NR 507 Study Guide Questions with 100%
Verified Correct Answers
Hypersensitivity: Type 1
-Mediated IgE
-inflammation due to mast cell degranultion
-anaphylactic, asthma, hay fever
treatment: epinephrine
Hypersensitivity: Type 2
-Cytotoxic reaction: tissue specific
-binds to the antigen ON THE CELL SURFACE
-macrophages are primary effectors cells involved
cause tissue damage or alter function
Hypersensitivity 2
-Graves Disease (hyperthyroidism) -alters function not destroy
-blood transfusions reaction- transfused erythrocytes are destroyed by agglutination or lysis
-drug allergies
-hemolytic anemia
Hypersensitivity: type 3
-Not organ specific
-antibody binds to soluble antigen OUTSIDE THE CELL SURFACE that was released into
the blood or bodily fluids, and the complex is then deposited in the tissues
Hypersensitivity: Type 3
,-rheumatoid arthritis
-Systemic Lupus Erythematosus (SLE)
-Raynaud's
Hives (urticaria)
Hypersensitivity Type 1
First responders to innate the immune system
Neutrophils
Allergic contact dermatitis
Hypersensitivity Type IV
Type 2 cytotoxic hypersensitivity mediated by:
-IgG IgM
-macrophages are primary effort cells
-
Serum Sickness
type 3 hypersensitivity
Hypersensitivity IV,
-is more of a delayed immune response.
-mediated T-cells attack tissue directly (no antibodies)
Autoimmune
-diseases in which the body makes antibodies directed against its own tissues
-
, Primary immunodeficiency
-Genetic; inherited
-result of single gene defects
-B and T cell deficiencies
secondary immunodeficiency
-acquired
Common cause of secondary immunodeficiency
Malnutrition
Most patients diagnosed with SLE will have a positive ?
antinuclear antibody (ANA)
SLE (lupus)
-Facial rash
-vasculitis
- tissue inflammation
Renal disease associated with autoimmunity?
Glomerulonephritis
Sjogren's syndrome
Inflammation in salivary and lacrimal glands
SLE
Autoantibodies and auto-active t-cells against DNA and nucleoprotein
Sjögren's syndrome immune changes:
Verified Correct Answers
Hypersensitivity: Type 1
-Mediated IgE
-inflammation due to mast cell degranultion
-anaphylactic, asthma, hay fever
treatment: epinephrine
Hypersensitivity: Type 2
-Cytotoxic reaction: tissue specific
-binds to the antigen ON THE CELL SURFACE
-macrophages are primary effectors cells involved
cause tissue damage or alter function
Hypersensitivity 2
-Graves Disease (hyperthyroidism) -alters function not destroy
-blood transfusions reaction- transfused erythrocytes are destroyed by agglutination or lysis
-drug allergies
-hemolytic anemia
Hypersensitivity: type 3
-Not organ specific
-antibody binds to soluble antigen OUTSIDE THE CELL SURFACE that was released into
the blood or bodily fluids, and the complex is then deposited in the tissues
Hypersensitivity: Type 3
,-rheumatoid arthritis
-Systemic Lupus Erythematosus (SLE)
-Raynaud's
Hives (urticaria)
Hypersensitivity Type 1
First responders to innate the immune system
Neutrophils
Allergic contact dermatitis
Hypersensitivity Type IV
Type 2 cytotoxic hypersensitivity mediated by:
-IgG IgM
-macrophages are primary effort cells
-
Serum Sickness
type 3 hypersensitivity
Hypersensitivity IV,
-is more of a delayed immune response.
-mediated T-cells attack tissue directly (no antibodies)
Autoimmune
-diseases in which the body makes antibodies directed against its own tissues
-
, Primary immunodeficiency
-Genetic; inherited
-result of single gene defects
-B and T cell deficiencies
secondary immunodeficiency
-acquired
Common cause of secondary immunodeficiency
Malnutrition
Most patients diagnosed with SLE will have a positive ?
antinuclear antibody (ANA)
SLE (lupus)
-Facial rash
-vasculitis
- tissue inflammation
Renal disease associated with autoimmunity?
Glomerulonephritis
Sjogren's syndrome
Inflammation in salivary and lacrimal glands
SLE
Autoantibodies and auto-active t-cells against DNA and nucleoprotein
Sjögren's syndrome immune changes:
