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CARDIAC + CCRN EXAM QUESTIONS WITH 100% VERIFIED ANSWERS

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CARDIAC + CCRN EXAM QUESTIONS WITH 100% VERIFIED ANSWERSAcute Coronary Syndrome (definition) - ANSWER Syndrome of acute myocardial ischemia caused by atherosclerotic plaque rupture and thrombus formation Acute Coronary Syndrome (etiology) - ANSWER Same as Coronary Artery Dz, Angina Pectoris, and Myocardial Infarction Coronary Artery Disease (definition) - ANSWER A progressive disease of the coronary arteries that results in their narrowing of obstruction CAD (pathophysiology) - ANSWER 1.Arteriosclerosis: a group of diseases characterized by thickening and loss of elasticity (calcification) of arterial walls 2. Atherosclerosis: the most common form of arteriosclerosis. a. A chronic disease process characterized by the build-up of fatty plague along the subintimal layer of arteries leading to a decrease in arterial luman. 3. Decreased blood flow and oxygen supply to the myocardium lead to imbalance between oxygen supply and oxygen demand. a. Gradual or partial occlusion: ischemia causing angina b. Sudden or complete occlusion: necrosis causing MI Pathologic Consequence of Atherosclerosis and Arteriosclerosis - ANSWER 1. Angina pectoris 2. MI 3. Heart failure 4. Dysrhythmias caused ischemia 5. Sudden death Angina Pectoris (etiology) - ANSWER Factors that decrease supply a. Arteriosclerosis/atherosclerosis b. Coronary artery spasm c. Aortitis d. Dysrhythmias e. Anemia f. Shock Factors that increase demand: a. HTN b. Aortic valve dz c. Tachydysrhythmias d. HF e. Hyperthyroidism Angina Pectoris (clinical presentation - subjective) - ANSWER 1. Subjective CARDIAC + CCRN EXAM QUESTIONS WITH 100% VERIFIED ANSWERS Page 2 of 119 2. Pain may radiate to shoulders, back, arms, jaw, neck, and epigastrium b. Dyspnea c. N/V d. Anxiety e. Weakness Angina Pectoris (objective) - ANSWER a. Tachycardia b. Hypotension or HTN c. Tachypnea d. Pallor e. Diaphoresis g. S4 Angina Pectoris (Diagnostic) - ANSWER a. Serum 1. Isoenzymes: negative for cardiac damage 2. Fasting glucose: to identify DM or glucose intolerance as a risk factor b. ECG 1. ST segment depression in unstable angina 2. ST segment elevation in variant angina 3. Ventricular dysrhythmias may be present c. Cardiac cauterization: coronary artery occlusion 75% or greater; may be negative in variant angina but develop occlusion from spasm Types of Angina - ANSWER Stable and Unstable Angina Stable Angina (signs and symptoms) - ANSWER - Unchanging frequency, duration, and severity - Predictable to the patient - ST segment depression may occur during pain Angina Pectoris (Collaborative Management) - ANSWER 1. Relieve chest pain a. Nitro b. Calcium channel blockers (especially for variant angina) c. Morphine sulfate may be required for unstable angina 2. Increase oxygen demand a. nasal canula 5L/min during ischemic pain unless contraindicated by lung dz. b. Blood transfusion if angina caused by anemia c. Platelet aggregation inhibitors (e.g., aspirin or ticlopidine to prevent platelet aggregation; heparin may be prescribed to prevent clotting or extension of clot d. IABP - may be used in unstable angina; IABP increases coronary artery perfusion pressure (CAPP) 3. Decrease oxygen demand a. Removal of provoking factors a. Substernal chest discomfort that usually lasts 1 to 4 minutes and subsides with rest and/or nitro use. 1. Discomfort may be described as burning, squeezing, tightness, pressure, heaviness, indigestion, or aching. Page 3 of 119 Percutaneous Coronary Intervention (procedures) - ANSWER PTCA (percutaneous transluminal coronary angioplasty): inflation of a balloon-tipped catheter in an area of coronary artery stenosis from plague; plague is pushed back against the wall of vessel and fractured (controlled trauma) Coronary Artery Stent: Use of a metal that acts as a scaffolding device to support a coronary artery and maintain potency after PTCA Brachytherapy: use of intracoronary irradiation to reduce risk of restenosis Coronary atherectomy: removal of plaque from coronary artery by a high-speed diamond tipped (rotational) or shaving (directional) device PCI (indications) - ANSWER Unstable or chronic angina Acute or post acute MI Post-CABG with postoperative angina Patient must be surgical candidate (in case of coronary artery dissection) PCI (contraindications) - ANSWER Left main CAD (unless there is a patent bypass around it, referred to as protected) Stenosis of coronary artery at orifice Variant angina Myocardial Infarction - ANSWER Death of a portion of the myocardium MI (etiology) - ANSWER Most are caused by arteriosclerosis and thrombosis Arteriosclerosis/atherosclerosis Coronary artery thrombosis Coronary artery spasm Cocaine-induced: excessive sympathetic stimulation causes tachycardia, HTN, arterial vasoconstriction, and spasm; coronary artery spasm may cause MI, especially non-Q wave infarction MI (patho) - ANSWER Atherosclerosis with unstable plaque Plaque rupture may be caused by inflammation and/or infection C-reactive protein (CRP) rises rapidly following an inflammatory response

