u
peritonitis: spontaneous, absent of a source of bacterial infection primary
peritonitis: inciting cause secondary
Is FIP primary or secondary peritonitis? primary
what are 2 examples of aseptic secondary peritonitis? bile, uroperitoneum
what is the major example of septic secondary peritonitis? GI leakage
Potential sources of GI septic peritonitis:
- of prior GI sx site
- , , or with perforation
- necrosis at site dehiscence, FB, ulceration, neoplasia, intussusception, penetrating, feeding tubes
- gastric necrosis
- abd injury
- dehiscence of gastric or jejunal
Dehiscence is most likely to occur at - days post GI sx 3-5
T/F: prophylactic post-op abx will prevent dehiscence F
Risk factors for dehiscence:
- delayed
- pre-existing
- albuminemia
enteral nutrition, septic peritonitis, hypo, linear, blood, technique
- FB
- multiple intestinal procedures
- administration of products
- poor surgical
what are the 2 most common sites for GI ulceration? stomach, duodenum
what are the 3 most common clinical signs of GI ulcers? vom, hematamesis, melena
Severity of septic peritonitis will partly depend on the source
Are there higher bacterial loads as you move orad or aborad in the intestines? aborad
T/F: typically peritonitis is polymicrobial T
what are the 2 most common bacteria involved in septic peritonitis? e coli, bacteroides fragilis
What is a common sequelae to sepsis where pro-inflamm mediators recruit and activate
SIRS
leukocytes and endothelium?
Initially, animals with septic peritonitis will present with a (hyper/hypo) dynamic shock and
hyper, hypo
progress to (hyper/hypo)dynamic shock.
Is this hyper or hypodynamic shock?
- fever
- injected MM hyper
- tachycardia
- bounding pulses
Is this hyper or hypodynamic shock?
- hypothermia
hypo
- hypotension
- pale MM
Are cats more prone to tachycardia or bradycardia when in shock from peritonitis? bradycardia
increased, decreased
1/3
peritonitis: spontaneous, absent of a source of bacterial infection primary
peritonitis: inciting cause secondary
Is FIP primary or secondary peritonitis? primary
what are 2 examples of aseptic secondary peritonitis? bile, uroperitoneum
what is the major example of septic secondary peritonitis? GI leakage
Potential sources of GI septic peritonitis:
- of prior GI sx site
- , , or with perforation
- necrosis at site dehiscence, FB, ulceration, neoplasia, intussusception, penetrating, feeding tubes
- gastric necrosis
- abd injury
- dehiscence of gastric or jejunal
Dehiscence is most likely to occur at - days post GI sx 3-5
T/F: prophylactic post-op abx will prevent dehiscence F
Risk factors for dehiscence:
- delayed
- pre-existing
- albuminemia
enteral nutrition, septic peritonitis, hypo, linear, blood, technique
- FB
- multiple intestinal procedures
- administration of products
- poor surgical
what are the 2 most common sites for GI ulceration? stomach, duodenum
what are the 3 most common clinical signs of GI ulcers? vom, hematamesis, melena
Severity of septic peritonitis will partly depend on the source
Are there higher bacterial loads as you move orad or aborad in the intestines? aborad
T/F: typically peritonitis is polymicrobial T
what are the 2 most common bacteria involved in septic peritonitis? e coli, bacteroides fragilis
What is a common sequelae to sepsis where pro-inflamm mediators recruit and activate
SIRS
leukocytes and endothelium?
Initially, animals with septic peritonitis will present with a (hyper/hypo) dynamic shock and
hyper, hypo
progress to (hyper/hypo)dynamic shock.
Is this hyper or hypodynamic shock?
- fever
- injected MM hyper
- tachycardia
- bounding pulses
Is this hyper or hypodynamic shock?
- hypothermia
hypo
- hypotension
- pale MM
Are cats more prone to tachycardia or bradycardia when in shock from peritonitis? bradycardia
increased, decreased
1/3
