WCU PATHOPHYSIOLOGY EXAM QUESTIONS WITH
CORRECT ANSWERS
TERITARY |PREVENTION |- |CORRECT |ANSWER✔✔-Ex: |Counseling, |Rehabilitation, |Management |
programs, |Support |groups. |
Goal |is |prevent |furthur |damage |and |maximize |quality |of |life.
SECONDARY |PREVENTION |- |CORRECT |ANSWER✔✔-Ex: |Interventions, |Recommending, |Assisting
|with |finding |work, |Assisting |with |medication. |
Goal |is |to |slow |or |stop |progress.
PRIMARY |PREVENTION |- |CORRECT |ANSWER✔✔-Ex: |Education, |Legistlation, |Regular |exams, |
Vaccinations
Goal |is |to |protect |healthy |people |from |developing |disease.
LATENT |VS. |PRODROMAL |- |CORRECT |ANSWER✔✔-1) |LATENT: |
Exposure |--- |First |S/S
2) |PRODROMAL:
First |S/S |--- |Disease
Endemic |- |CORRECT |ANSWER✔✔-(adj.) |native |or |confined |to |a |particular |region |or |people; |
characteristic |of |or |prevalent |in |a |field
Subclinical |- |CORRECT |ANSWER✔✔-Stage |of |a |disease |or |disorder |not |severe |enough |to |
produce |symptoms |that |can |be |detected |or |diagnosed.
,Functions |NORMALLY.
What |is |an |example |of |etiology? |- |CORRECT |ANSWER✔✔-ETIOLOGY: |Study |of |causes |or |reason
|of |disease. |
1) |Latrogenic: |UNKNOWN
2) |Idiopathic: |UNINTENDED |or |UNWANTED
Allostasis |- |CORRECT |ANSWER✔✔-The |body's |ability |to |maintain |a |STABLE |physiological |
environment |by |adjusting |and |CHANGING |to |meet |internal |and |external |demands.
What |hormones |secrete |during |stress |response? |- |CORRECT |ANSWER✔✔-1) |Coritsol: |
breakdowns |fat |and |protein. |
2)ACTH: |
3)Epinephrine: |
Released |by |adrenal |medullas.
Selyes |three |phases |of |stress |- |CORRECT |ANSWER✔✔-1)Alarm
2)Resistance
3)Exhaustion
The |sympathetic |nervous |system |is |mediated |by |- |CORRECT |ANSWER✔✔-NOREPINEPHRINE
What |are |the |types |of |intracellular |accumulations? |- |CORRECT |ANSWER✔✔-1) |Normal |
Intracellular |accumulation: |Water, |lipids, |proteins, |and |carbohydrates, |that |accumulates |in |
EXCESS. |
,2) |Abnormal |Intracellular |accumulation: |
-Exogenous: |minerals |or |infectious |agent |product.
-Endogenous: |
-abnormal |synthesis |or |metabolism
Types |of |Cellular |Adaptation |- |CORRECT |ANSWER✔✔-1) |Hyperplasia: |INCREASE |in |the |
NUMBER |of |cells. |
-EX: |A |benign |prostatic |hypertrophy |in |middle |aged |men. |
2) |Hypertrophy: |INCREASE |in |the |SIZE |of |cells
3) |Atrophy: |REDUCTION |in |the |SIZE |of |cells. |
-EX: |Skeletal |muscle |atrophy |occurs |when |an |extremity |has |been |immobilized. |
4) |Metaplasia: |CHANGES |to |the |cells |to |adapt |to |new |environments.
5) |Anaplasia: |LOSS |of |cellular |differentiation. |
-Indication |of |malignant |tumors. |
6) |Dysplasia:Cell |MATURATION |and |differentiation |are |delayed.
-Loss |of |uniformity |of |cells. |
-Precursor |to |cancer
EX: |Pap |smear |shows |dysplasia |cells |of |the |cervix
7) |Neoplasia: |NEW |or |expended |GROWTH |in |an |area |of |the |body. |
EX: |Benign |neoplasm |is |uterine |fibroid |tumors. |
, 8) |Apoptosis: |The |genetic |instructions |provided |DNA |to |command |CELLULAR |SELF-
DESTRUCTION
Types |of |Necrosis |- |CORRECT |ANSWER✔✔-1) |Coagulation: |Low |oxygen |levels |(blood |clot)
-Lysomal |changes
-Cyltosol |congeals/coagulates
-Stroma
2) |Liquifaction: |Bacterial |infection. |
-Neutrophils |creats |pus. |
-Cause |abcesses. |
3) |Caseous: |A |slow, |progressive |process |that |produces |thick, |cheesy |pus. |
-Tuberculosis
4) |Fat: |Inflammatory |reaction |with |large |number |of |WBC. |
-Inflammatory |disease |of |pancreas.