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CARDIAC + CCRN EXAM QUESTIONS WITH 100% VERIFIED ANSWERS




Acute Coronary Syndrome (definition) - ANSWER Syndrome of acute myocardial ischemia caused
by atherosclerotic plaque rupture and thrombus formation

Acute Coronary Syndrome (etiology) - ANSWER Same as Coronary Artery Dz, Angina Pectoris, and
Myocardial Infarction

Coronary Artery Disease (definition) - ANSWER A progressive disease of the coronary arteries that
results in their narrowing of obstruction

CAD (pathophysiology) - ANSWER 1.Arteriosclerosis: a group of diseases characterized by
thickening and loss of elasticity (calcification) of arterial walls

2. Atherosclerosis: the most common form of arteriosclerosis.
a. A chronic disease process characterized by the build-up of fatty plague along the subintimal layer
of arteries leading to a decrease in arterial luman.

3. Decreased blood flow and oxygen supply to the myocardium lead to imbalance between oxygen
supply and oxygen demand.
a. Gradual or partial occlusion: ischemia causing angina
b. Sudden or complete occlusion: necrosis causing MI

Pathologic Consequence of Atherosclerosis and Arteriosclerosis - ANSWER 1. Angina pectoris
2. MI
3. Heart failure
4. Dysrhythmias caused ischemia
5. Sudden death

Angina Pectoris (etiology) - ANSWER Factors that decrease supply
a. Arteriosclerosis/atherosclerosis
b. Coronary artery spasm
c. Aortitis
d. Dysrhythmias
e. Anemia
f. Shock

Factors that increase demand:
a. HTN
b. Aortic valve dz
c. Tachydysrhythmias
d. HF
e. Hyperthyroidism

Angina Pectoris (clinical presentation - subjective) - ANSWER 1. Subjective


Page 1 of 119

,a. Substernal chest discomfort that usually lasts 1 to 4 minutes and subsides with rest and/or nitro use.
1. Discomfort may be described as burning, squeezing, tightness, pressure, heaviness, indigestion, or
aching.

