. c- cCORRECT cANS✔✔A
Research chas cindicated cthat cduring cthe cinflammatory cresponse cto cP. cacnes, cboth cthe cclassica
ccomplement cpathways care cactivated. cWhich cof cthe cfollowing care coutcomes cof cactivating ccom
A. cInflammation
B. cGeneration cof cantibodies cagainst cP. cacnes
C. cActivation cof cT ccytotoxic ccells
D. cOpsonization
E. cCytolysis c- cCORRECT cANS✔✔A, cD, c& cE
Part cof cthe creason cfor cScott's cacne cis cthe coverproduction cof csebum. cScott's cinitial cinstinct cis c
cof csebum cwould cbe cthe cquickest croute cto celiminating chis cacne; chowever, csebum cactually cpla
cmaintaining cthe cskin cas ca cfirst cline cof cdefense cagainst cpathogens. cWhich cof cthe cfollowing cbe
csebum cas ca ccomponent cof cthe cfirst cline cof cdefense?
A. cSebum ccontains cenzymes cthat cinhibit cthe cgrowth cof cpathogens.
B. cSebum ccreates can canaerobic cenvironment, cthereby cpreventing cthe cgrowth cof cpathogenic co
C. cSebum chelps cto ckeep cthe cpH cof cthe cskin clow, cwhich cinhibits cthe cgrowth cof cmicroorganism
D. cSebum cforms ca cprotective clayer cover cthe cskin. c- cCORRECT cANS✔✔C c& cD
Dr. cSmith cassures cScott cthat calthough cinflammation cmay csound cscary, cthere care cseveral cposs
cScott's cmoderate cacne. cWhich cof cthe cfollowing cwould cbe cthe cmost cappropriate cfor cScott's cca
A. cEliminating cthe cintake cof cfatty cfoods
B. cTopical capplication cof csalicylic cacid
C. cA cprescription cfor cAccutane
D. cTopical ctreatment cwith ca cproduct ccontaining cbenzoyl cperoxide
E. cAn coral cantibiotic csuch cas cerythromycin
F. cClear cLight, ca cnonchemical, clight-based csystem c- cCORRECT cANS✔✔D, cE, c& cF
Acne cis cthe cresult cof cthe cinflammatory cresponse. cIn cthis ccase, cthe cinflammation cis cresulting cin
c(physically cand cemotionally) cfor cScott. cHowever, cinflammation cis ca ccritical ccomponent cof ca cno
