clinical manifestation of AGN
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-sudden edmea, hematuria, proteinuria, HTN
-smoky or coffee colored urine
-oliguria
-CVA tendencies
clinical manifestations of respiratory alkalosis
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, -hypocapnea
-hypoclacemia
-hypokalemia
small atrophied, scarred kidneys; occurs because of repeated kidney infection
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What is different about Chronic Pyelonephritis?
pathophysiology of CKD
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-fluid filled cysts in both kidney
-hyperplasia in renal epithelial cells
-formation of cysts
-out pocketing of nephron tubule walls
-cysyts proliferate (develop RBC)
-kidneys englarge
-vessels susceptible to rupture
-pain
-cysts
treatment of RCC
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-radiation
-targeted cancer therapy (immune modulators)
, -surgery (localized-nephrectomy, removal of metastatic tumors)
nephrolithiasis (renal calculi)
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formation of stones, most common in white men (recurrence is common)
Respiratory acidosis
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A drop in blood pH due to hypoventilation (too little breathing) and a
resulting accumulation of Co2.
the cells take in H+ and kick out K+ to help balance
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Why does a K+ deficiency happen with acidosis?
collecting tubule
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, distal tubules empty into single larger collecting tubule; responds to ADH
(helps hold on to fluid); regulates secretion of acid; merges into the larger
collecting ducts; more than 99% of the original filtrate is reabsorbed by the
time it reaches pelvis
clinical manifestations of acute pyelonephritis
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-CVA tenderness
-fever
-pyuria
-dysuria
-chills, groin pain, N&V
-complications (septic shock, ARDS, chronic kidney disease due to
scarring)
bicarbonate ions take up released H+ ion and become carbonic acid; excreted
through respiratory system
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What happens if there is too much acid?
ADPKD
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-sudden edmea, hematuria, proteinuria, HTN
-smoky or coffee colored urine
-oliguria
-CVA tendencies
clinical manifestations of respiratory alkalosis
Give this one a try later!
, -hypocapnea
-hypoclacemia
-hypokalemia
small atrophied, scarred kidneys; occurs because of repeated kidney infection
Give this one a try later!
What is different about Chronic Pyelonephritis?
pathophysiology of CKD
Give this one a try later!
-fluid filled cysts in both kidney
-hyperplasia in renal epithelial cells
-formation of cysts
-out pocketing of nephron tubule walls
-cysyts proliferate (develop RBC)
-kidneys englarge
-vessels susceptible to rupture
-pain
-cysts
treatment of RCC
Give this one a try later!
-radiation
-targeted cancer therapy (immune modulators)
, -surgery (localized-nephrectomy, removal of metastatic tumors)
nephrolithiasis (renal calculi)
Give this one a try later!
formation of stones, most common in white men (recurrence is common)
Respiratory acidosis
Give this one a try later!
A drop in blood pH due to hypoventilation (too little breathing) and a
resulting accumulation of Co2.
the cells take in H+ and kick out K+ to help balance
Give this one a try later!
Why does a K+ deficiency happen with acidosis?
collecting tubule
Give this one a try later!
, distal tubules empty into single larger collecting tubule; responds to ADH
(helps hold on to fluid); regulates secretion of acid; merges into the larger
collecting ducts; more than 99% of the original filtrate is reabsorbed by the
time it reaches pelvis
clinical manifestations of acute pyelonephritis
Give this one a try later!
-CVA tenderness
-fever
-pyuria
-dysuria
-chills, groin pain, N&V
-complications (septic shock, ARDS, chronic kidney disease due to
scarring)
bicarbonate ions take up released H+ ion and become carbonic acid; excreted
through respiratory system
Give this one a try later!
What happens if there is too much acid?
ADPKD
Give this one a try later!