2. Pain may radiate to shoulders, back, arms, jaw, neck, and epigastrium
b. Dyspnea
c. N/V
d. Anxiety
e. Weakness

Angina Pectoris (objective) - ANSWER a. Tachycardia
b. Hypotension or HTN
c. Tachypnea
d. Pallor
e. Diaphoresis
g. S4

Angina Pectoris (Diagnostic) - ANSWER a. Serum
1. Isoenzymes: negative for cardiac damage
2. Fasting glucose: to identify DM or glucose intolerance as a risk factor

b. ECG
1. ST segment depression in unstable angina
2. ST segment elevation in variant angina
3. Ventricular dysrhythmias may be present
c. Cardiac cauterization: coronary artery occlusion 75% or greater; may be negative in variant angina
but develop occlusion from spasm

Types of Angina - ANSWER Stable and Unstable Angina

Stable Angina (signs and symptoms) - ANSWER - Unchanging frequency, duration, and severity
- Predictable to the patient
- ST segment depression may occur during pain

Angina Pectoris (Collaborative Management) - ANSWER 1. Relieve chest pain
a. Nitro
b. Calcium channel blockers (especially for variant angina)
c. Morphine sulfate may be required for unstable angina

2. Increase oxygen demand
a. nasal canula 5L/min during ischemic pain unless contraindicated by lung dz.
b. Blood transfusion if angina caused by anemia
c. Platelet aggregation inhibitors (e.g., aspirin or ticlopidine to prevent platelet aggregation; heparin
may be prescribed to prevent clotting or extension of clot
d. IABP - may be used in unstable angina; IABP increases coronary artery perfusion pressure (CAPP)

3. Decrease oxygen demand
a. Removal of provoking factors


Page 2 of 119

,1. Activity cessation immediately when chest pain occurs
2. Bed rest during pain; semi-Fowler position usually most comfortable
b. NTG as prescribed
1. NTG in doses less than 1 mcg/kg/min is a predominately venous dilator (decreases after load and
preload as well); it decreases myocardial workload and myocardial oxygen consumption by
decreasing preload.

Percutaneous Coronary Intervention (procedures) - ANSWER PTCA (percutaneous transluminal
coronary angioplasty): inflation of a balloon-tipped catheter in an area of coronary artery stenosis
from plague; plague is pushed back against the wall of vessel and fractured (controlled trauma)

Coronary Artery Stent: Use of a metal that acts as a scaffolding device to support a coronary artery
and maintain potency after PTCA

Brachytherapy: use of intracoronary irradiation to reduce risk of restenosis

Coronary atherectomy: removal of plaque from coronary artery by a high-speed diamond tipped
(rotational) or shaving (directional) device

PCI (indications) - ANSWER Unstable or chronic angina
Acute or post acute MI
Post-CABG with postoperative angina
Patient must be surgical candidate (in case of coronary artery dissection)

PCI (contraindications) - ANSWER Left main CAD (unless there is a patent bypass around it,
referred to as protected)
Stenosis of coronary artery at orifice
Variant angina

Myocardial Infarction - ANSWER Death of a portion of the myocardium

MI (etiology) - ANSWER Most are caused by arteriosclerosis and thrombosis

Arteriosclerosis/atherosclerosis

Coronary artery thrombosis

Coronary artery spasm

Cocaine-induced: excessive sympathetic stimulation causes tachycardia, HTN, arterial
vasoconstriction, and spasm; coronary artery spasm may cause MI, especially non-Q wave infarction

MI (patho) - ANSWER Atherosclerosis with unstable plaque

Plaque rupture may be caused by inflammation and/or infection

C-reactive protein (CRP) rises rapidly following an inflammatory response



Page 3 of 119

, Inadequate oxygenation causes anaerobic metabolism - which causes lactic acidoses

Prolonged ischemic causes electrical and mechanical death of myocardium

Ischemia, injury, and acidosis causes electrical irritability; this potentially leads to PVCs, VT, VF

Left main coronary artery

Location:
ECG leads:
Complications: - ANSWER Extensive anterior

V1 - V6

Sudden Cardiac death
HF
Cariogenic shock

Left anterior descending artery

Location:
ECG leads:
Complications: - ANSWER Septal

V1 - V2

Sinus Tachy
AF

Anterior

V3 - V4

Ventricular aneurysm
HF
Cariogenic Shock

Left Circumflex Artery

Location:
ECG Leads:
Complications: - ANSWER Lateral

High: I, aVL
Low: V5, V6

Dysrhythmias
HF


Page 4 of 119

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