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NURS 6501 Mod 1-4 Latest Update Questions with Approved Answers Guaranteed Pass

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NURS 6501 Mod 1-4 Latest Update Questions with Approved Answers Guaranteed Pass - Answer -Primary hyperthyroidism is associated with excess PTH secretion by the parathyroid glands from parathyroid adenomas, parathyroid hyperplasia, parathyroid carcinoma, or genetic causes that lead to multiple endocrine neoplasia. In A 65-year-old male with a history of a fib has had sudden onset abdominal pain. He has been vomiting, had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9. CBC reveals white blood cell count of 15,000. What is most likely the mechanism behind his current symptoms - Answer -Elderly patients are at risk due to atherosclerosis which lead to thrombus formation. Embolic origins usually arise from cardiac arrhythmias usually a fib which leads to pooling of the blood in the atria A 65-year-old male with a history of a fib has had sudden onset abdominal pain. He has been vomiting, had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9. CBC reveals white blood cell count of 15,000. What is most likely the mechanism behind his current symptoms - Answer -This disruption leads to malperfusion of the involved end organ leading to ischemia and ultimately infarction. A 65-year-old male with a history of a fib has had sudden onset abdominal pain. He has been vomiting, had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9. CBC reveals white blood cell count of 15,000. What is most likely the mechanism behind his current symptoms - Answer -Thrombosis of the superior mesenteric artery, whether due to thrombotic or embolic origin, causes a sudden decrease or interruption in the primary blood flow to most of the small bowel as well as the ascending colon.

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NURS 6501 Mod 1-4 Latest Update
Questions with Approved Answers
Guaranteed Pass
- Answer -Primary hyperthyroidism is associated with excess PTH secretion by the parathyroid
glands from parathyroid adenomas, parathyroid hyperplasia, parathyroid carcinoma, or genetic causes
that lead to multiple endocrine neoplasia. In

A 65-year-old male with a history of a fib has had sudden onset abdominal pain. He has been vomiting,
had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9. CBC reveals
white blood cell count of 15,000. What is most likely the mechanism behind his current symptoms -
Answer -Elderly patients are at risk due to atherosclerosis which lead to thrombus formation.
Embolic origins usually arise from cardiac arrhythmias usually a fib which leads to pooling of the blood in
the atria

A 65-year-old male with a history of a fib has had sudden onset abdominal pain. He has been vomiting,
had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9. CBC reveals
white blood cell count of 15,000. What is most likely the mechanism behind his current symptoms -
Answer -This disruption leads to malperfusion of the involved end organ leading to ischemia and
ultimately infarction.

A 65-year-old male with a history of a fib has had sudden onset abdominal pain. He has been vomiting,
had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9. CBC reveals
white blood cell count of 15,000. What is most likely the mechanism behind his current symptoms -
Answer -Thrombosis of the superior mesenteric artery, whether due to thrombotic or embolic
origin, causes a sudden decrease or interruption in the primary blood flow to most of the small bowel as
well as the ascending colon.

A patient exhibited classic signs of type one diabetes. Explain the pathophysiology of polyuria -
Answer -Polyuria is increased urine frequency and a clinical manifestation of type 1 diabetes.
Polyuria occurs because hyper glycemia has an osmotic diuretic effect. Glucose levels filtered by the
kidneys exceed what can be reabsorbed, resulting in glycosuria and large amounts of water loss

Ab - Answer -Antibody, past or chronic phase of infection

Ag - Answer -Antigen, acute phase of infection

And what are the treatment goes for managing pheochromocytoma - Answer -Treatment goals for
pheochromocytomas involve managing excess catecholamine to prevent hypertensive emergencies. This
may be accomplished with alpha and beta blockers or calcium channel blocker's, usually initiated weeks
before surgery.

, And what are the treatment goes for managing pheochromocytoma - Answer -Unless the tumor is
large or metastasis is suspected, the tumor may be removed via laparoscopic surgery. Medication
therapy is provided before, during, and after surgery. malignant tumors are typically managed with
surgery and chemo therapy

Anti-HBc - Answer -Appears at the onset of symptoms in acute hepatitis B and remains for life

Anti-HBs (hepatitis B surface antibody) - Answer -Indicates recovery and immunity from HBV
infection. anti-HBs also develops in a person who has been successfully vaccinated against hepatitis B

Define SIADH and identify any patient characteristics that may have contributed to the development of
SIADH - Answer -ADH is necessary for the kidneys to regulate fluids but excess ADH can lead to
hypervolemia, hypotonic hyponatremia, Edema, weight gain, and G.I. upset.

Define SIADH and identify any patient characteristics that may have contributed to the development of
SIADH - Answer -As sodium levels drop, symptoms of lethargy, confusion, muscle twitching,
seizures, and neurological damage may present. The patient's history of diabetes and treatment with
metformin leads to an increased risk for SIADH

Define SIADH and identify any patient characteristics that may have contributed to the development of
SIADH - Answer -Syndrome of inappropriate anti-diuretic hormone (SIADH) is a disorder
characterized by excessive anti-diuretic hormone (ADH) production in the body without physiological
stimuli to trigger its release.

Describe how gallstones of formed and why they caused the symptoms that the patient presented with -
Answer -Gallstones are formed from impaired metabolism of cholesterol, bilirubin, and bile acids.
Gallstones are always composed of cholesterol, unconjugated bilirubin, bilirubin salts, fatty acids,
calcium carbonate and phosphates and mucin glycoproteins.

Describe how gallstones of formed and why they caused the symptoms that the patient presented with -
Answer -Gallstones form in bile that is super saturated and can begin the process of cholesterol
crystal formation. More crystals aggregate thus enlarging and forming stones.

Describe how gallstones of formed and why they caused the symptoms that the patient presented with -
Answer -These stones may lie dormant or start to move down the cystic or common bile duct.
These stones can cause biliary stasis, bacterial infections, biliary parasites

Describe the factors that could have contributed to the development of a DVT in this patient and explain
how each of the factors could cause DVT - Answer -Platelets become activated, then aggregate,
forming clots. Venous stasis is a result of obesity, patients advanced age, and inability to perform
physical therapy therapy.

Describe the factors that could have contributed to the development of a DVT in this patient and explain
how each of the factors could cause DVT - Answer -Virchow's Triad caused damage to the walls of
the vessels. Injury to the intimal layer of the vessel, antiplatelet substances such as nitric oxide and
prostacyclin, along with the expression of collagen on the vessel wall, causes adherence to the platelets
to the vessel wall.
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