5) |Gangrenous: |Combination |of |coagulation |and |liquifcation. |
-Dry, |Wet, |Gas
What |are |side |effects |of |chemotherapy? |- |CORRECT |ANSWER✔✔-1) |Anemia
2)Nausea
3)Bleeding
4) |Infection
CORRECT ANSWERS
TERITARY |PREVENTION |- |CORRECT |ANSWER✔✔-Ex: |Counseling, |Rehabilitation, |Management |
programs, |Support |groups. |
Goal |is |prevent |furthur |damage |and |maximize |quality |of |life.
SECONDARY |PREVENTION |- |CORRECT |ANSWER✔✔-Ex: |Interventions, |Recommending, |Assisting
|with |finding |work, |Assisting |with |medication. |
Goal |is |to |slow |or |stop |progress.
PRIMARY |PREVENTION |- |CORRECT |ANSWER✔✔-Ex: |Education, |Legistlation, |Regular |exams, |
Vaccinations
Goal |is |to |protect |healthy |people |from |developing |disease.
LATENT |VS. |PRODROMAL |- |CORRECT |ANSWER✔✔-1) |LATENT: |
Exposure |--- |First |S/S
2) |PRODROMAL:
First |S/S |--- |Disease
Endemic |- |CORRECT |ANSWER✔✔-(adj.) |native |or |confined |to |a |particular |region |or |people; |
characteristic |of |or |prevalent |in |a |field
Subclinical |- |CORRECT |ANSWER✔✔-Stage |of |a |disease |or |disorder |not |severe |enough |to |
produce |symptoms |that |can |be |detected |or |diagnosed.
,Functions |NORMALLY.
What |is |an |example |of |etiology? |- |CORRECT |ANSWER✔✔-ETIOLOGY: |Study |of |causes |or |reason
|of |disease. |
1) |Latrogenic: |UNKNOWN
2) |Idiopathic: |UNINTENDED |or |UNWANTED
Allostasis |- |CORRECT |ANSWER✔✔-The |body's |ability |to |maintain |a |STABLE |physiological |
environment |by |adjusting |and |CHANGING |to |meet |internal |and |external |demands.
What |hormones |secrete |during |stress |response? |- |CORRECT |ANSWER✔✔-1) |Coritsol: |
breakdowns |fat |and |protein. |
2)ACTH: |
3)Epinephrine: |
Released |by |adrenal |medullas.
Selyes |three |phases |of |stress |- |CORRECT |ANSWER✔✔-1)Alarm
2)Resistance
3)Exhaustion
The |sympathetic |nervous |system |is |mediated |by |- |CORRECT |ANSWER✔✔-NOREPINEPHRINE
What |are |the |types |of |intracellular |accumulations? |- |CORRECT |ANSWER✔✔-1) |Normal |
Intracellular |accumulation: |Water, |lipids, |proteins, |and |carbohydrates, |that |accumulates |in |
EXCESS. |
,2) |Abnormal |Intracellular |accumulation: |
-Exogenous: |minerals |or |infectious |agent |product.
-Endogenous: |
-abnormal |synthesis |or |metabolism
Types |of |Cellular |Adaptation |- |CORRECT |ANSWER✔✔-1) |Hyperplasia: |INCREASE |in |the |
NUMBER |of |cells. |
-EX: |A |benign |prostatic |hypertrophy |in |middle |aged |men. |
2) |Hypertrophy: |INCREASE |in |the |SIZE |of |cells
3) |Atrophy: |REDUCTION |in |the |SIZE |of |cells. |
-EX: |Skeletal |muscle |atrophy |occurs |when |an |extremity |has |been |immobilized. |
4) |Metaplasia: |CHANGES |to |the |cells |to |adapt |to |new |environments.
5) |Anaplasia: |LOSS |of |cellular |differentiation. |
-Indication |of |malignant |tumors. |
6) |Dysplasia:Cell |MATURATION |and |differentiation |are |delayed.
-Loss |of |uniformity |of |cells. |
-Precursor |to |cancer
EX: |Pap |smear |shows |dysplasia |cells |of |the |cervix
7) |Neoplasia: |NEW |or |expended |GROWTH |in |an |area |of |the |body. |
EX: |Benign |neoplasm |is |uterine |fibroid |tumors. |
, 8) |Apoptosis: |The |genetic |instructions |provided |DNA |to |command |CELLULAR |SELF-
DESTRUCTION
Types |of |Necrosis |- |CORRECT |ANSWER✔✔-1) |Coagulation: |Low |oxygen |levels |(blood |clot)
-Lysomal |changes
-Cyltosol |congeals/coagulates
-Stroma
2) |Liquifaction: |Bacterial |infection. |
-Neutrophils |creats |pus. |
-Cause |abcesses. |
3) |Caseous: |A |slow, |progressive |process |that |produces |thick, |cheesy |pus. |
-Tuberculosis
4) |Fat: |Inflammatory |reaction |with |large |number |of |WBC. |
-Inflammatory |disease |of |pancreas.
5) |Gangrenous: |Combination |of |coagulation |and |liquifcation. |
-Dry, |Wet, |Gas
What |are |side |effects |of |chemotherapy? |- |CORRECT |ANSWER✔✔-1) |Anemia
2)Nausea
3)Bleeding
4) |Infection