NUR 232 PEDS EVALUATION TEST QUESTIONS AND
ANSWERS SET A+
✔✔Collaborative interventions Acid base imbalance -✔✔-Correcting underlying problem
✔✔Diabetes Mellitus -✔✔Chronic disorder characterized by hyperglycemia and insulin
resistance
✔✔Type 1 diabetes -✔✔destruction of pancreatic beta cells
-absolute insulin deficiency
-skinny
-abrupt onset
-requires synthetic insulin
✔✔Type 2 diabetes -✔✔insulin resistance
-typically overweight
-gradual onset
-frequent family history
-lifestyle (long term consequences)
-may or may not require insulin
✔✔Type 1 diabetes s/s -✔✔-polyuria
-polydyspia
-polyphagia
-weight loss
-enuresis/nocturia
-irritability
-shortened attention span
-lowered frustration tolerance
-dry skin
-blurred vision
-poor wound healing
-fatigue
-flushed skin
, -headache
-frequent infections
-hyperglycemia
-DKA
✔✔Euglycemia -✔✔80-120
✔✔DKA -✔✔when glucose in unavailable body breaks down fat and turns into ketones
BG is 240 or higher
-ketonuria
-acetone breath
-kussmauls
-dehydration
-coma
-hyperkalemia
✔✔DKA management -✔✔Increase BG checks
-Increased fluids (isotonic solution)/potassium (must urinate first)
-admitted to ICU
-weights
-strict I/O
-cardiac monitor
-Oxygen given
-foley Cath may be placed
REGULAR insulin drip
DS-5
✔✔DM longterm consequences -✔✔Microvascular and macrovascular
-retinopathy
-nephropathy
-neuropathy
Prevalence 10-15 years after puberty
Poor vascular control= appears in 2.5- 3 years after diagnosis
Good control= postponed for 20 years
✔✔comorbidites of DM -✔✔Hypertension
-cardiovascular disease
-thyroid function
✔✔DM diagnostics -✔✔-fasting blood glucose
-random blood glucose
-oral glucose tolerance test
-A1C goal is < 6.5%
✔✔DM assessment -✔✔Health history/ risk factors
ANSWERS SET A+
✔✔Collaborative interventions Acid base imbalance -✔✔-Correcting underlying problem
✔✔Diabetes Mellitus -✔✔Chronic disorder characterized by hyperglycemia and insulin
resistance
✔✔Type 1 diabetes -✔✔destruction of pancreatic beta cells
-absolute insulin deficiency
-skinny
-abrupt onset
-requires synthetic insulin
✔✔Type 2 diabetes -✔✔insulin resistance
-typically overweight
-gradual onset
-frequent family history
-lifestyle (long term consequences)
-may or may not require insulin
✔✔Type 1 diabetes s/s -✔✔-polyuria
-polydyspia
-polyphagia
-weight loss
-enuresis/nocturia
-irritability
-shortened attention span
-lowered frustration tolerance
-dry skin
-blurred vision
-poor wound healing
-fatigue
-flushed skin
, -headache
-frequent infections
-hyperglycemia
-DKA
✔✔Euglycemia -✔✔80-120
✔✔DKA -✔✔when glucose in unavailable body breaks down fat and turns into ketones
BG is 240 or higher
-ketonuria
-acetone breath
-kussmauls
-dehydration
-coma
-hyperkalemia
✔✔DKA management -✔✔Increase BG checks
-Increased fluids (isotonic solution)/potassium (must urinate first)
-admitted to ICU
-weights
-strict I/O
-cardiac monitor
-Oxygen given
-foley Cath may be placed
REGULAR insulin drip
DS-5
✔✔DM longterm consequences -✔✔Microvascular and macrovascular
-retinopathy
-nephropathy
-neuropathy
Prevalence 10-15 years after puberty
Poor vascular control= appears in 2.5- 3 years after diagnosis
Good control= postponed for 20 years
✔✔comorbidites of DM -✔✔Hypertension
-cardiovascular disease
-thyroid function
✔✔DM diagnostics -✔✔-fasting blood glucose
-random blood glucose
-oral glucose tolerance test
-A1C goal is < 6.5%
✔✔DM assessment -✔✔Health history/ risk factors